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Dissertations / Theses on the topic 'Addisons disease'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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1

Gebre-Medhin, Gennet. "Clinical and experimental studies of organ-specific autoimmune diseases : With special reference to Addison's disease and autoimmune hepatitis : by Gennet Gebre-Medhin." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5043-1/.

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2

Wolf, Jutta Manuela. "Psychosocial Stress and Addison's Disease." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2006. http://nbn-resolving.de/urn:nbn:de:swb:14-1142418669410-00716.

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Die Psychoneuroimmunologie beschäftigt sich unter anderem mit der Frage, ob und unter welchen Bedingungen psychosozialer Stress krank macht. Humanstudien, die dieser Frage nachgehen, können dabei meist nur korrelative Zusammenhänge aufdecken. Um trotzdem Aussagen zu Mechanismen, Ursache-Wirkungsbeziehungen und klinische Relevanz treffen zu können, muss z.B. auf Befunde aus der Tierforschung oder aus in vitro-Studien zurückgegriffen werden. Ziel der vorliegenden Arbeit war es, eine Methode zu finden, welche eine breitere Interpretationsgrundlage für korrelative Befunde aus Humanstudien liefert.

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3

Santana, Ana Paula Álvaro. "Hipoadrenocorticismo primário no cão : estudo retrospectivo de 10 casos clínicos." Bachelor's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2009. http://hdl.handle.net/10400.5/983.

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Dissertação de Mestrado Integrado em Medicina Veterinária<br>O hipoadrenocorticismo é uma doença endócrina, pouco comum em cães (ou aparentemente sub-diagnosticada), que resulta de uma produção insuficiente de mineralocorticóides e de glucocorticóides pela glândula adrenal. A destruição do córtex da adrenal caracteriza o Hipoadrenocorticismo primário, ou Doença de Addison, e é a causa mais comum da insuficiência do córtex em cães. Esta doença pode ter várias origens, entre as quais uma destruição auto-imune ou uma causa idiopática. O hipoadrenocorticismo primário é uma doença sistémica d

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4

Mitchell, Anna Louise. "Genomic analyses of familial and sporadic autoimmune Addison's disease." Thesis, University of Newcastle upon Tyne, 2013. http://hdl.handle.net/10443/2477.

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Autoimmune Addison’s disease (AAD) is a rare and highly heritable endocrinopathy. It is a complex genetic disease, meaning that it is due to a combination of interacting environmental and genetic factors. To date, the majority of the substantial genetic component to AAD aetiology remains undefined. In this study, a combination of hypothesis-driven (candidate gene) and discovery-driven (genome-wide) approaches have been used to search for novel genetic determinants of AAD. PCR-based approaches were undertaken to study the potential role of the CYP21A1P pseudogene in AAD. CYP21A1P is highly homo

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5

Gan, Earn Hui. "Pathophysiology and novel therapeutic approaches in autoimmune Addison's disease." Thesis, University of Newcastle upon Tyne, 2015. http://hdl.handle.net/10443/2801.

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Autoimmune Addison’s disease (AAD) is a debilitating condition and affected patients rely on lifelong steroid replacement. Despite treatment, many patients have increased morbidity and mortality. The disease’s rarity has precluded large scale genomic or cellular studies in humans, resulting in an incomplete picture of the pathophysiology of AAD. This thesis details my research on the pathophysiology and novel therapeutic approaches in AAD. I performed two candidate gene studies on susceptibility alleles that have been implicated in other autoimmune diseases to explore potential causal pathways

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6

Boag, Alisdair Matthew. "An immunological and genetic investigation of canine hypoadrenocorticism (Addison's Disease)." Thesis, Royal Veterinary College (University of London), 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618317.

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7

Garelli, Silvia. "La sopravvivenza dei pazienti affetti da morbo di Addison in Italia." Doctoral thesis, Università degli studi di Padova, 2016. http://hdl.handle.net/11577/3425236.

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Introduction: Survival of patients affected by Addison’s Disease (AD) is controversial, because only few studies have been published so far. We decided to evaluate survival of an Italian group of patients, affected by AD of various causes. Materials and Methods: We examinated 1315 patients (58.6% females and 41.4% males) affected by AD, followed-by Endocrine Units all over Italy: the sum of follow-up years was 16983. 70.5% of patients has autoimmune AD (AAD), 113 with APS-1 and 814 with con AAD not APS-1 correlated (AAD not APS-1); 29.5% has non autoimmune AD, 98 of which with post-tubercu

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8

Ross, Ian Louis. "The aetiopathogenesis, cardiovascular and metabolic complications, and pharmacogenomics of Addison's disease in South Africa." Doctoral thesis, University of Cape Town, 2011. http://hdl.handle.net/11427/10862.

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Includes abstract.<br>Includes bibliographical references.<br>This thesis aimed to address a number of unanswered research questions in Addison's Disease: investigate whether autoimmunity is the predominant cause of Addison's disease in South Africa and if a human leukocyte (HLA) DQ antigen association exists; the extent to which lipids, lipoproteins and biochemical markers of cardiovascular disease are abnormal; the degree to which replacement doses of hydrocortisone are supra-physiological; the impact of glucocorticoid receptor (GCR) polymorphisms on risk factors, markers of card

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9

Madadi, Nazanin. "Assessing the effect of Addison's disease on patient quality of life within the South African context." Master's thesis, University of Cape Town, 2008. http://hdl.handle.net/11427/11648.

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Includes bibliographical references (leaves 43-46).<br>This study finds that our patients do indeed have the same QOL, and that we should therefore be able to apply the European findings here. Measuring QOL alone, however, gives us very general information which is not sufficient for our study. The second aim of this research is to compare different aspects of QOL in AD, as this has not been researched before. This knowledge should help clinicians to recognise QOL impairment better, which will enable modifiable risk factors to be adjusted with early intervention. (For example, psychological im

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10

Henry, Michelle. "Associations between sleep architecture, cortisol concentrations, cognitive performance, and quality of life in patients with Addison's disease." Doctoral thesis, Faculty of Humanities, 2019. http://hdl.handle.net/11427/30377.

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Recent literature in the neurosciences suggests that there are mechanistic relations between sleep disruption and cognitive (particularly memory) deficits, and that varying concentrations of the hormone cortisol may play a particularly important role in mediating those relations. Because patients with Addison’s disease (AD) experience consistent and predictable periods of sub- and supra-physiological cortisol concentrations (due to lifelong glucocorticoid replacement therapy), and because they frequently report disrupted sleep and poor memory, those presenting with that endocrinological disord

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11

Elfström, Peter. "Associated disorders in celiac disease." Doctoral thesis, Örebro universitet, Hälsoakademin, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-5223.

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Background: Celiac disease (CD) is an autoimmune disorder that affects genetically susceptible individuals and is induced by dietary gluten. Treatment consists of a lifelong gluten-free diet. CD is common and affects about 1% of the general population. The classic symptoms include diarrhea and malabsorption, but many patients have only mild symptoms or no symptoms at all. The proportion of individuals presenting with atypical symptoms or discovered only when investigating an associated condition of CD is increasing. Aims: The aim of this thesis was to investigate the risk of possible associate

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12

Mendes, Marta Sofia Miguel Ribeiro. "Insuficiência adrenal em cães: estudo retrospectivo de 6 casos clínicos." Master's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2013. http://hdl.handle.net/10400.5/5873.

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Dissertação de Mestrado Integrado em Medicina Veterinária. Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária<br>O Hipoadrenocorticismo Primário é uma doença endócrina relativamente rara, afectando sobretudo cães. As suas causas são uma destruição imuno-mediada ou idiopática do córtex da glândula adrenal, que dão origem a uma insuficiente produção de corticosteróides, nomeadamente de glucocorticóides e de mineralocorticóides. O Hipoadrenocorticismo primário é uma doença que não tem idade, sexo ou raça específica, apesar de ser mais comum em fêmeas esterilizadas. Os seus sinais

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13

Bianchi, Matteo. "Genetic Studies of Immunological Diseases in Dogs and Humans." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-319962.

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This thesis presents genetic studies aiming at enlarging our knowledge regarding the genetic factors underlying two immune-mediated diseases, hypothyroidism and autoimmune Addison’s disease (AAD), in dogs and humans, respectively. Genetic and environmental factors are indicated to contribute to canine hypothyroidism, which can be considered a model for human Hashimoto’s thyroiditis (HT). In Paper I we performed the first genome-wide association (GWA) study of this disease in three high-risk dog breeds (Gordon Setter, Hovawart and Rhodesian Ridgeback). Using an integrated GWA and meta-analysis

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14

Grenbäck, Eva. "Galanin in the pituitary and in human circulation : clinical and experimental studies /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-980-7/.

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15

Gambelunghe, Giovanni. "Immunogenetic studies in autoimmune endocrine diseases /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-691-x/.

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16

Alimohammadi, Mohammad. "Molecular Targets in Autoimmune Polyendocrine Syndrome Type1 and Their Clinical Implications." Doctoral thesis, Uppsala universitet, Institutionen för medicinska vetenskaper, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9549.

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Autoimmune diseases occur when the immune system attacks and destroys healthy body tissue. Autoimmunity is known to cause a wide range of disorders, and is suspected to be responsible for many more. Most autoimmune disorders are chronic and cause severe morbidity for the patients, and are also costly for society. A majority of these disorders are today considered as complex diseases with incompletely known etiology. Hence, model systems for studying the pathogenesis of autoimmunity are important to unravel its causes. Autoimmune Polyendocrine Syndrome Type 1 (APS-1), (OMIM 240300), is a rare a

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17

Gagliardi, Lucia. "Regulation of cortisol secretion in humans: relation to vasopressin action at the adrenals in macronodular and micronodular adrenocortical tumours; and well-being in Addison’s Disease." Thesis, 2011. http://hdl.handle.net/2440/78635.

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The hypothalamic-pituitary-adrenal (HPA) axis exhibits tight physiological regulation on a circadian and ultradian basis in humans. Key central regulators include the peptides corticotrophin-releasing hormone (CRH) and arginine vasopressin (VP), acting at the pituitary, and at peripheral structures relevant to the HPA axis and other components of the stress system. Altered regulation has many causes, frequently related to tumorigenesis, and can lead to disease due to an excess of the HPA axis end-organ hormone cortisol, as in Cushing’s syndrome (CS), or cortisol deficiency, as in Addison’s dis

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18

Wolf, Jutta Manuela [Verfasser]. "Psychosocial stress and Addison's disease : a new approach to evaluate the relevance of endocrine stress responses for health and disease / von Wolf, Jutta Manuela." 2006. http://d-nb.info/97944408X/34.

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19

Ameixa, Mariana Chícharo Mansinhos. "Hipoadrenocorticismo canino : revisão de literatura e descrição de 4 casos clínicos." Master's thesis, 2021. http://hdl.handle.net/10437/11996.

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Orientação: Rute Teixeira ; co-orientação: Pedro Morais de Almeida<br>Esta dissertação foi realizada no âmbito da conclusão do Mestrado Integrado em Medicina Veterinária da Universidade Lusófona de Humanidades e Tecnologias e teve como objetivo o desenvolvimento do tema Hipoadrenocorticismo canino. Inclui uma revisão bibliográfica extensa sobre o tema e apresentação e discussão de 4 casos clínicos. O Hipoadrenocorticismo primário canino decorre da falha ou incapacidade da glândula adrenal em secretar hormonas esteroides. Em cerca de 95% dos casos, os animais apresentam simultaneamente défice

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20

Rodrigues, Ana Marta Castelão. "Alterações eletrolíticas no hipoadrenocorticismo canino: estudo de 10 casos clínicos." Master's thesis, 2017. http://hdl.handle.net/10437/8351.

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Orientação: Margarida Alves ; co-orientação: Pedro Almeida<br>O Hipoadrenocorticismo é uma doença endócrina de ocorrência incomum no cão e deve-se, na maioria dos casos, à destruição imunomediada das glândulas adrenais. Esta doença é caracterizada pela deficiente produção de corticosteroides que, consequentemente, origina alterações em vários sistemas. Com o presente trabalho pretendeu-se avaliar retrospectivamente a evolução dos valores eletrolíticos (sódio, potássio e cloro) ao longo do tratamento, em cães com hipoadrenocorticismo primário. Como objectivo secundário, pretendeu-se relacionar

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21

Reis, Ana Amélia do Nascimento. "Distúrbios das glândulas suprarrenais." Master's thesis, 2016. http://hdl.handle.net/10284/5802.

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Este trabalho apresenta essencialmente dois objetivos. O primeiro refere-se ao funcionamento, anatomia, histologia e metabolismo das glândulas suprarrenais e o segundo trata dos principais distúrbios destas glândulas com a respetiva caraterização, diagnóstico e tratamento. As glândulas suprarrenais fazem parte do sistema endócrino do organismo humano. São divididas anatomicamente em duas partes, o córtex e a medula. O córtex suprarrenal segrega os mineralocorticóides, os glucocorticóides e os androgénios, enquanto a medula suprarrenal é responsável pela secreção de adrenalina (epinefrin

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