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Dissertations / Theses on the topic 'Structure of rat heart'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Rawlins, D. J. "Quantitative aspects of rat heart structure and function." Thesis, University of York, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.374191.

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2

Kakarla, Sunil Kumar. "Age-related DGC structure and function in the F344/N X BN rat heart." Huntington, WV : [Marshall University Libraries], 2007. http://www.marshall.edu/etd/descript.asp?ref=791.

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3

Stassen, Frank. "Structural and functional changes in peripheral resistance arteries during experimental heart failure." [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=5919.

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4

Pope, Adèle Joanna. "Characterising myocardial remodelling in hypertensive heart disease. Structural and functional changes in the spontaneously hypertensive rat." Thesis, University of Auckland, 2011. http://hdl.handle.net/2292/8517.

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Myocytes of varying orientation across the ventricular wall are grouped into layers, separated by cleavage planes. The role of extracellular matrix, in particular collagen, in this laminar organisation has not been fully delineated due to an inability to truly appreciate its 3D relationships. This thesis utilised an unique laser confocal scanning microscopy system to produce high resolution 3D images of normal adult rat myocardium to elucidate the arrangement of collagen with respect to myocytes. Perimysium was shown to have an ordered arrangement that played a direct role in laminar or

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5

Grytsiuk, M. I. "Mortality structure and rate due to ischemic heart disease among residents of Chernivtsi region." Thesis, БДМУ, 2017. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/12159.

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6

Яркова, А. А. "Використання методу растрової електронної мікроскопії при вивченні будови серця щурів". Thesis, Сумський державний університет, 2015. http://essuir.sumdu.edu.ua/handle/123456789/41547.

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Система кровообігу, а в більш загальному вигляді – система, що забезпечує циркуляцію рідини тіла і виконує основні функції: живлення, виділення, дихання, підтримується порожнистим м'язовим органом – серцем. Серцевий м'яз, міокард, –основний робочий компонент стінки серця. На сьогоднішній день повністю доведено, що особливості будови серцевого м'яза впливають на функціонування серця.

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7

Speller, Lassiter F. "The Effects of Training and Individual Differences in Heart Rate Variability on the Golf Swing’s Coordination Structure." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1345224279.

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8

Davidoff, Allen Warren. "Congestive heart failure in the rat." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0022/MQ31340.pdf.

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9

Jans, Sylvia Wilhelmina Sophia. "Annexin V in the rat heart." [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=5915.

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10

Mohamed, Osama Yousif. "Arrhythmias in the isolated rat heart." Thesis, University of Bath, 1985. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.355303.

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The present study has investigated the effects of potassium, calcium and magnesium on ligation induced arrhythmias in the isolated rat heart. An attempt was made to elucidate the mechanism of action of potassium, however, more work is required to fully explain the action of this caution. Low calcium perfusion was found to reduce arrhythmias, however, calcium antagonists and magnesium, the physiological calcium antagonist, were only effective when they reduced the heart rate. Next, the possible roles of prostaglandins, lysophospholipids and free radicals in ligation induced arrhythmias in the i

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11

Snoeckx, Luc Henricus Elisabeth Hyacinthus. "Ischemia tolerance of the hypertrophied rat heart." Maastricht : Maastricht : Rijksuniversiteit Limburg ; University Library, Maastricht University [Host], 1987. http://arno.unimaas.nl/show.cgi?fid=5399.

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12

Vork, Michaël Maria. "Fatty acid-binding protein in rat heart." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1993. http://arno.unimaas.nl/show.cgi?fid=6232.

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Proefschrift Maastricht.<br>Samenvatting in het Nederlands. Ten dele eerder verschenen en nog te verschijnen art. Auteursnaam op rug en omslag: Michaël Vork. Met lit. opg. en een samenvatting in het Nederlands.

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13

Awad, Wael Ibrahim Issa. "Ischaemic preconditioning in the neonatal rat heart." Thesis, King's College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391636.

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14

Moskalenko, E. A. "Features of the structure of heart." Thesis, Sumy State University, 2017. http://essuir.sumdu.edu.ua/handle/123456789/55332.

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Introduction. Knowledge of century features of a structure of heart of newborns is necessary for diagnostics and treatment of defects of heart, in connection with the high level of birth rate of children with defects of heart. Congenital heart diseases meet frequency of 6-8 cases on each one thousand childbirth which represents 30% of all defects of development. They win first place on mortality of newborns and children of the first year of life.

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15

Romppanen, H. (Hannu). "Regulation of adrenomedullin gene expression in the rat heart." Doctoral thesis, University of Oulu, 1999. http://urn.fi/urn:isbn:9514254783.

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16

Smith, David Michael. "Studies on protein turnover in the perfused rat heart." Thesis, Imperial College London, 1985. http://hdl.handle.net/10044/1/37857.

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17

Silverman, David A. "Red blood cell spacing in capillaries of rat heart." Thesis, University of Ottawa (Canada), 1996. http://hdl.handle.net/10393/9668.

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Theoretical studies have demonstrated a pronounced effect of red blood cell (RBC) spacing on tissue oxygen supply. In spite of this, realistic values for RBC spacing and related capillary hematocrit (Hct) are not known in the heart. One of the possible reasons is the lack of proper methodology. Thus, the goal of this research study was twofold: (i) to develop a method to rapidly freeze rat heart in situ, following which RBCs and capillary walls could be simultaneously visualized, and (ii) to apply this methodology in rat hearts to establish whether differences exist in capillary Hct and RBC sp

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18

Zhang, Ying Ming. "Sarcoplasmic reticulum and myofilament function in failing rat heart." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ34874.pdf.

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19

De, Roode Michael R. "The effects of Taurine depletion on rat heart electrophysiology /." Thesis, McGill University, 1989. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59626.

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Taurine is an amino acid found in high concentration (20-30 mM) in mammalian heart. Treatment of rats with the transport antagonist guanidinoethyl sulfonate (GES) depletes cardiac taurine ($>$70%). Electrocardiograms were recorded weekly in restrained unanaesthetized rats. GES treatment caused a selective prolongation of the QT interval which was correlated with the degree of myocardial taurine depletion (r$ sp2$ = 0.92, p $<$ 0.001). Ventricular muscle action potential durations (APD$ sb{95}$) from taurine-depleted hearts were significantly prolonged compared to control (88 +/- 8 ms vs. 66 +/

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20

Keon, Claudia Anne. "Myocardial energy transduction in the isolated working rat heart." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244563.

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21

Peter, Markus Guenter. "Endothelin receptor expression in human, rat and porcine heart." Thesis, University of Cambridge, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627471.

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22

Johnson, Paula Denise 1959. "Spontaneous congenital heart malformations in the Sprague-Dawley rat." Thesis, The University of Arizona, 1992. http://hdl.handle.net/10150/278074.

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The spontaneous rate of congenital cardiac defects in the commonly used Sprague-Dawley rat has not been extensively researched. The incidence of general congenital defects has been studied thoroughly and reported as very low incidence in this rat. Teratology studies using mammalian models are of increasing importance as the number of environmental contaminants increases. It is essential to know if the spontaneous congenital cardiac defect rate of this most common mammalian model in order to plan statistically appropriate research protocols. Using a thorough and detailed method of evaluating th

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23

Bumeister, L. V. "Structural changes of rat's heart under the influence of hypoosmolar hyperhydration." Thesis, Sumy State University, 2017. http://essuir.sumdu.edu.ua/handle/123456789/53942.

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Actuality: A cardiovascular diseases are the leading cause of death and disability population in the most countries of the world. Disorders of water-salt homeostasis are very common problems encountered in clinical medicine. Disorders of water and electrolyte balance accompany a significant number of diseases of the endocrine, urinary, cardiovascular systems and have a place in daily clinical practice, causing violations of the structure and function of organs.

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24

Du, Ying. "Ischemic and pharmacological preconditioning of rat myocardium : effects on ischemia-reperfusion injury /." View abstract or full-text, 2005. http://library.ust.hk/cgi/db/thesis.pl?BICH%202005%20DU.

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25

Kwak, Hyo Bum. "Exercise training modulates apoptotic signaling in the aging rat heart." Thesis, Texas A&M University, 2004. http://hdl.handle.net/1969.1/2780.

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Aging is characterized by a progressive decline in cardiac function. A critical contributor to the age-related impairment in heart function is the loss of cardiac myocytes through ??apoptosis??, or programmed cell death. A dramatic increase in the rate of apoptosis has been reported with aging in the rat left ventricle. In contrast, exercise training not only improves cardiac function, but also reduces the risk of heart disease. However, the ability of exercise training to modulate apoptotic signaling and apoptosis in the aging heart remains unknown. Therefore, the purpose of this study was to

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26

Kim, Sung Ouk. "Regulation of protein kinases during postnatal development of rat heart." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ34562.pdf.

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27

Fu, Shing-yan Karen, and 符誠欣. "Cigarette smoke-induced inflammatory changes in rat heart in vivo." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B48333864.

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Cigarette smoke (CS) is a well-established risk factor to cardiovascular health and the most preventable cause of death. Countless studies have demonstrated its harm to health and many more studies investigating its pathogenic mechanisms. While the CS-induced pathogenic mechanism of cardiovascular dysfunction has been mainly attributed to a combination of oxidative imbalance, vascular endothelial dysfunction, inflammation and modification of lipid profile, the focus of the current study was on the mediators of inflammation and the activation of signal pathways. In this study, we investigated

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28

Harding, Richard James. "Biochemical pathways underlying cellular damage in the perfused rat heart." Thesis, University of Liverpool, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241343.

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29

Duncombe-Moore, Christopher. "Anomalous kinetic behaviour of rat heart 6-phosphofructo-1-kinase." Thesis, University of Liverpool, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293706.

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30

Daniels, Stephanie. "Biochemical mechanisms of cellular damage in the isolated rat heart." Thesis, University of Liverpool, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293744.

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31

Masithulela, Fulufhelo James. "Computational biomechanics in the remodelling rat heart post myocardial infarction." Doctoral thesis, University of Cape Town, 2016. http://hdl.handle.net/11427/20555.

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Cardiovascular diseases account for one third of all deaths worldwide, more than 33% of which are related to ischemic heart disease, including myocardial infarction (MI). This thesis seeks to provide insight and understanding of mechanisms during different stages of MI by utilizing finite element (FE) modelling. Three-dimensional biventricular rat heart geometries were developed from cardiac magnetic resonance images of a healthy heart and a heart with left ventricular (LV) infarction two weeks and four weeks after infarct induction. From these geometries, FE models were established. To repre

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32

Flint, Nigel Stuart. "Arrhythmogenic potential of alpha-adrenoceptor stimulation in the rat heart." Master's thesis, University of Cape Town, 1985. http://hdl.handle.net/11427/26526.

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A recent proposal is that the alpha₁-adrenoceptor may mediate the arrhythmogenic effect of catecholamines during acute myocardial ischaernia. The purpose of this thesis was to explore the role of alpha₁ and alpha₂-adrenoceptor stimulation on vulnerability to ventricular fibrillation in the norrnoxic rat ventricular myocardium and further to evaluate the possible underlying cellular mechanism. The model used was the isolated perfused rat heart (Langendorff technique) in which ventricular fibrillation was electrically induced. The amount of current required to produce ventricular fibrillation wa

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33

Scudamore, Charles Henry. "Allograft enhancement in a rat heart transplant model : effect of T and B cell immunization." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/24916.

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Acute and chronic rejection continue to be the most important problems in maintaining a functioning organ allograft. Despite advances in immunosuppression, organs are still damaged or destroyed by the recipient's immune system. In order to protect the transplanted organ it is necessary to overwhelm the host's immune system and thus expose the host to the complications of invasion from fungi, bacteria, protazoa, reduction in oncologic surveillance, and reduction of stem cell production. Donor specific immunosuppression would provide graft protection and allow maintenance of the host's immunolo

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34

Ceiler, Deborah Lynn. "Vascular compliance and resistance changes during experimental heart failure." Maastricht : Maastricht : Univrsiteit Maastricht ; University Library, Maastricht University [Host], 1998. http://arno.unimaas.nl/show.cgi?fid=6056.

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35

Mahey, Rajesh. "Regulation of the calcium transport atpase of rat heart sarcoplasmic reticulum." Thesis, University of British Columbia, 1986. http://hdl.handle.net/2429/26901.

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The sarcoplasmic reticulum Ca²⁺ -pumping ATPase is the primary system responsible for the removal of calcium from the sarcoplasm during relaxation of skeletal and cardiac muscles. Since the rat heart SR is used frequently in our laboratory to study the Ca²⁺ -transport defects in disease states, the Ca²⁺ - ATPase activity of this system was characterized. Calmodulin (CaM) and cAMP-dependent protein kinase (cAMP-PK) are known to regulate the dog cardiac SR Ca²⁺ -pump. The effects of these regulators on the rat heart SR Ca²⁺ -pump were studied. Studies were also carried out to investigate t

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36

Dolinsky, Vernon Wayne. "Regulation of phospholipid remodelling by thyroid hormones in the rat heart." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/mq23284.pdf.

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37

Detillieux, Karen A. "Adrenergic regulation of rat FGF-2 gene expression in the heart." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ45146.pdf.

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38

Emter, Craig Allen. "Low-intensity exercise training in the Spontaneously Hypertensive Heart Failure rat." Diss., Connect to online resource, 2006. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3239394.

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39

Takada, Shinji. "Receptors and actions of angiotensin IV in the perinatal rat heart." Thesis, University of Cambridge, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368513.

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40

Connaughton, Mark. "Aspects of ischaemia and reperfusion injury in the isolated rat heart." Thesis, King's College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266208.

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41

Hirai, Taku. "Evaluation of myocardial infarct size in rat heart by pinhole SPECT." Kyoto University, 2000. http://hdl.handle.net/2433/180846.

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42

Hustler, Brenda Irene. "Investigations into mechanisms of magnesium homeostasis in the isolated rat heart." Thesis, University of Central Lancashire, 1997. http://clok.uclan.ac.uk/19282/.

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Magnesium (Mg24) is essential for normal functioning of the heart. It is the second most abundant divalent cation in living cells and plays vital roles in cellular regulation. Preservation of intracellular Mg 24 concentration ([Mg24]) is essential in the heart. The concentration of ionized Mg 24 is similar in both the extracellular and intracellular milieu of cardiac myocytes although a large electrochemical gradient exists which will tend to promote Mg24 influx across the sarcolemmal membrane. Therefore homeostatic mechanism(s) must exist which will extrude Mg 24 against this gradient. The ex

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43

Blackwell, Christopher P. "Free radicals and reperfusion-induced arrhythmias in the isolated rat heart." Thesis, University of Bath, 1988. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.380418.

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44

Zakaria, Mohamed Naguib Mohamed. "Reperfusion induced arrhythmias in the isolated rat heart : the role of oxygen free radicals and the ionic environment of the heart." Thesis, University of Bath, 1985. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.355315.

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Reperfusion of the isolated rat heart following 10 min of coronary artery ligation under constant flow conditions results in the development of arrhythmias - premature ventricular contractions (PVCs), ventricular tachycardia (VT) and ventricular fibrillation (VF). Increasing concentrations of magnesium (0 - 4.8 mM) and/or potassium (2.5 - 10.0 mM) attenuate, while calcium (0.6 - 2.4 mM) exacerbates these arrhythmias. The protective effects of magnesium and potassium were additive. Magnesium reduced heart rate, perfusion pressure and developed tension. Potassium reduced perfusion pressure and i

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45

Lau, Siu-ling. "Characterization of adenosine transport in rat cardiomyocytes, H9c2 /." View the Table of Contents & Abstract, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31494894.

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46

Kilborn, Michael John. "Postnatal changes in electrophysiological properties of rat cardiomyocytes." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.292673.

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47

Swann, Ian D. "Studies of the kallikrein-kinin system in normal and ischaemic rat hearts." Thesis, University of Essex, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236512.

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48

Workman, A. J. "A study of adenosine triphosphate-sensitive potassium channels in rat hearts." Thesis, De Montfort University, 1996. http://hdl.handle.net/2086/4147.

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49

Osypiw, Jacqueline Connett. "Heterogeneity of rat hepatocytes." Thesis, Liverpool John Moores University, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261422.

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50

Ho, Peter D. "Regulation of morphology and intracellular calcium by Ras in rat neonatal cardiac myocytes /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2000. http://wwwlib.umi.com/cr/ucsd/fullcit?p9984293.

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