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Books on the topic 'The polymorphism of genes'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Masatoshi, Nei, ed. Humanpolymorphic genes. Oxford University Press, 1988.

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2

Roychoudhury, Arun K. Human polymorphic genes: World distribution. Oxford University Press, 1988.

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3

Berkowitz, Noah C. Functional importance of polymorphic subregions in the C3H anti I-Ab alloresponse. [Columbia University], 1993.

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4

Oelbaum, Raymond Stuart. An analysis of four candidate genes for non-insulin-dependent diabetes using restriction fragment length polymorphism markers. University of Manchester, 1994.

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5

Koen, Vandenbroeck, ed. Cytokine gene polymorphisms in multifactorial conditions. CRC Taylor & Francis, 2006.

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6

Ellegren, Hans. Genome analysis with microsatellite markers. Dept. of Animal Breeding and Genetics, Swedish University of Agricultural Sciences, 1993.

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7

George, Stamatoyannopoulos, and Nienhuis Arthur W, eds. Experimental approaches for the study of hemoglobin switching: Proceedings of the Fourth Conference on Hemoglobin Switching, October 1-3, 1984. A.R. Liss, 1985.

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8

Kiaris, Hippokratis. Genes, polymorphisms, and the making of societies: How genetic behavioral traits influence human cultures. Universal-Publishers, 2012.

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9

George, Stamatoyannopoulos, Nienhuis Arthur W, and Conference on Hemoglobin Switching (7th : 1990 : Airlie, Va.), eds. The Regulation of hemoglobin switching. Johns Hopkins University Press, 1991.

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10

Liu, Zhanjiang. Next generation sequencing and whole genome selection in aquaculture. Wiley-Blackwell, 2011.

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11

George, Stamatoyannopoulos, and Nienhuis Arthur W, eds. Hemoglobin switching: Proceedings of the Sixth Conference on Hemoglobin Switching, held in Airlie, Virginia, September 24-27, 1988. A.R. Liss, 1989.

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12

Bieber, Heidi. Traces of antiquity in genes: Comparative study of serum protein polymorphisms between populations of Africa, Europe and Amerindians of Central America. Shaker Verlag, 1997.

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13

Stanley, H. Eugene, ed. Liquid Polymorphism. John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118540350.

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14

Danks, H. V., ed. Insect life-cycle polymorphism. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-017-1888-2.

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15

Hamilton, W. D., and J. C. Howard, eds. Infection, Polymorphism and Evolution. Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-0077-6.

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16

Aga, Syed Sameer, Mujeeb Zafar Banday, and Saniya Nissar. Genetic Polymorphism and Disease. CRC Press, 2022. http://dx.doi.org/10.1201/9781003246244.

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17

G, Brittain H., ed. Polymorphism in pharmaceutical solids. M. Dekker, 1999.

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18

1936-, Hamilton W. D., Howard Jonathan 1943-, and Royal Society (Great Britain), eds. Infection, polymorphism, and evolution. Chapman & Hall, 1997.

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19

Bernstein, Joel. Polymorphism in molecular crystals. Oxford University Press, 2007.

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20

Meseguer, José. Relating models of polymorphism. Center for the Study of Language and Information, 1988.

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21

G, Brittain H., ed. Polymorphism of pharmaceutical solids. 2nd ed. Informa Healthcare, 2009.

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22

Sameer, Aga Syed, Mujeeb Zafar Banday, and Saniya Nissar, eds. Genetic Polymorphism and cancer susceptibility. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6699-2.

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23

Hilfiker, Rolf, and Markus von Raumer, eds. Polymorphism in the Pharmaceutical Industry. Wiley-VCH Verlag GmbH & Co. KGaA, 2018. http://dx.doi.org/10.1002/9783527697847.

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24

Mitchell, John Edward. Structural polymorphism in repeated DNA. University of Portsmouth, School of Biological Sciences, 1995.

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25

Coquand, Thierry. Domain theoretic models of polymorphism. University of Cambridge, Computer Laboratory, 1987.

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26

Rolf, Hilfiker, ed. Polymorphism in the pharmaceutical industry. Wiley-VCH, 2006.

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27

J, Henry Robert, ed. Plant genotyping II: SNP technology. CABI, 2008.

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28

de, Vienne D., ed. Molecular markers in plant genetics and biotechnology. Science Publishers, 2003.

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29

Lewin, Benjamin. Genes. 3rd ed. Wiley, 1987.

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30

Vandenbroeck, Koen. Cytokine Gene Polymorphisms in Multifactorial Conditions. Taylor & Francis Group, 2006.

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31

Vandenbroeck, Koen. Cytokine Gene Polymorphisms in Multifactorial Conditions. Taylor & Francis Group, 2006.

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32

Han, Shihui. Gene-culture interaction on human behavior and the brain. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780198743194.003.0007.

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Chapter 7 reviews empirical findings that allow consideration of biological and environmental influences on human behavior from an evolutionary perspective (e.g., gene-culture coevolution) and from a perspective of individual development (e.g., gene-culture interaction). It also reviews imaging genetic studies that link genes with brain functional organization. It introduces a cultural neuroscience paradigm for investigating genetic influences on the coupling of brain activity and culture by presenting two studies that examined how serotonin transporter functional polymorphism and oxytocin rec

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33

Vandenbroeck, Koen. Cytokine Gene Polymorphisms in Multifactorial Conditions. CRC, 2006.

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34

Walsh, Bruce, and Michael Lynch. Using Molecular Data to Detect Selection: Signatures from Multiple Historical Events. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198830870.003.0010.

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This chapter examines the search for a pattern of repetitive adaptive substitutions over evolutionary time. In contrast with the previous chapter, only a modest number of tests toward this aim have been proposed. The HKA and McDonald-Kreitman tests contrast the polymorphism to divergence ratio between different genomic classes (such as different genes or silent versus replacement sites within the same gene). These approaches can detect an excess of substitutions, which allows one to estimate the fraction of adaptive sites. This chapter reviews the empirical data on estimates of this fraction a

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35

Morell-Ducos, Fausto. COMT and morphine use in cancer pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0082.

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The landmark paper discussed in this chapter is ‘Genetic variation in the catechol-O-methyltransferase (COMT) gene and morphine requirements in cancer patients with pain’, published by Rakvåg et al. in 2008. Genetic variation contributes to differences in pain sensitivity and response to analgesics. Catecholamines are involved in the modulation of pain and are metabolized by catchol-O-methyltransferase (COMT). Genetic variability in the COMT gene may therefore contribute to differences in pain sensitivity and response to analgesics. It has been shown that a polymorphism in the COMT gene, Rs468

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36

(Editor), Graham R. Taylor, and Ian N. Day (Editor), eds. Guide to Mutation Detection. Wiley, 2005.

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37

Samuels, Jack, Marco A. Grados, Elizabeth Planalp, and O. Joseph Bienvenu. Genetic Understanding of OCD and Spectrum Disorders. Edited by Gail Steketee. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195376210.013.0025.

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This chapter reviews the evidence for the genetic etiology of OCD and spectrum conditions. A genetic basis is supported by the familial aggregation of OCD; evidence for involvement of genes of major effect in segregation analyses; and higher concordance for OCD in identical than non-identical twins. Recent studies also support linkage of OCD to specific chromosomal regions and association of OCD with specific genetic polymorphisms. However, specific genes causing OCD have not yet been firmly established. The search for genes is complicated by the clinical and etiologic heterogeneity of OCD, as

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38

A Primer of Genome Science. Sinauer Associates, 2001.

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39

Stomatoyannopoulos, George, and Arthur W. Nienhuis. Hemoglobin Switching, Part A: Transcriptional Regulation: Proceedings of the Sixth Conference on Hemoglobin Switching Held in Airlie, Virginia, sept. Wiley-Liss, 1989.

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40

Karabon, Lidia. The Role of Polymorphisms in Co-Signalling Molecules' Genes in Susceptibility to B-Cell Chronic Lymphocytic Leukaemia. INTECH Open Access Publisher, 2012.

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41

Kiaris, Hippokratis. Genes, Polymorphisms, and the Making of Societies: A Genetic Perspective of the Divergence Between East and West. Universal Publishers, 2021.

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42

Nienhuis, Arthur W., and George Stomatoyannpoulos. Hemoglobin Switching, Part B: Cellular and Molecular Mechanisms (Progress in Clinical and Biological Research, Vol 316B). Wiley-Liss, 1989.

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43

Hemoglobin switching: Proceedings of the Sixth Conference on Hemoglobin Switching, held in Airlie, Virginia, September 24-27, 1988 (Progress in clinical and biological research). A.R. Liss, 1989.

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44

Fabbri, Chiara, and Alessandro Serretti. The treatment of bipolar disorder in the era of personalized medicine: myth or promise? Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0031.

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Bipolar disorder (BD) is a chronic disease associated with high personal and socio-economic burden. Genetics accounts for 20–95% of variability in central nervous system drug disposition and pharmacodynamics, thus genetic markers are considered a promising way to develop tailored treatments and improve the prognosis of the disease. Among mood stabilizers, lithium response was the most investigated phenotype and the most replicated genes are involved in synaptic plasticity (BDNF), serotonergic (SLC6A4) and dopaminergic (DRD1) neurotransmission, and second messenger cascades (GSK3B). Relevant ph

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45

Meijerink, Eduardus Hendrikus Petrus. Determination of susceptibility to oedema disease by the identification of polymorphisms in the [alpha](1,2)fucosyltransferase genes of the pig. 1999.

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46

Divan, Aysha, and Janice A. Royds. 2. DNA. Oxford University Press, 2016. http://dx.doi.org/10.1093/actrade/9780198723882.003.0002.

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Another significant milestone was the publication in 2003 of the complete sequence of the human genome—the entire DNA contained within the forty-six chromosomes located in the nucleus of each human somatic (body) cell. Once this was published, further worldwide projects were launched to work out what the functions of these genes and other regions of the genome actually were. ‘DNA’ outlines the components of the human genome and their organization; DNA replication; mutations and correction mechanisms; polymorphisms; and new DNA technologies, including gene cloning, the polymerase chain reaction

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47

Boudreau, Joseph F., and Eric S. Swanson. Polymorphism. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198708636.003.0012.

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Polymorphism is a programming style that is supported by C++ and other languages which allows objects of different classes to be treated in a common way and permits high level abstractions for C++ functions. With proper use, polymorphism can become the basis of robust, flexible code that anticipates future extensions without requiring modification to an existing code base. It underpins a programming style called object-oriented programming, which can be used to manage the complexity of a computing project, and which must be fully understood to function in a modern programming environment. The

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48

Hilfiker, Rolf, ed. Polymorphism. Wiley, 2006. http://dx.doi.org/10.1002/3527607889.

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49

Blackley, T. R. Polymorphism. Independently Published, 2018.

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50

Geschonke, Kirsten. Einfluss eines Polymorphismus im Promotor des intestinalen Fatty Acid Binding Protein Genes auf dessen Expression sowie auf die Höhe von Plasmatriglyceridspiegeln. 2006.

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