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Relevant bibliographies by topics / Addisons disease

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Academic literature on the topic 'Addisons disease'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Addisons disease"

1

Bathula, Akanksha, Sanjana Nagulapally, Vaishnavi Nelikanti, Vairale Ashwini, Ankireddy Neha, and Devi Yasa Gayatri. "Addisons Disease: A Brief Review." International Journal of Innovative Science and Research Technology (IJISRT) 9, no. 2 (2024): 7. https://doi.org/10.5281/zenodo.10691605.

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Addison’s disease is a rare condition which occurs due to improper function or damage of the adrenal glands. The two outer layers of cells of the adrenal glands i.e. adrenal cortex are responsible for the production of several steroid hormones. Addison’s disease, also known as primary adrenal insufficiency, result from the insufficient production of these two hormones, cortisol and aldosterone. These hormones help regulate metabolism, blood pressure and stress response. The absence of cortisol and aldosterone can affect almost every organ and tissue in the body. The symptoms of Add

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2

Sak, H., I. Adali, and F. Manoudi. "TROUBLE PSYCHOTIQUE BREF REVELANT UNE DECOMPENSATION DE LA MALADIE DADDISON : A PROPOS DUN CAS." International Journal of Advanced Research 11, no. 04 (2023): 414–16. http://dx.doi.org/10.21474/ijar01/16674.

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Addisons disease is a relatively rare endocrine disorder thatgenerally results from a slow progresss deficiency in the adrenal cortex. The psychiatric symptoms at the onset of the disease are typically represented by a depressive state and instinctive disorders, which may be confused with a psychiatric etiology, delay diagnosis, and/or constitute a circumstance for the discovery of this disease. Furthermore, the occurrence of psychotic disorders is rarely described in cases of adrenal insufficiency, and can sometimes occur during an acute decompensation of the disease. The link between these t

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Jones, David E. J. "Addisons' other disease: primary biliary cirrhosis as a model autoimmune disease." Clinical Medicine 3, no. 4 (2003): 351–56. http://dx.doi.org/10.7861/clinmedicine.3-4-351.

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OE, KENJI. "A case of Addisons' disease complicated by low gonadotropin hypogonadism." Nihon Naika Gakkai Zasshi 82, no. 6 (1993): 911–13. http://dx.doi.org/10.2169/naika.82.911.

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Rouimi, Leila, Sana Abourazzak, Sanae Chaouki, and Moustapha Hida. "APECED SYNDROME (FAMILY OBSERVATION)." International Journal of Advanced Research 11, no. 03 (2023): 693–97. http://dx.doi.org/10.21474/ijar01/16483.

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Introduction: APECED (Autoimmune Polyendocrinopathy Candidiasis Ectodermal Dystrophy) is an autosomal recessive disease characterized by the association of autoimmune endocrine involvement, mucocutaneous candidiasis and ectodermal tissue involvement. Observation: We report the case of 2 brothers from a 2nd degree consanguineous marriage with a history of 2 deaths in the siblings for whom the diagnosis of APECED syndrome was retained on clinical and biological criteria (genetic study in progress).The eldest had candidous onychomycosis of the hands and feet, generalized mucocutaneous candidiasis

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Muralinath, E., Devi Pooja, Chbukdhara Prasanta, et al. "Drugs Acting On Adreno Cortico Trophic Hormones." Journal of Advanced Research and Reviews in Virology & Microbiology 1, no. 1 (2024): 13–15. https://doi.org/10.5281/zenodo.10837718.

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<em>The production of adreno corticotropic hormone ( ACTH ) occurs by pituitary gland and activates the adrenal glands to release cortisol. The synthetic drugs namely corticosteroids mimic the effects of natural cortisol. Addisons disease is manifested by adrenal I sufficient iency and frequently necessitate corticosteroid replacement therapy to control normal physiological activities. Glucocorticoid receptor modulators (GRMs) influence glucocorticoid receptors and minimize the side effects related broad _ activity corticosteroids. Synthetic ACTH analogs play a role in replicating the hormone

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Bhandhavi, V., and A. Rabia. "A Middle-Aged Adult with Persistent Fatigue and Hyperpigmentation." medtigo Journal of Medicine 2, no. 1 (2024): e3032213. https://doi.org/10.63096/medtigo3062213.

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Addison's disease is a rare endocrine illness caused by inadequate adrenal activity resulting from multiple reasons. It frequently presents with weight loss. The patients may seek advice from other experts and may even need to be admitted to the hospital immediately before the problem is diagnosed. Autoimmune polyendocrine syndromes (APS) kinds 1 & 2 sometimes exhibit a high prevalence of coexistence with other autoimmune illnesses. In this case, we report an individual who sought assistance from the medical team subsequent to prior referrals to gastrointestinal services as a result of end

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Tverskaya, S. S. "The Literary Description of Addison's Disease (From the Story by I. S. Turgenev "A Living Relic")." Medicina 8, no. 2 (2020): 117–26. http://dx.doi.org/10.29234/2308-9113-2020-8-2-117-126.

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Retrospective diagnosis of disease of Lukerya, female protagonist of the story is Turgenev's "Living relic" from “A Sportsman's Sketches” series. The aim of the study is to clarify the diagnosis. Based on the acute onset of the disease, due to injury, the presence of "bronze" skin pigmentation, pronounced adynamia, exhaustion, Addison's disease is diagnosed. Given the detailed literary characteristics of the disease, it is proposed to name Addison's disease as "Addison–Turgenev's disease" and "Lukerya's disease".

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Raman, Srimathy, Prakruthi KP, Vinutha G, and Padmalatha Venkataram. "Management considerations in Addison’s disease complicating pregnancy." Indian Journal of Obstetrics and Gynecology Research 8, no. 2 (2021): 273–74. http://dx.doi.org/10.18231/j.ijogr.2021.057.

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Addison’s disease is a rare endocrine disorder. We present the management of a pregnant lady with Addison’s disease. She was managed jointly with the endocrinologists and the titrated dose of hydrocortisone and fludrocortisone dosage was regulated. Her antenatal care was uneventful and she had emergency caesarean section at 39 weeks in view of abnormal CTG. Her labour was appropriately covered with rescue steroids. However she went into hypotension immediately after delivery. She was resuscitated and was managed further in ICU where high dose steroids were given. She made an uneventful recover

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Fadeev, V. V., I. I. Buziashvili, and I. I. Dedov. "Etiological and clinical structure of primary chronic adrenal insufficiency: retrospective analysis of 426 cases." Problems of Endocrinology 44, no. 6 (1998): 22–26. http://dx.doi.org/10.14341/probl11664.

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A retrospective analysis of case histories of 426 patients with primary chronic adrenal insufficiency (1-AI), followed up at Endocrinology Research Center, Russian Academy of Medical Sciences, and/or Endocrinology Clinic of I. M. Setchenov Moscow Medical Academy in 1954-1977, was carried out in order to assess the etiological and clinical structure of this disease. The idiopathic form of Addison 's disease prevailed over its tuberculous form (70 and 30%), respectively); dynamic assessment of morbidity over the above period showed a progressive predominance of the idiopathic form. 1-AI is more

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Dissertations / Theses on the topic "Addisons disease"

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Gebre-Medhin, Gennet. "Clinical and experimental studies of organ-specific autoimmune diseases : With special reference to Addison's disease and autoimmune hepatitis : by Gennet Gebre-Medhin." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5043-1/.

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2

Wolf, Jutta Manuela. "Psychosocial Stress and Addison's Disease." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2006. http://nbn-resolving.de/urn:nbn:de:swb:14-1142418669410-00716.

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Die Psychoneuroimmunologie beschäftigt sich unter anderem mit der Frage, ob und unter welchen Bedingungen psychosozialer Stress krank macht. Humanstudien, die dieser Frage nachgehen, können dabei meist nur korrelative Zusammenhänge aufdecken. Um trotzdem Aussagen zu Mechanismen, Ursache-Wirkungsbeziehungen und klinische Relevanz treffen zu können, muss z.B. auf Befunde aus der Tierforschung oder aus in vitro-Studien zurückgegriffen werden. Ziel der vorliegenden Arbeit war es, eine Methode zu finden, welche eine breitere Interpretationsgrundlage für korrelative Befunde aus Humanstudien liefert.

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Santana, Ana Paula Álvaro. "Hipoadrenocorticismo primário no cão : estudo retrospectivo de 10 casos clínicos." Bachelor's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2009. http://hdl.handle.net/10400.5/983.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>O hipoadrenocorticismo é uma doença endócrina, pouco comum em cães (ou aparentemente sub-diagnosticada), que resulta de uma produção insuficiente de mineralocorticóides e de glucocorticóides pela glândula adrenal. A destruição do córtex da adrenal caracteriza o Hipoadrenocorticismo primário, ou Doença de Addison, e é a causa mais comum da insuficiência do córtex em cães. Esta doença pode ter várias origens, entre as quais uma destruição auto-imune ou uma causa idiopática. O hipoadrenocorticismo primário é uma doença sistémica d

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Mitchell, Anna Louise. "Genomic analyses of familial and sporadic autoimmune Addison's disease." Thesis, University of Newcastle upon Tyne, 2013. http://hdl.handle.net/10443/2477.

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Autoimmune Addison’s disease (AAD) is a rare and highly heritable endocrinopathy. It is a complex genetic disease, meaning that it is due to a combination of interacting environmental and genetic factors. To date, the majority of the substantial genetic component to AAD aetiology remains undefined. In this study, a combination of hypothesis-driven (candidate gene) and discovery-driven (genome-wide) approaches have been used to search for novel genetic determinants of AAD. PCR-based approaches were undertaken to study the potential role of the CYP21A1P pseudogene in AAD. CYP21A1P is highly homo

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Gan, Earn Hui. "Pathophysiology and novel therapeutic approaches in autoimmune Addison's disease." Thesis, University of Newcastle upon Tyne, 2015. http://hdl.handle.net/10443/2801.

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Autoimmune Addison’s disease (AAD) is a debilitating condition and affected patients rely on lifelong steroid replacement. Despite treatment, many patients have increased morbidity and mortality. The disease’s rarity has precluded large scale genomic or cellular studies in humans, resulting in an incomplete picture of the pathophysiology of AAD. This thesis details my research on the pathophysiology and novel therapeutic approaches in AAD. I performed two candidate gene studies on susceptibility alleles that have been implicated in other autoimmune diseases to explore potential causal pathways

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Boag, Alisdair Matthew. "An immunological and genetic investigation of canine hypoadrenocorticism (Addison's Disease)." Thesis, Royal Veterinary College (University of London), 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618317.

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Garelli, Silvia. "La sopravvivenza dei pazienti affetti da morbo di Addison in Italia." Doctoral thesis, Università degli studi di Padova, 2016. http://hdl.handle.net/11577/3425236.

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Introduction: Survival of patients affected by Addison’s Disease (AD) is controversial, because only few studies have been published so far. We decided to evaluate survival of an Italian group of patients, affected by AD of various causes. Materials and Methods: We examinated 1315 patients (58.6% females and 41.4% males) affected by AD, followed-by Endocrine Units all over Italy: the sum of follow-up years was 16983. 70.5% of patients has autoimmune AD (AAD), 113 with APS-1 and 814 with con AAD not APS-1 correlated (AAD not APS-1); 29.5% has non autoimmune AD, 98 of which with post-tubercu

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Ross, Ian Louis. "The aetiopathogenesis, cardiovascular and metabolic complications, and pharmacogenomics of Addison's disease in South Africa." Doctoral thesis, University of Cape Town, 2011. http://hdl.handle.net/11427/10862.

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Includes abstract.<br>Includes bibliographical references.<br>This thesis aimed to address a number of unanswered research questions in Addison's Disease: investigate whether autoimmunity is the predominant cause of Addison's disease in South Africa and if a human leukocyte (HLA) DQ antigen association exists; the extent to which lipids, lipoproteins and biochemical markers of cardiovascular disease are abnormal; the degree to which replacement doses of hydrocortisone are supra-physiological; the impact of glucocorticoid receptor (GCR) polymorphisms on risk factors, markers of card

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Madadi, Nazanin. "Assessing the effect of Addison's disease on patient quality of life within the South African context." Master's thesis, University of Cape Town, 2008. http://hdl.handle.net/11427/11648.

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Includes bibliographical references (leaves 43-46).<br>This study finds that our patients do indeed have the same QOL, and that we should therefore be able to apply the European findings here. Measuring QOL alone, however, gives us very general information which is not sufficient for our study. The second aim of this research is to compare different aspects of QOL in AD, as this has not been researched before. This knowledge should help clinicians to recognise QOL impairment better, which will enable modifiable risk factors to be adjusted with early intervention. (For example, psychological im

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Henry, Michelle. "Associations between sleep architecture, cortisol concentrations, cognitive performance, and quality of life in patients with Addison's disease." Doctoral thesis, Faculty of Humanities, 2019. http://hdl.handle.net/11427/30377.

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Recent literature in the neurosciences suggests that there are mechanistic relations between sleep disruption and cognitive (particularly memory) deficits, and that varying concentrations of the hormone cortisol may play a particularly important role in mediating those relations. Because patients with Addison’s disease (AD) experience consistent and predictable periods of sub- and supra-physiological cortisol concentrations (due to lifelong glucocorticoid replacement therapy), and because they frequently report disrupted sleep and poor memory, those presenting with that endocrinological disord

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Books on the topic "Addisons disease"

1

Association, Canada Medical, ed. Typical case of Addison's disease: With remarks. Lovell, 1986.

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Anderson, Linda. What you can do about adrenal insufficiency. Clinical Center Communications, National Institutes of Health, 1988.

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Parker, James N., and Philip M. Parker. Addison's disease: A medical dictionary, bibliography, and annotated research guide to Internet references. ICON Health Publications, 2003.

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Justin, Lam, and Lam Carrie, eds. Advanced symptoms of adrenal fatigue syndrome: A metabolic perspective. Adrenal Institute Press, 2016.

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5

Pearce, Prof Simon, Spain, Sarah, Pearce, Prof Simon. Living with Addisons Disease: A Guide for People with Addisons, Supporters and Professionals. Addison's Disease Self-Help Group, 2019.

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Typical case of Addison's disease: With remarks. Lovell, printer, 1986.

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Addison Disease: A Guide on Addison's Disease Cure, Causes, Prevention and Management. Independently Published, 2021.

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8

Winqvist, Ola. Autoantigens in Addison's Disease. Almqvist & Wiksell Internat., 1994.

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9

PILBERY. Standby Cpd: Addison's Disease. Class Publishing, 2012.

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Addison Disease: Healing Principles, Treatment and Natural Remedy for Addison Disease. Independently Published, 2021.

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Book chapters on the topic "Addisons disease"

1

Falorni, Alberto. "Addison’s Disease." In Immunoendocrinology: Scientific and Clinical Aspects. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-478-4_24.

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La Rosa, Stefano. "Addison’s Disease." In Endocrine Pathology. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-62345-6_5243.

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Kannan, C. R. "Addison’s Disease." In The Adrenal Gland. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1001-3_2.

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La Rosa, Stefano. "Addison’s Disease." In Encyclopedia of Pathology. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-319-28845-1_5243-1.

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Scharnagl, Hubert, Winfried März, Markus Böhm, et al. "Addison’s Disease." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8409.

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Spanheimer, Robert G. "Addison’s Disease." In Early Diagnosis and Treatment of Endocrine Disorders. Humana Press, 2003. https://doi.org/10.1007/978-1-59259-378-1_13.

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van der Wal, Jacqueline E. "Addison’s Disease: Primary." In Encyclopedia of Pathology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-28845-1_2790-1.

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van der Wal, Jacqueline E. "Addison’s Disease: Primary." In Encyclopedia of Geoarchaeology. Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-3-319-28085-1_2790.

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Oette, Mark, Marvin J. Stone, Hendrik P. N. Scholl, et al. "Morbus Addison." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_5009.

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Bartholdi, Deborah, Albert Schinzel, Deborah Bartholdi, et al. "X-linked Addison’s Disease." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8063.

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Conference papers on the topic "Addisons disease"

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Saraswat, Nidhi, Mayur Agarwal, and Syed Harron. "The Smart Early Detection of Addison's Disease Using Time Series Analysis." In 2024 International Conference on Optimization Computing and Wireless Communication (ICOCWC). IEEE, 2024. http://dx.doi.org/10.1109/icocwc60930.2024.10470911.

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