Relevant bibliographies by topics / Arrhythmic syndrome

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Arrhythmic syndrome'

Author: Grafiati
Published: 28 May 2022
Last updated: 30 July 2025

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Journal articles on the topic "Arrhythmic syndrome"

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van Bavel, Joanne J. A., Henriëtte D. M. Beekman, Agnieszka Smoczyńska, Marcel A. G. van der Heyden, and Marc A. Vos. "IKs Activator ML277 Mildly Affects Repolarization and Arrhythmic Outcome in the CAVB Dog Model." Biomedicines 11, no. 4 (2023): 1147. http://dx.doi.org/10.3390/biomedicines11041147.

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Long QT syndrome type 1 with affected IKs is associated with a high risk for developing Torsade de Pointes (TdP) arrhythmias and eventually sudden cardiac death. Therefore, it is of high interest to explore drugs that target IKs as antiarrhythmics. We examined the antiarrhythmic effect of IKs channel activator ML277 in the chronic atrioventricular block (CAVB) dog model. TdP arrhythmia sensitivity was tested in anesthetized mongrel dogs (n = 7) with CAVB in series: (1) induction experiment at 4 ± 2 weeks CAVB: TdP arrhythmias were induced with our standardized protocol using dofetilide (0.025
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Eremeev, A. "Atrial extrasystoles associated with hypervitaminosis D3 (clinical case)." Vrach 35, no. 4 (2024): 52–56. http://dx.doi.org/10.29296/25877305-2024-04-10.

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The article discusses a clinical case of a patient with hypervitaminosis D3. Cholecalciferol intoxication occurred against the background of long-term oral use of vitamin complexes and was manifested by the manifestation of arrhythmic syndrome – frequent atrial extrasystole of the “P on T” type. The probable pathogenetic cause of arrhythmia was a violation of ion homeostasis in the form of hypercalcemia. Calcium imbalance, affecting cardiac conduction and automatism, can provoke cardiac arrhythmias by increasing the dispersion of refractoriness. At the electrophysiological level, the cause of
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Agarwal, Nitin, Payodh Chaudhary, Malini Kulshrestha, and RR Chaudhary. "Brugada Syndrome: Killer Genetic Heart Disorder." International Journal of Advanced and Integrated Medical Sciences 1, no. 4 (2016): 186–87. http://dx.doi.org/10.5005/jp-journals-10050-10058.

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ABSTRACT Brugada syndrome is a rare inherited arrhythmic disorder, which predisposes to ventricular arrhythmia that is responsible for sudden and unexpected nocturnal death syndrome. How to cite this article Chaudhary P, Agarwal N, Kulshrestha M, Chaudhary RR. Brugada Syndrome: Killer Genetic Heart Disorder. Int J Adv Integ Med Sci 2016; 1(4):186-187.
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Balasundaram, Lakshmi, Bharatraj Kidambi, and Surya Singaravelu. "Thorough QT/QTc (TQT) study." International Journal of Basic & Clinical Pharmacology 10, no. 8 (2021): 1019. http://dx.doi.org/10.18203/2319-2003.ijbcp20212933.

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With a number of drugs entering the market, cardiac safety remains a cause of major concern for the regulatory authorities, before approval. The incidence of drug induced arrhythmia with non-cardiovascular drugs is low, however the result is fatal, hence much focus is being given to assess the pro-arrhythmic potential of a drug. The arrhythmogenic risk of the drug is higher if the patient is on polypharmacy or has other risk factors such as an electrolyte imbalance or an underlying structural heart disease. QT prolongation can be either due to congenital causes such as Long QT syndromes (LQTS)
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Asatryan, Babken, Marina Rieder, Alessandro Castiglione, and Katja E. Odening. "Arrhythmic risk during pregnancy and postpartum in patients with long QT syndrome." Herzschrittmachertherapie + Elektrophysiologie 32, no. 2 (2021): 180–85. http://dx.doi.org/10.1007/s00399-021-00757-4.

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AbstractCongenital long QT syndrome (LQTS) is a genetic disorder characterized by a prolonged QT interval in the surface electrocardiogram (ECG) that predisposes affected individuals to arrhythmic syncope, ventricular torsades-de-pointes, and sudden cardiac death at a young age. Investigations of large patient cohorts revealed sex-related differences in the LQTS phenotype. Adult women with LQTS are at higher risk for cardiac arrhythmias than are adult men with LQTS. Sex hormones are thought to play the primary role for these gender differences. Clinical experience and translational studies ind
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Probst, Vincent, Stéphanie Chatel, Jean-Baptiste Gourraud, and Hervé Le Marec. "Risk Stratification and Therapeutic Approach in Brugada Syndrome." Arrhythmia & Electrophysiology Review 1 (2012): 17. http://dx.doi.org/10.15420/aer.2012.1.17.

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Brugada syndrome (BrS) is a clinical entity characterised by an incomplete right bundle branch block associated with an ST segment elevation in the right precordial leads and a risk of ventricular arrhythmia and sudden death in the absence of structural abnormalities. Patients with a personal history of sudden death have an annual arrhythmia risk of recurrence as high as 10 %. Similarly, the presence of syncope is consistently associated with an increased arrhythmic risk. This risk can be estimated at about 1.5 % per year. The risk is lower in asymptomatic patients. Regarding the relatively hi
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Koplan, B. A., and W. G. Stevenson. "Sudden arrhythmic death syndrome." Heart 93, no. 5 (2007): 547–48. http://dx.doi.org/10.1136/hrt.2006.108134.

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Alharbi, Mishari, Islam Ibrahim, Sultan Alzahrani, et al. "Lifestyle Modification and Medical Treatment of Long QT Syndrome." JOURNAL OF HEALTHCARE SCIENCES 04, no. 05 (2024): 195–200. http://dx.doi.org/10.52533/johs.2024.40501.

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Long QT Syndrome (LQTS) is a genetic or acquired cardiac disorder characterized by a prolonged QT interval on an electrocardiogram, predisposing individuals to life-threatening arrhythmias and sudden cardiac death. Management of LQTS involves both lifestyle modifications and pharmacological treatments to mitigate risks and enhance patient outcomes. Lifestyle interventions include avoiding strenuous physical activities, especially swimming, and managing emotional stress to prevent arrhythmic triggers. Dietary modifications, such as maintaining adequate levels of potassium and magnesium, are ess
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De Nigris, Angelica, Angela Pepe, Giangiacomo Di Nardo, et al. "Brugada Pattern in a Child with Severe SARS-CoV-2 Related Multisystem Inflammatory Syndrome." Pediatric Reports 13, no. 3 (2021): 504–10. http://dx.doi.org/10.3390/pediatric13030058.

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This report presents the first case of Brugada pattern complicated by a supraventricular arrhythmia in a child with SARS-CoV-2 related Multisystem Inflammatory Syndrome in Children (MIS-C). A 7-year-old boy came to our Emergency Department with 7 days of abdominal pain and fever. MIS-C was diagnosed on the basis of the clinical, laboratory and instrumental tests. On admission, ECG showed type 1 Brugada pattern in the right precordial leads. During hospitalization the onset of supraventricular arrhythmias complicated the clinical picture. This case underlines management complexity of supraventr
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Vásquez, Javier Pinos, Tiago Luiz Luz Leiria, Marcelo Lapa Kruse, and Gustavo Glotz de Lima. "Early Repolarization Pattern and Idiopatic Sustained Monomorphic Ventricular Tachycardia: an Infrequent Combination." Journal of Cardiac Arrhythmias, no. 2 (May 22, 2021): 57–62. http://dx.doi.org/10.24207/jca.v34i2.3443.

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Malignant early repolarization as cause of sudden death has taken on great importance in recent years. It has been described as an entity capable of producing ventricular arrhythmic events, causing from episodes of syncope to sudden cardiac death. Ventricular fibrillation is the typical arrhythmia in these patients, with no clear relationship to date with idiopathic monomorphic ventricular tachycardia. Electrocardiographic markers related to the development of arrhythmic events in early repolarization syndrome have been described. They seem not only related to the development of ventricular fi
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Dissertations / Theses on the topic "Arrhythmic syndrome"

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Raju, Hariharan. "Sudden arrhythmic death syndrome : underlying cardiological, pathological and genetic aetiology." Thesis, St George's, University of London, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.656852.

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BACKGROUND: Sudden arrhythmic death syndrome (SADS) describes sudden, premature and unexpected deaths, which remain unexplained despite autopsy. Approximately one-third of SADS relates to inherited conditions such as Brugada syndrome (BrS), long QT syndrome (LQT) and catecholaminergic polymorphic ventricular tachycardia (CPVT). AIMS: We aimed to evaluate clinical utility for novel histopathological and genetic molecular investigation following SADS. METHODS: SADS cases were investigated by: [1] morphometric analysis of picrosirius-red histological myocardial staining; [2] a novel enrichment (F
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Matthews, Gareth David Kingsley. "The rate-dependence of pro-arrhythmic properties in murine SCN5A+/- hearts modeling the Brugada syndrome." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648741.

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Сміян, Олександр Іванович, Александр Иванович Смиян, Oleksandr Ivanovych Smiian та ін. "Вегетативная регуляция сердечного ритма у детей с малыми аномалиями серца". Thesis, Издательство СумГУ, 2010. http://essuir.sumdu.edu.ua/handle/123456789/6127.

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Behr, Elijah Raphael. "Sudden arrhythmic death syndrome : an epidemiological and pathological phenomenon and its clinical and genetic implications." Thesis, University of London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416571.

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Goldsworthy, Christopher. "Genetic testing for sudden arrhythmic death syndrome and the coroners' system of England and Wales." Thesis, Cardiff University, 2016. http://orca.cf.ac.uk/97980/.

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The author of this thesis examines how SADS is made and remade within interdisciplinary professional practice. Whilst recent sociological scholarship has followed the discourse of ‘molecularization’ when examining the construction of biomedical categories, I instead place the genetic as part of a broader clinical and medico-legal system. Whilst it is accepted that there are genetic aspects of SADS this does not reduce the usefulness of other disciplinary explanations in practice. This thesis is situated around the molecular autopsy, a technology simultaneously employed to identify the cause of
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Papadakis, Michael. "Sudden arrhythmic death syndrome in the UK : the experience of a tertiary cardiogenetics centre in South London." Thesis, King's College London (University of London), 2014. http://kclpure.kcl.ac.uk/portal/en/theses/sudden-arrhythmic-death-syndrome-in-the-uk(6b89384a-1ba9-42a7-bc44-c58d65d2461a).html.

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Background: In a proportion of sudden cardiac deaths (SCD), no structural pathology can be identified and a diagnosis of sudden arrhythmic death syndrome (SADS) is advocated. Recognition of SADS is important since a significant proportion is attributed to inherited cardiac conditions, and evaluation of relatives may identify individuals at risk. Aims: The aims of the thesis are: To investigate the magnitude of SADS in young (≤35 years) and athletic individuals; To investigate the impact of expert cardiac pathology; To evaluate a large cohort of SADS families to ascertain: 1.The diagnostic yiel
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Bakshi, Ruchika. "Asymptomatic long QT syndrome : a qualitative exploration of adolescent and parent experiences of living with the risk of a sudden arrhythmic death." Thesis, University of London, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.583261.

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The sudden death of a' child is a risk that families with Long O'T syndrome face. Preliminary research has shown that young people and their families with both Long O'T syndrome and asymptomatic Long O'T syndrome are at risk of developing psychological difficulties. To date however, the psychological processes that mediate the challenges associated with Long O'I syndrome within the context of adolescent development have not been fully explored. The present study conducted in-depth individual interviews with five young people aged between 12 and 15 years with a diagnosis of asymptomatic Long O'
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Gray, Belinda Ruth. "Clinical and Genetic Studies in Inherited Arrhythmia Syndromes." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/15994.

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Inherited arrhythmia syndromes, or “ion channelopathies”, is a term encompassing a number of different diseases including long QT syndrome (LQTS), catecholaminergic polymorphic ventricular tachycardia (CPVT), Brugada syndrome (BrS), short QT syndrome (SQTS) and idiopathic ventricular fibrillation (IVF). In inherited arrhythmia syndromes, ventricular arrhythmias originate due to abnormalities with intracellular ion channels, predominantly involving potassium, sodium and calcium handling, due to genetic mutations in genes encoding for channel proteins. The cellular abnormalities in these conditi
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Martin, Claire Adriana. "Mechanisms of arrhythmogenesis in a murine model of Brugada syndrome." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648347.

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Cross, Stephen. "Phenotypic heterogeneity in the Romano-Ward syndrome." Thesis, University of Aberdeen, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295289.

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The Romano-Ward syndrome is a familial disorder inherited as an autosomal dominant trait, the gene of which has been linked to the HLA locus on chromosome 6 or the Harvey-ras oncogene on chromosome 11. The genetic linkage, left and right ventricular function, and the integrity of the baroreflex were studied in subjects from two unrelated families with this condition. Neither family showed genetic linkage to either the HLA locus or to the Harvey-ras gene. Blood pool tomography was used to assess left and right ventricular function: left ventricular wall motion abnormalities were seen only in on
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Books on the topic "Arrhythmic syndrome"

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G, Benditt David, and Benson D. Woodrow, eds. Cardiac preexcitation syndromes: Origins, evaluation, and treatment. Nijhoff, 1986.

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Schwartz, Peter J. The long QT syndrome. Futura Pub. Co., 1997.

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Charles, Antzelevitch, ed. The Brugada syndrome. Futura Pub. Co., 1999.

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Clinical approach to sudden cardiac death syndromes. Springer, 2010.

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Ramrakha, Punit, and Jonathan Hill, eds. Arrhythmias. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199643219.003.0010.

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The cardiac conduction system 478Bradycardia: general approach 480Sinus bradycardia 482Sinus pause 482Sick sinus syndrome 482Atrioventricular block 483Bundle branch block 484Tachycardia: general approach 486Tachycardia: emergency management 488Tachyarrhythmias: classification 490ECG diagnosis of tachyarrhythmias 492Supraventricular tachycardia 494...
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Long Qt Syndrome An Issue Of Cardiac Electrophysiology Clinics. W.B. Saunders Company, 2012.

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QT Prolongation and ventricular arrhythmias. New York Academy of Sciences, 1992.

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Brugada, Pedro, Ramon Brugada, Charles Antzelevitch, and Joseph Brugada. Brugada Syndrome: From Bench to Bedside. Wiley & Sons, Limited, John, 2007.

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Brugada, Pedro, Ramon Brugada, Charles Antzelevitch, and Joseph Brugada. Brugada Syndrome: From Bench to Bedside. Wiley & Sons, Incorporated, John, 2008.

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Brugada, Pedro, Ramon Brugada, Charles Antzelevitch, and Joseph Brugada. Brugada Syndrome: From Bench to Bedside. Wiley & Sons, Incorporated, John, 2008.

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Book chapters on the topic "Arrhythmic syndrome"

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Sarquella-Brugada, Georgia, Oscar Campuzano, and Josep Brugada. "Sudden Arrhythmic Death Syndrome and Ventricular Tachycardia in Children." In Pediatric and Congenital Cardiology, Cardiac Surgery and Intensive Care. Springer London, 2023. http://dx.doi.org/10.1007/978-1-4471-4999-6_138-2.

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Monopoli, Giulia, Mohammad Javad Sadeghinia, Eivind Westrum Aabel, et al. "Arrhythmic Mitral Valve Syndrome: Insights from Left Ventricular End-Systolic Shape Analysis." In Lecture Notes in Computer Science. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-94559-5_3.

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Parikh, Milind G. "Arrhythmia Syndromes." In Cardiac Electrophysiology. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-75326-9_9.

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Kean, Adam C., and Prince J. Kannankeril. "Genetic Arrhythmia Syndromes." In Pediatric Cardiology. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-07563-6_47.

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Kean, Adam C., and Prince J. Kannankeril. "Genetic Arrhythmia Syndromes." In Pediatric Cardiology. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-030-42937-9_47-1.

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Caligari, Silvia, Vincenzo Randazzo, Carla Giustetto, Fiorenzo Gaita, and Eros Pasero. "ECG Images Analysis for the Prediction of Fatal Arrhythmic Events in Patients with Brugada Syndrome Based on a Vision Transformer Model." In Smart Innovation, Systems and Technologies. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-0994-9_2.

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Musat, Dan L., Delia Cotiga, Walter Pierce, and Aysha Arshad. "Arrhythmias Complicating Acute Myocardial Infarction—Bradyarrhythmias." In Acute Coronary Syndrome. Springer London, 2008. http://dx.doi.org/10.1007/978-1-84628-869-2_16.

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Johnson, Jonathan N., and Michael J. Ackerman. "Long QT Syndrome." In Management of Cardiac Arrhythmias. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-161-5_19.

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Cotiga, Delia, Tina Sichrovsky, Kataneh Maleki, and Suneet Mittal. "Ventricular Arrhythmias in Patients After Myocardial Infarction." In Acute Coronary Syndrome. Springer London, 2008. http://dx.doi.org/10.1007/978-1-84628-869-2_18.

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Chitnis, Nishad, and B. Hygriv Rao. "Arrhythmias in acute coronary syndrome." In Acute Coronary Syndromes. CRC Press, 2020. http://dx.doi.org/10.1201/9780429025396-6.

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Conference papers on the topic "Arrhythmic syndrome"

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Malidze, David, Rishu Bansal, Marina Noniashvili, Natia JoJua, Tinatin Gognadze, and Mreisa Ahmad. "Cardiac Arrhythmias in Patients with Myocarditis in the Post-COVID-19 Period." In Socratic Lectures 8. University of Lubljana Press, 2023. http://dx.doi.org/10.55295/psl.2023.ii1.

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To date, several cardiotropic viruses have been implicated as causes of myocarditis. The most detected are parvovirus B19, and human herpes virus 6. Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) are the respiratory viruses that have recently triggered the unprecedented pandemic process. The involvement and impact of these viruses in viral cardiovascular disease are under study. Despite extensive research into the pathological mechanisms of viral infections of the cardiovascular system, our knowledge regarding their treatment and management is still incomplete. A condition caused
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Lee, Sharen, Jiandong Zhou, Konstantinos P. Letsas, et al. "Pairwise Feature Interactions to Predict Arrhythmic Risk of Brugada Syndrome." In 2021 Computing in Cardiology (CinC). IEEE, 2021. http://dx.doi.org/10.23919/cinc53138.2021.9662913.

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Toh, T., C. Coyne, K. Stewart, A. Muir, and D. McCall. "33 The diagnostic yield of screening first-degree relatives in sudden arrhythmic death syndrome." In Irish Cardiac Society Annual Scientific Meeting & AGM, Thursday October 5th – Saturday October 7th 2017, Millennium Forum, Derry∼Londonderry, Northern Ireland. BMJ Publishing Group Ltd and British Cardiovascular Society, 2017. http://dx.doi.org/10.1136/heartjnl-2017-ics17.33.

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Caligari, Silvia, Vincenzo Randazzo, Fiorenzo Gaita, Carla Giustetto, Michele Millesimo, and Eros Pasero. "A neural network approach for the prediction of arrhythmic events in patients with Brugada syndrome via ECG features analysis." In 2024 IEEE 22nd Mediterranean Electrotechnical Conference (MELECON). IEEE, 2024. http://dx.doi.org/10.1109/melecon56669.2024.10608666.

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Bari, Vlasta, Beatrice De Maria, Giulia Girardengo, et al. "Multiscale Complexity Analysis of Short QT Interval Variability Series Stratifies the Arrhythmic Risk of Long QT Syndrome Type 1 Patients." In 2018 Computing in Cardiology Conference. Computing in Cardiology, 2018. http://dx.doi.org/10.22489/cinc.2018.053.

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Bari, Vlasta, Giulia Girardengo, Beatrice De Maria, et al. "Frequency Domain Heart Period and QT Interval Variability Markers Are Linked to Arrhythmic Risk in Long QT Syndrome Type 2." In 2020 Computing in Cardiology Conference. Computing in Cardiology, 2020. http://dx.doi.org/10.22489/cinc.2020.065.

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Carron, J., C. McGorrian, K. Haverin, et al. "23 The sads heart of the matter: a review of the sudden arrhythmic death syndrome (SADS) biobank in the mater misericordiae university hospital; the cornerstone of a national strategy." In Irish Cardiac Society Annual Scientific Meeting & AGM, Thursday October 17th – Saturday October 19th 2019, Galway, Ireland. BMJ Publishing Group Ltd and British Cardiovascular Society, 2019. http://dx.doi.org/10.1136/heartjnl-2019-ics.23.

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Aragon, Jose Gerald, Mohammed A. Rattu, Alex Gechlik, Nicholas Coan, James A. Espinosa, and Alan Lucerna. "Brief Review: The Refeeding Syndrome." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.88_2024.

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Here we briefly review the definition, epidemiology, pathophysiology, clinical risk scenarios, prevention and treatment of the refeeding syndrome. Refeeding syndrome is a condition which arises when a severely malnourished individual has been restarted with increased nutrition in a relatively short period of time. The refeeding process can lead to shifts in a number of fluids and electrolytes. The hallmark feature is hypophosphatemia. The true incidence of refeeding syndrome is unknown. This is said to be partly due to the lack of a universally accepted definition. Populations that are likely
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Pocevski, Božidar, Predrag Pocevski, and Marija Pocevski. "Health and social aspects of corona virus disease 2019 pandemic and post-COVID society." In Proceedings of the International Congress Public Health - Achievements and Challenges. Institute of Public Health of Serbia "Dr Milan Jovanović Batut", 2024. http://dx.doi.org/10.5937/batutphco24132p.

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It was proven so far, that Severe Acute Respiratory Syndrome induced by Corona Virus 2 (SARSCoV-2) is highly pathogenic virus, which spread rapidly. The spectrum of COVID-19 ranges from asymptomatic infection to severe respiratory failure. COVID-19 caused by SARS-CoV-2 is characterized primary as pulmonary disease. After three years of pandemic and several variants of corona viruses, in scientific circles and, more significant, in practice it turned out that SARS-CoV-2 shows neurological and cardiovascular manifestations. In practice we are facing with post-covid and long-covid symptoms. There
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Gupta, Raghav, Yara Assadi, Shaniece Nicole Lawrence, Erika Jeanie Pitsker, Michael Scott Jr Bickford, and Victoria Amber Saniko. "Potential Mechanisms for New Onset Atrial Fibrillation in COVID-19 Patients." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.201_2024.

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Introduction: This study shows the prevalence of New-Onset Atrial Fibrillation in COVID-19 patients and highlights the most prevalent explanatory pathologic theories for the correlation. Methods: The authors carried out a literature review over PubMed using the keywords “atrial fibrillation” and “long-term COVID.” 48 articles were reviewed. Articles relating to new onset of atrial fibrillation in COVID-19 patients were included while articles posted before 2020, not related to COVID and atrial fibrillation were excluded. Results: The prevalence of atrial fibrillation in COVID patients is often
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