Relevant bibliographies by topics / Synthèse HP

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  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Synthèse HP'

Author: Grafiati
Published: 25 May 2024
Last updated: 31 July 2025

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Journal articles on the topic "Synthèse HP"

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Pelaprat, N., C. Brondino, G. Rigal, and B. Boutevin. "Synthèse d'alkylphosphonates à longues chaines hydrocarbonées, modifications chimiques et applications—II. Addition de HP(O)(OEt)2 sur les alcènes et phosphonation directe d'halogénures d'alkyles." European Polymer Journal 32, no. 6 (1996): 761–66. http://dx.doi.org/10.1016/0014-3057(95)00189-1.

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Zhu, Hongming, Aijun Sun, Yunzeng Zou, and Junbo Ge. "Inducible Metabolic Adaptation Promotes Mesenchymal Stem Cell Therapy for Ischemia." Arteriosclerosis, Thrombosis, and Vascular Biology 34, no. 4 (2014): 870–76. http://dx.doi.org/10.1161/atvbaha.114.303194.

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Objective— Ischemic tissue is an environment with limited oxygen and nutrition availability. The poor retention of mesenchymal stem cells (MSC) in ischemic tissues greatly limits their therapeutic potential. The aim of this study was to determine whether and how inducible metabolic adaptation enhances MSC survival and therapy under ischemia. Approach and Results— MSC were subjected to glycogen synthase 1–specific small interfering RNA or vehicle treatment, and then sublethal hypoxic preconditioning (HP) was applied to induce glycogenesis. The treated cells were subjected to ischemic challenge.
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Wang, Li, Yuan Zhou, Jun Peng, Zhe Zhang, De-Jian Jiang, and Yuan-Jian Li. "Role of endogenous nitric oxide synthase inhibitor in gastric mucosal injury." Canadian Journal of Physiology and Pharmacology 86, no. 3 (2008): 97–104. http://dx.doi.org/10.1139/y08-003.

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To explore the role of the endogenous nitric oxide synthase (NOS) inhibitor asymmetric dimethylarginine (ADMA) in gastric mucosal injury, 3 models of gastric mucosal injury induced by ethanol, indomethacin, or cold stress were used in rats. The cultured human gastric mucosal epithelial cell line GES-1 infected by Helicobacter pylori (Hp) was selected to mimic human gastric mucosal injury. Gastric mucosal ulcer index (UI), levels of ADMA and NO, and activity of dimethylarginine dimethylaminohydrolase (DDAH) were determined in the mucosal injury models; in Hp-infected or ADMA-treated GES-1 cells
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Hoque, Sanzana, Yuki Kondo, Nodoka Sakata, et al. "Differential Effects of 2-Hydroxypropyl-Cyclodextrins on Lipid Accumulation in Npc1-Null Cells." International Journal of Molecular Sciences 21, no. 3 (2020): 898. http://dx.doi.org/10.3390/ijms21030898.

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Niemann–Pick disease type C (NPC) is an autosomal recessive disorder characterized by abnormal accumulation of free cholesterol and sphingolipids in lysosomes. The iminosugar miglustat, which inhibits hexosylceramide synthesis, is used for NPC treatment, and 2-hydroxypropyl-β-cyclodextrin (HP-β-CD), a cyclic oligosaccharide derivative, is being developed to treat NPC. Moreover, therapeutic potential of 2-hydroxypropyl-γ-cyclodextrin (HP-γ-CD) was shown in NPC models, although its mechanism of action remains unclear. Here, we investigated the effects of HP-β-CD, HP-γ-CD, and their homolog 2-hyd
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Plotnikova, Ekaterina Yu, Lyubov A. Yaroshenko, and Alyona O. Kutsaeva. "Nitric oxide system in patients infected with Helicobacter pylori of different genotypes." Clinical review for general practice 4, no. 1 (2023): 49–52. http://dx.doi.org/10.47407/kr2023.4.1.00189.

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Introduction. Helicobacter pylori (Hp) is a class I biological carcinogen and the third leading cause of cancer death in the world, according to the World Health Organization. Hp infection represents a global public health problem in the 4.4 billion people living with Hp worldwide. The pathogenesis of helicobacteriosis is multifaceted: disorders in the nitric oxide (NO) system are involved in the development of the disease, along with other mechanisms. These aspects of the pathogenesis of helicobacteriosis are not well understood. In particular, such parts of the general problem as the signifi
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Doherty, Neil C., Feifei Shen, Nigel M. Halliday, et al. "In Helicobacter pylori, LuxS Is a Key Enzyme in Cysteine Provision through a Reverse Transsulfuration Pathway." Journal of Bacteriology 192, no. 5 (2010): 1184–92. http://dx.doi.org/10.1128/jb.01372-09.

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ABSTRACT In many bacteria, LuxS functions as a quorum-sensing molecule synthase. However, it also has a second, more central metabolic function in the activated methyl cycle (AMC), which generates the S-adenosylmethionine required by methyltransferases and recycles the product via methionine. Helicobacter pylori lacks an enzyme catalyzing homocysteine-to-methionine conversion, rendering the AMC incomplete and thus making any metabolic role of H. pylori LuxS (LuxSHp) unclear. Interestingly, luxS Hp is located next to genes annotated as cysK Hp and metB Hp, involved in other bacteria in cysteine
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Shih, Jui-Hu, Yow-Fu Tsai, I.-Hsun Li, Ming-Hua Chen та Yuahn-Sieh Huang. "Hp-s1 Ganglioside Suppresses Proinflammatory Responses by Inhibiting MyD88-Dependent NF-κB and JNK/p38 MAPK Pathways in Lipopolysaccharide-Stimulated Microglial Cells". Marine Drugs 18, № 10 (2020): 496. http://dx.doi.org/10.3390/md18100496.

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Hp-s1 ganglioside is isolated from the sperm of sea urchin (Hemicentrotus pulcherrimus). In addition to neuritogenic activity, the biological function of Hp-s1 in neuroinflammation is unknown. In this study, we investigated the anti-neuroinflammatory effect of Hp-s1 on lipopolysaccharide (LPS)-stimulated microglial cells. MG6 microglial cells were stimulated with LPS in the presence or absence of different Hp-s1 concentrations. The anti-inflammatory effect and underlying mechanism of Hp-s1 in LPS-activated microglia cells were assessed through a Cell Counting kit-8 assay, Western blot analysis
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Liu, Jie, Irene Ginis, Maria Spatz та John M. Hallenbeck. "Hypoxic preconditioning protects cultured neurons against hypoxic stress via TNF-α and ceramide". American Journal of Physiology-Cell Physiology 278, № 1 (2000): C144—C153. http://dx.doi.org/10.1152/ajpcell.2000.278.1.c144.

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Brief “preconditioning” ischemia induces ischemic tolerance (IT) and protects the animal brain from subsequent otherwise lethal ischemia. Identification of the signaling steps most proximal to the development of the IT will allow induction of the resistance to ischemia shortly after the onset of stroke. Animal studies demonstrate a key role of tumor necrosis factor-α (TNF-α) in induction of IT. The sphingolipid ceramide is known as a second messenger in many of the multiple effects of TNF-α. We hypothesized that ceramide could mediate IT. We demonstrate that preconditioning of rat cortical neu
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Niepolski, Leszek, and Kamila Malinowska-Loba. "Association of Circulating Endothelial Nitric Oxide Synthase Levels with Phosphataemia in Patients on Haemodialysis." Biomedicines 12, no. 3 (2024): 687. http://dx.doi.org/10.3390/biomedicines12030687.

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The amount of evidence indicates that hyperphosphataemia (HP) can induce endothelial damage and significantly impair endothelial nitric oxide synthase (eNOS) expression. There are no clinical studies that have assessed HP and its correlation with circulating eNOS concentration in patients with end-stage renal disease (ESRD). Our preliminary study aimed to evaluate the relationship between plasma inorganic phosphorus (P) levels and circulating plasma eNOS concentration in patients on haemodialysis (HD). A total of 50 patients on HD were enrolled to the study. They were divided into groups accor
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Huang, Ho-Shiang, Ming-Chieh Ma, and Jun Chen. "Chronic l-arginine administration increases oxidative and nitrosative stress in rat hyperoxaluric kidneys and excessive crystal deposition." American Journal of Physiology-Renal Physiology 295, no. 2 (2008): F388—F396. http://dx.doi.org/10.1152/ajprenal.00405.2007.

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Hyperoxaluric kidneys show an impaired diuretic response to acute infusion of l-arginine. In this study, we examined the chronic effect of l-arginine supplementation on CaOx crystal formation in hyperoxaluric rat kidneys. Eight groups were tested: control (received drinking water), L group (received l-arginine, 0.6%), LN group [received NG-nitro-l-arginine methyl ester (l-NAME, 10 mg/kg)], L + LN group (received l-arginine + l-NAME), HP group [received hydroxyl-l-proline (HP, 5%) mixed with chow to induce hyperoxaluria], L + HP group (received HP + l-arginine), HP + LN group, and L + HP + LN g
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Dissertations / Theses on the topic "Synthèse HP"

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Abushammala, Haneen. "Tuning the multiorbital Mott transition of BaCoS2." Electronic Thesis or Diss., Sorbonne université, 2021. http://www.theses.fr/2021SORUS100.

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Dans cette thèse nous ajustons la phase d’isolant de Mott du BaCoS2, un modèle multiorbital pour l’étude des corrélations électroniques. Nous avons développer une nouvelle technique de self-flux afin de produire des cristaux de qualité et de taille supérieures. Puis nous avons confirmé une phase stable orthorhombique Cmma à l’ambiante où la distorsion orthorhombique est concomitante à l’établissement d’un ordre AFM de longue portée. Ensuite, nous avons exploré la possibilité d’induire un état métallique dans du BaCoS2 via pression chimique et dopage électronique comme paramètres de contrôle. P
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Book chapters on the topic "Synthèse HP"

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Prakasam, M., F. Balima, S. Cygan, P. Klimczyk, L. Jaworska, and A. Largeteau. "Ultrahigh pressure SPS (HP-SPS) as new syntheses and exploration tool in materials science." In Spark Plasma Sintering. Elsevier, 2019. http://dx.doi.org/10.1016/b978-0-12-817744-0.00009-x.

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