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Relevant bibliographies by topics / Whole-brain map

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Journal articles

Academic literature on the topic 'Whole-brain map'

Author: Grafiati

Published: 10 March 2023

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Journal articles on the topic "Whole-brain map"

1

Brittin, Christopher A., Steven J. Cook, David H. Hall, Scott W. Emmons, and Netta Cohen. "A multi-scale brain map derived from whole-brain volumetric reconstructions." Nature 591, no. 7848 (2021): 105–10. http://dx.doi.org/10.1038/s41586-021-03284-x.

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Lee, Dongha, and Hae-Jeong Park. "A populational connection map for the whole brain white matter." IBRO Reports 6 (September 2019): S153. http://dx.doi.org/10.1016/j.ibror.2019.07.486.

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Bernat, James L. "THE BIOPHILOSOPHICAL BASIS OF WHOLE-BRAIN DEATH." Social Philosophy and Policy 19, no. 2 (2002): 324–42. http://dx.doi.org/10.1017/s0265052502192132.

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Notwithstanding these wise pronouncements, my project here is to characterize the biological phenomenon of death of the higher animal species, such as vertebrates. My claim is that the formulation of “whole-brain death” provides the most congruent map for our correct understanding of the concept of death. This essay builds upon the foundation my colleagues and I have laid since 1981 to characterize the concept of death and refine when this event occurs. Although our society's well-accepted program of multiple organ procurement for transplantation requires the organ donor first to be dead, the

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Shi, Yuhang, S. Johanna Vannesjo, Karla L. Miller, and Stuart Clare. "Template-based field map prediction for rapid whole brain B0 shimming." Magnetic Resonance in Medicine 80, no. 1 (2017): 171–80. http://dx.doi.org/10.1002/mrm.27020.

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Jung, Youjin, Raymond P. Viviano, Sanneke van Rooden, Jeroen van der Grond, Serge A. R. B. Rombouts, and Jessica S. Damoiseaux. "White Matter Hyperintensities and Apolipoprotein E Affect the Association Between Mean Arterial Pressure and Objective and Subjective Cognitive Functioning in Older Adults." Journal of Alzheimer's Disease 84, no. 3 (2021): 1337–50. http://dx.doi.org/10.3233/jad-210695.

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Background: White matter hyperintensities (WMH) show a robust relationship with arterial pressure as well as objective and subjective cognitive functioning. In addition, APOE ɛ4 carriership may influence how arterial pressure affects cognitive functioning. Objective: To determine the role of region-specific WMH burden and APOE ɛ4 carriership on the relationship between mean arterial pressure (MAP) and cognitive function as well as subjective cognitive decline (SCD). Methods: The sample consisted of 87 cognitively unimpaired middle-aged to older adults aged 50–85. We measured WMH volume for the

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Morone, Flaviano, Kevin Roth, Byungjoon Min, H. Eugene Stanley, and Hernán A. Makse. "Model of brain activation predicts the neural collective influence map of the brain." Proceedings of the National Academy of Sciences 114, no. 15 (2017): 3849–54. http://dx.doi.org/10.1073/pnas.1620808114.

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Efficient complex systems have a modular structure, but modularity does not guarantee robustness, because efficiency also requires an ingenious interplay of the interacting modular components. The human brain is the elemental paradigm of an efficient robust modular system interconnected as a network of networks (NoN). Understanding the emergence of robustness in such modular architectures from the interconnections of its parts is a longstanding challenge that has concerned many scientists. Current models of dependencies in NoN inspired by the power grid express interactions among modules with

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7

Deleo, Francesco, Seok-Jun Hong, Fatemeh Fadaie, et al. "Whole-brain multimodal MRI phenotyping of periventricular nodular heterotopia." Neurology 95, no. 17 (2020): e2418-e2426. http://dx.doi.org/10.1212/wnl.0000000000010648.

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ObjectiveTo test the hypothesis that in periventricular nodular heterotopia (PVNH) structure and function of cortical areas overlying the heterotopic gray matter are preferentially affected.MethodsWe studied a group of 40 patients with PVNH and normal-appearing cortex and compared their quantitative MRI markers of brain development, structure, and function to those of 43 age- and sex-matched healthy controls. Inspired by models of neocortical development suggesting that neuronal migration follows a curvilinear path to preserve topologic correspondence between the outer ventricular zone and the

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8

Tao, Jingshan, Yong Cai, Yisheng Dai, Yingdi Xie, Hailing Liu, and Xiaojin Zang. "Value of 4D CT Angiography Combined with Whole Brain CT Perfusion Imaging Feature Analysis under Deep Learning in Imaging Examination of Acute Ischemic Stroke." Computational Intelligence and Neuroscience 2022 (June 13, 2022): 1–9. http://dx.doi.org/10.1155/2022/2286413.

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This study was aimed at investigating the application of deep learning 4D computed tomography angiography (CTA) combined with whole brain CT perfusion (CTP) imaging in acute ischemic stroke (AIS). A total of 46 patients with ischemic stroke were selected from the hospital as the research objects. Image quality was analyzed after the 4D CTA images were obtained by perfusion imaging. The results showed that whole brain perfusion imaging based on FCN can achieve automatic segmentation. FCN segmentation results took a short time, an average of 2-3 seconds, and the Dice similarity coefficient (DSC)

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9

Jehl, Markus, Ekaterina Mikhaylova, Valerie Treyer, et al. "Attenuation Correction Using Template PET Registration for Brain PET: A Proof-of-Concept Study." Journal of Imaging 9, no. 1 (2022): 2. http://dx.doi.org/10.3390/jimaging9010002.

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NeuroLF is a dedicated brain PET system with an octagonal prism shape housed in a scanner head that can be positioned around a patient’s head. Because it does not have MR or CT capabilities, attenuation correction based on an estimation of the attenuation map is a crucial feature. In this article, we demonstrate this method on [18F]FDG PET brain scans performed with a low-resolution proof of concept prototype of NeuroLF called BPET. We perform an affine registration of a template PET scan to the uncorrected emission image, and then apply the resulting transform to the corresponding template at

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10

Yang, Hsiao T., and Kevin J. Cummings. "Brain stem serotonin protects blood pressure in neonatal rats exposed to episodic anoxia." Journal of Applied Physiology 115, no. 12 (2013): 1733–41. http://dx.doi.org/10.1152/japplphysiol.00970.2013.

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In neonatal rodents, a loss of brain stem serotonin [5-hydroxytryptamine (5-HT)] in utero or at birth compromises anoxia-induced gasping and the recovery of heart rate (HR) and breathing with reoxygenation (i.e., autoresuscitation). How mean arterial pressure (MAP) is influenced after an acute loss of brain stem 5-HT content is unknown. We hypothesized that a loss of 5-HT for ∼1 day would compromise MAP during episodic anoxia. We injected 6-fluorotryptophan (20 mg/kg ip) into rat pups (postnatal days 9–10 or 11–13, n = 22 treated, 24 control), causing a ∼70% loss of brain stem 5-HT. Pups were

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