Relevant bibliographies by topics / TGF-β Pathway

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'TGF-β Pathway'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "TGF-β Pathway"

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Sopel, Nina, Alexandra Ohs, Mario Schiffer та Janina Müller-Deile. "A Tight Control of Non-Canonical TGF-β Pathways and MicroRNAs Downregulates Nephronectin in Podocytes". Cells 11, № 1 (2022): 149. http://dx.doi.org/10.3390/cells11010149.

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Nephronectin (NPNT) is an extracellular matrix protein in the glomerular basement membrane that is produced by podocytes and is important for the integrity of the glomerular filtration barrier. Upregulated transforming growth factor β (TGF-β) and altered NPNT are seen in different glomerular diseases. TGF-β downregulates NPNT and upregulates NPNT-targeting microRNAs (miRs). However, the pathways involved were previously unknown. By using selective inhibitors of the canonical, SMAD-dependent, and non-canonical TGF-β pathways, we investigated NPNT transcription, translation, secretion, and regul
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Ismaeel, Ahmed, Jeong-Su Kim, Jeffrey S. Kirk, Robert S. Smith, William T. Bohannon та Panagiotis Koutakis. "Role of Transforming Growth Factor-β in Skeletal Muscle Fibrosis: A Review". International Journal of Molecular Sciences 20, № 10 (2019): 2446. http://dx.doi.org/10.3390/ijms20102446.

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Transforming growth factor-beta (TGF-β) isoforms are cytokines involved in a variety of cellular processes, including myofiber repair and regulation of connective tissue formation. Activation of the TGF-β pathway contributes to pathologic fibrosis in most organs. Here, we have focused on examining the evidence demonstrating the involvement of TGF-β in the fibrosis of skeletal muscle particularly. The TGF-β pathway plays a role in different skeletal muscle myopathies, and TGF-β signaling is highly induced in these diseases. In this review, we discuss different molecular mechanisms of TGF-β-medi
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Li, Yuchao, Difan Deng, Chris Tina Höfer та ін. "Liebig’s law of the minimum in the TGF-β/SMAD pathway". PLOS Computational Biology 20, № 5 (2024): e1012072. http://dx.doi.org/10.1371/journal.pcbi.1012072.

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Cells use signaling pathways to sense and respond to their environments. The transforming growth factor-β (TGF-β) pathway produces context-specific responses. Here, we combined modeling and experimental analysis to study the dependence of the output of the TGF-β pathway on the abundance of signaling molecules in the pathway. We showed that the TGF-β pathway processes the variation of TGF-β receptor abundance using Liebig’s law of the minimum, meaning that the output-modifying factor is the signaling protein that is most limited, to determine signaling responses across cell types and in single
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Moradi-Marjaneh, Reyhaneh, Majid Khazaei, Gordon A. Ferns та Seyed H. Aghaee-Bakhtiari. "The Role of TGF-β Signaling Regulatory MicroRNAs in the Pathogenesis of Colorectal Cancer". Current Pharmaceutical Design 24, № 39 (2019): 4611–18. http://dx.doi.org/10.2174/1381612825666190110150705.

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Colorectal cancer (CRC) is one of the most common cancers globally and is associated with a high mortality rate. The transforming growth factor beta (TGF-β) signaling pathway plays an important role in normal intestinal tissue function, but has also been implicated in the development of CRC. MicroRNAs (miRNAs) have also recently emerged as important regulators of cancer development and progression. They act by targeting multiple signaling pathways including the TGF-β signaling pathway. There is growing evidence demonstrating that miRNAs target various components of the TGF-β signaling pathway,
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Elliott, Rebecca L., and Gerard C. Blobe. "Role of Transforming Growth Factor Beta in Human Cancer." Journal of Clinical Oncology 23, no. 9 (2005): 2078–93. http://dx.doi.org/10.1200/jco.2005.02.047.

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Transforming growth factor beta (TGF-β) is a ubiquitous and essential regulator of cellular and physiologic processes including proliferation, differentiation, migration, cell survival, angiogenesis, and immunosurveillance. Alterations in the TGF-β signaling pathway, including mutation or deletion of members of the signaling pathway and resistance to TGF-β-mediated inhibition of proliferation are frequently observed in human cancers. Although these alterations define a tumor suppressor role for the TGF-β pathway in human cancer, TGF-β also mediates tumor-promoting effects, either through diffe
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Lamouille, Samy, та Rik Derynck. "Cell size and invasion in TGF-β–induced epithelial to mesenchymal transition is regulated by activation of the mTOR pathway". Journal of Cell Biology 178, № 3 (2007): 437–51. http://dx.doi.org/10.1083/jcb.200611146.

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Epithelial to mesenchymal transition (EMT) occurs during development and cancer progression to metastasis and results in enhanced cell motility and invasion. Transforming growth factor-β (TGF-β) induces EMT through Smads, leading to transcriptional regulation, and through non-Smad pathways. We observe that TGF-β induces increased cell size and protein content during EMT. This translational regulation results from activation by TGF-β of mammalian target of rapamycin (mTOR) through phosphatidylinositol 3-kinase and Akt, leading to the phosphorylation of S6 kinase 1 and eukaryotic initiation fact
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Nlandu Khodo, Stellor, Surekha Neelisetty, Luke Woodbury та ін. "Deleting the TGF-β receptor in proximal tubules impairs HGF signaling". American Journal of Physiology-Renal Physiology 310, № 6 (2016): F499—F510. http://dx.doi.org/10.1152/ajprenal.00446.2015.

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Transforming growth factor-β (TGF-β) and hepatocyte growth factor (HGF) play key roles in regulating the response to renal injury but are thought to mediate divergent effects on cell behavior. However, how TGF-β signaling alters the response to HGF in epithelia, the key site of HGF signaling in the injured kidney, is not well studied. Contrary to our expectation, we showed that deletion of the TGF-β type II receptor in conditionally immortalized proximal tubule (PT) cells impaired HGF-dependent signaling. This reduced signaling was due to decreased transcription of c-Met, the HGF receptor, and
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Dong, Mei, та Gerard C. Blobe. "Role of transforming growth factor-β in hematologic malignancies". Blood 107, № 12 (2006): 4589–96. http://dx.doi.org/10.1182/blood-2005-10-4169.

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AbstractThe transforming growth factor-β (TGF-β) signaling pathway is an essential regulator of cellular processes, including proliferation, differentiation, migration, and cell survival. During hematopoiesis, the TGF-β signaling pathway is a potent negative regulator of proliferation while stimulating differentiation and apoptosis when appropriate. In hematologic malignancies, including leukemias, myeloproliferative disorders, lymphomas, and multiple myeloma, resistance to these homeostatic effects of TGF-β develops. Mechanisms for this resistance include mutation or deletion of members of th
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García-Sánchez, Omar, Sandra M. Sancho-Martínez, José Miguel López-Novoa та Francisco J. López-Hernández. "Activation of the ALK-5 Pathway is not per se Sufficient for the Antiproliferative Effect of TGF-β1 on Renal Tubule Epithelial Cells". Cellular Physiology and Biochemistry 37, № 4 (2015): 1231–39. http://dx.doi.org/10.1159/000430246.

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Background/Aims: Defective tissue repair underlies renal tissue degeneration during chronic kidney disease (CKD) progression. Unbalanced presence of TGF-β opposes effective cell proliferation and differentiation processes, necessary to replace damaged epithelia. TGF-β also retains arrested cells in a fibrotic phenotype responsible for irreversible scarring. In order to identify prospective molecular targets to prevent the effect of TGF-β during CKD, we studied the signaling pathways responsible for the antiproliferative effect of this cytokine. Methods: Tubule epithelial HK2 and MDCK cells wer
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Jayaraman, Selvaraj. "Molecular docking analysis of imiquimod with the TGF-β targets for oral carcinoma". Bioinformation 19, № 4 (2023): 467–70. http://dx.doi.org/10.6026/97320630019467.

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TGF-β signalling pathway is the main signalling pathways that regulate various biological functions such as cell proliferation, apoptosis, metabolic dysregulation, and metastasis in many cancer cells. Previous studies have elaborated the role of TGF-β signalling targets have a significant regulatory function in various cancers. Moreover targeting the epithelial to mesenchymal transition markers in oral squamous cell carcinoma not yet elucidated. Therefore, it is of interest to document the molecular docking analysis of TGF-β signalling pathway targets such as Smad2, GATA2 and MAFG with imiquim
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Dissertations / Theses on the topic "TGF-β Pathway"

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Kowanetz, Marcin. "Novel Regulators of the TGF-β Signaling Pathway". Doctoral thesis, Uppsala University, Ludwig Institute for Cancer Research, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-5891.

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<p>The transforming growth factor-β (TGF-β) superfamily consists of related multifunctional cytokines, which include TGF-βs, activins, and bone morphogenetic proteins (BMPs) and coordinate several biological responses in diverse cell types. The biological activity of TGF-β members is executed by transmembrane serine/threonine kinase receptors and intracellular Smad proteins. The effects of TGF-β on the epithelium are of high interest. Carcinomas (tumors of epithelial origin) are the most common type of human cancer and frequently exhibit aberrant responses to TGF-β. Therefore, TGF-β can be def
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Sheils, Emma. "The TGF-β signalling pathway in Trichinella spiralis : phylogenetic and functional analysis of TGF-β ligands". Thesis, University of Aberdeen, 2011. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=184012.

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The release of genome sequence information is revolutionising the study of helminth parasites by providing important datasets for comparative genomics, allowing the comprehensive analysis of signalling pathways that regulate nematode development. Much of the current knowledge of nematode signalling pathways is based on studies of the free-living model Caenorhabditis elegans. The recent availability of the Trichinella spiralis genome sequence presented an opportunity to study signalling pathways of this, and other, parasitic nematodes, providing a phylum-wide overview of a given signalling path
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Rodón, Ahnert Laura. "Hyperactivation of the TGF-β signaling pathway in glioblastoma: mechanisms and consequences". Doctoral thesis, Universitat de Barcelona, 2012. http://hdl.handle.net/10803/96417.

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The TGF-β pathway is currently considered a therapeutic target in advanced tumors, including glioblastoma (GBM), and several anti-TGFβ agents are in clinical trials and have shown promising results .A thorough understanding of the molecular mechanisms involved in the protumorigenic function of TGF-β will facilitate the identification of markers of response that can be used to stratify patients to be treated with anti-TGFβ compounds and, moreover, will allow for the discovery of new therapeutic agents. TGF-β is highly active in high-grade glioma, and elevated TGF-β activity confers poor prog
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Kimbrough-Allah, Mawiyah. "Regulation of the PI3-Kinase/PTEN Signaling Pathway by TGF-β in Prostate Cancer Cells". DigitalCommons@Robert W. Woodruff Library, Atlanta University Center, 2018. http://digitalcommons.auctr.edu/cauetds/123.

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Transforming growth factor -β (TGF-β) plays an important role in the progression of prostate cancer. It acts as a tumor suppressor in normal epithelial cells but as a tumor promoter in advanced prostate cancer cells. The PI3-kinase pathway has been shown to play integral roles in many cellular processes including cell proliferation, survival, and cell migration in many cell types. PI3-kinase pathway mediates TGF-β effects on prostate cancer cell migration and invasion. Phosphatase and tensin homolog (PTEN), a tumor suppressor gene, inhibits PI3-kinase pathway and is frequently mutated in prost
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Zieba, Agata. "Application of Proximity Ligation Assay for Multidirectional Studies on Transforming Growth Factor-β Pathway". Doctoral thesis, Uppsala universitet, Molekylära verktyg, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-171952.

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A comprehensive understanding of how the body and all its components function is essential when this knowledge is exploited for medical purposes. The achievements in biological and medical research during last decades has provided us with the complete human genome and identified signaling pathways that governs the cellular processes that facilitates the development and maintenance of higher order organisms. This has brought about the realization that diseases such as cancer is a consequence of genomic aberrations that effects these signaling pathways, endowing cancer cells with the capacity to
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Cepeda, Edgar B. "Mechanisms regulating CXCR4 intracellular traffic and polarization in human hepatocellular carcinoma cells: cross-talk with the TGF-β pathway". Doctoral thesis, Universitat de Barcelona, 2015. http://hdl.handle.net/10803/667580.

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CXCR4 is a chemokine receptor originally discovered and cloned in leukocytes that functions by controling the migration to the inflammatory foci. The CXCR4 chemokine receptor and its ligand, SDF-1α (also named CXCL12), are two important molecules involved in the crosstalk between cancer cells and the microenvironment, which has a relevant role in tumour progression, and consequently are considered as promising targets for cancer therapy. The CXCR4/SDF-1α axis is implicated in the expansion and progression of a wide variety of tumors, including prostate, oesophageal, and liver cancer, among oth
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Zhou, Fa Yang. "Pinocembrin from Penthorum chinense Pursh suppresses hepatic stellate cells activation through a unified SirT3-TGF-β-Smad signaling pathway". Thesis, University of Macau, 2018. http://umaclib3.umac.mo/record=b3952122.

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Boneva, Stefaniya Konstantinova [Verfasser], Barbara [Akademischer Betreuer] Braunger та Frank [Akademischer Betreuer] Schweda. "Characterization of the Neuroprotective Function of the TGF-β Signaling Pathway in the Retina / Stefaniya Konstantinova Boneva ; Barbara Braunger, Frank Schweda". Regensburg : Universitätsbibliothek Regensburg, 2018. http://d-nb.info/115360647X/34.

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Ganapathy, Anuradha. "The role of the TGF-β signal transduction pathway in the development and progression of human squamous cell carcinoma of the skin". Thesis, University of Bristol, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503878.

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This study examined whether the transforming growth factor-β (TGF-P) signalling pathway switches from a tumour suppressing to a pro-metastatic function during the progression of human squamous cell carcinoma (SCC). Preliminary studies aimed to determine whether the HaCaT model of human SCC together with the in vivo technique of floor of the mouth (FOM) transplantation of cells to athymic mice provided a good model of human SCC. Following FOM inoculation, HaCaT was non-tumorigenic while the remaining cell lines displayed an increasingly aggressive phenotype in vivo confirming that the HaCaT mod
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Liu, Ye. "Generation and utilization of knockout mice to elucidate the functions of the TGF-β pathway in mammalian endodermal specification and placental development". The Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=osu1158673346.

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Books on the topic "TGF-β Pathway"

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Lefebvre, Rachel. Molecular dissection of TGF-β induced signal transduction pathways controlling T-cell differentiation. 2008.

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Book chapters on the topic "TGF-β Pathway"

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Spit, Maureen, and Peter ten Dijke. "TGF-β Pathway." In Encyclopedia of Molecular Pharmacology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-21573-6_10062-1.

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Spit, Maureen, and Peter ten Dijke. "TGF-β Pathway." In Encyclopedia of Molecular Pharmacology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57401-7_10062.

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Marathe, Nisha, та Akiko Hata. "TGF-β Signaling Pathway and MicroRNAs in Cardiovascular Disease". У TGF-β in Human Disease. Springer Japan, 2013. http://dx.doi.org/10.1007/978-4-431-54409-8_15.

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Hasanpourghadi, Mohadeseh, and Mohd Rais Mustafa. "TGF-β/Smad Signalling Pathway in Cancer." In Recent Trends in Cancer Biology: Spotlight on Signaling Cascades and microRNAs. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-71553-7_9.

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Wolfraim, Lawrence, Mizuko Mamura, Anita Roberts, and John J. Letterio. "Targeting the TGF-β Pathway In Vivo." In Cytokine Knockouts. Humana Press, 2003. http://dx.doi.org/10.1007/978-1-59259-405-4_24.

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Greenwood, Wendy, and Alejandra Bruna. "TGF-β and the SMAD Signaling Pathway in Carcinogenesis." In Predictive Biomarkers in Oncology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-95228-4_25.

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Torrens, Francisco, and Gloria Castellano. "MEK/ERK Pathway Overactivation in Liver Tumors: TGF-β Death." In Natural Pharmaceuticals and Green Microbial Technology. Apple Academic Press, 2020. http://dx.doi.org/10.1201/9781003003229-8.

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James, Antonisamy William, Hilal Mohiuddin Bhat, Shahid Yousuf, Masrat Jan, Muneeb U. Rehman, and Shahzada Mudasir Rashid. "TGF-β Signaling Pathway and Its Therapeutic Role in Cancer." In Cell Signaling Pathways and Their Therapeutic Implication in Cancers. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-2763-9_11.

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Chen, Xiaochu, and Lan Xu. "Genome-Wide RNAi Screening to Dissect the TGF-β Signal Transduction Pathway." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-2966-5_24.

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Liu, Irwin M., Stephen H. Schilling та Xiao-Fan Wang. "Cooperation Between TGF-β and Wnt Pathway Components in Regulating Mesenchymal Stem Cell Function". У Transforming Growth Factor-β in Cancer Therapy, Volume I. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-292-2_18.

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Conference papers on the topic "TGF-β Pathway"

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Johnston, Randy, Tim Warmke, Rick Wiese, David Hayes та Joseph Hwang. "Abstract 1237: TGF-β signaling pathway analysis using MILLIPLEX MAP TGF-β multiplex panel". У Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1237.

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Schorn, K., S. Hammad, C. De la Torre, M. Ebert, S. Dooley та A. Dropmann. "Effects of TGF-β2 on cholangiocyte activation and the canonical TGF-β signaling pathway". У Viszeralmedizin 2024. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1789859.

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Collum, S., T. Mertens, C. Wilson та ін. "Alternative Polyadenylation of TGF-β Pathway Components Modulates Pulmonary Hypertension". У American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5294.

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Chen, Liang, Meng Wang, Jin Peng та ін. "Abstract 5031: miR-21 targets TGF-β Pathway in breast cancer". У Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5031.

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Rao, Shuyun, Heather Levin, Jian Chen та ін. "Abstract 5330: Targeting hepatocellular carcinoma through TGF-β pathway E3 ligases". У Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5330.

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Ohshiro, Kazufumi, Jian Chen, Wilma Jogunoori та ін. "Abstract 4443: Targeting E3 ligase PJA1 via TGF-β pathway in hepatocellular carcinoma". У Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4443.

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Ohshiro, Kazufumi, Jian Chen, Wilma Jogunoori та ін. "Abstract 4443: Targeting E3 ligase PJA1 via TGF-β pathway in hepatocellular carcinoma". У Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4443.

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Yang, Yu-an, Kathleen Flanders, Jin-qui Chen та ін. "Abstract B13: Targeting the TGF-β pathway in breast cancer: Insights from preclinical studies". У Abstracts: AACR Special Conference: Advances in Breast Cancer; October 17-20, 2015; Bellevue, WA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.advbc15-b13.

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Bose, Deepak, Rema P, Suchetha S, Sivaranjith J, Lakshmi S та Rari P. Mony. "Altered expressions of TGF-β signaling pathway components: possible prognostic role in endometrial cancer". У ASGO 2023. Korean Society of Gynecologic Oncology, 2024. http://dx.doi.org/10.3802/jgo.2024.35.s1.0147.

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Yang, Yu-an, Christina Stuelten, Youngjae Bahn та ін. "Abstract 1645: A new TGF-b pathway reporter mouse for analysis of TGF-β signaling in normal homeostasis and cancer". У Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-1645.

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Reports on the topic "TGF-β Pathway"

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Osathanon, Thanaphum. Gene expression profile of continuous and intermittent compressive stress treated human periodontal ligament cells. Faculty of Dentistry Chulalongkorn University, 2019. https://doi.org/10.58837/chula.res.2019.7.

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Mechanical force regulates periodontal ligament cell (PDL) behavior. However, different force types lead to distinct PDL responses. Here, we report that pretreatment with an intermittent compressive force (ICF), but not a continuous compressive force (CCF), promoted human PDL (hPDL) osteogenic differentiation as determined by osteogenic marker gene expression and mineral deposition in vitro. ICF-induced osterix (OSX) expression was inhibited by cycloheximide and monensin. Although CCF and ICF significantly increased extracellular adenosine triphosphate (ATP) levels, pretreatment with exogenous
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