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Relevant bibliographies by topics / Autoimmune response / Dissertations / Theses

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Dissertations / Theses on the topic 'Autoimmune response'

Author: Grafiati

Published: 4 June 2021

Last updated: 27 July 2025

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1

Diaz, J.-L. "The autoimmune response to insulin." Thesis, University of Southampton, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233286.

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2

Levy, Jeremy Bernard. "Specificity of the autoimmune response in Goodpasture's disease." Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394436.

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3

Berl, Sabina [Verfasser]. "Neuronal Response to Experimental Autoimmune Encephalomyelitis / Sabina Berl." Mainz : Universitätsbibliothek Mainz, 2020. http://d-nb.info/1203322933/34.

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4

Ross, Calum Neil. "Specificity of the automimmune response in Goodpasture's disease." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338853.

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5

Hammond, Linda Joyce. "Thyrocytes as effectors and targets of the autoimmune response." Thesis, Queen Mary, University of London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.251689.

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6

Anderson, Bradley Clinton. "Immunodominance of the CD8+ T-cell response in autoimmune diabetes." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ38568.pdf.

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7

Newton, Sonja Grace. "The response to heat shock protein 60 in autoimmune models." Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324991.

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8

Stanford, Marianne Michelle. "Immunoregulation in murine experimental autoimmune thyroiditis." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0020/MQ54961.pdf.

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9

Anwar, Mohammad Ashraful. "SPARC modulates the spinal cord neuroimmune response in experimental autoimmune encephalomyelitis." Thesis, University of British Columbia, 2014. http://hdl.handle.net/2429/46047.

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SPARC (Secreted Protein Acidic and Rich in Cysteine), a secreted glycoprotein, regulates proliferation, migration and differentiation. SPARC is highly expressed in glia and blood vessels during CNS development. SPARC expression is maintained in tissues undergoing rapid turnover and its expression is highly upregulated during injury or disease. SPARC’s modulatory activity in glia and endothelia during injury lead us to investigate the role of SPARC in an animal model of CNS inflammation and demyelination with known BBB dysfunction: Experimental Autoimmune Encephalomyelitis (EAE). We discovered

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10

Liberal, Rodrigo Gouveia Vasconcelos Rodrigues. "Effectors and regulators of cellular immune response in autoimmune liver disease." Doctoral thesis, Faculdade de Medicina da Universidade do Porto, 2011. http://hdl.handle.net/10216/63783.

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11

Emonts, M. "Polymorphisms in immune response genes in infectious diseases and autoimmune diseases." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2008. http://hdl.handle.net/1765/14316.

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12

Liberal, Rodrigo Gouveia Vasconcelos Rodrigues. "Effectors and regulators of cellular immune response in autoimmune liver disease." Tese, Faculdade de Medicina da Universidade do Porto, 2011. http://hdl.handle.net/10216/63783.

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13

Nicolson, Donald Magnus. "The immunosuppression of murine delayed-type hypersensitivity responses to renal antigens." Thesis, University of Strathclyde, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.278524.

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14

Dromey, James Anthony. "Lymphocytes and the autoimmune response to islet autoantigens in Type 1 diabetes." Thesis, King's College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400496.

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15

Yang, Jinghui. "Peripheral immune response in chronic relapsing experimental autoimmune encephalomyelitis in SJL mice." Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/laa/haart/vk/yang/.

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16

Ma, Yun. "Characterisation of antigens target of humoral and cellular immune response in autoimmune liver disease." Thesis, King's College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266254.

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17

Wyatt, Rebecca Clare. "Developing novel reagents for characterising the B-cell autoimmune response in type 1 diabetes." Thesis, University of Bristol, 2017. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.743009.

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18

Carroll, Kaitlin R. "Targeted T Cell Ablation in Autoimmune Disease:The Therapeutic Potential of DNA Damage Response Molecule Manipulation." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1544100593082292.

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19

Pilli, Deepti. "The Autoimmune T cell Response Against the Dopamine-2 Receptor in Movement and Psychiatric Disorders." Thesis, The University of Sydney, 2020. https://hdl.handle.net/2123/22465.

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Autoimmunity and immune dysregulation are associated with a subset of movement and psychiatric disorders. This paradigm is largely supported by the discovery of autoantibodies against neuronal antigens, like the dopamine-2 receptor (D2R). T cells are a prominent cell subset in the immune system, however, their role in these diseases is unknown. Herein, we identified and characterised D2R-specific T cells in movement and psychiatric disorders in children and adults. In children with suspected autoimmune or neurodevelopmental movement and psychiatric disorders (n=24), activated D2R-specific T ce

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20

Beaudoin, Danelle Rae. "T-cell signaling in response to altered myelin basic protein peptides /." Thesis, Connect to this title online; UW restricted, 2003. http://hdl.handle.net/1773/8535.

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21

Castrillon, Carlos. "Characterizing the antibody response at the single cell level with droplet microfluidics." Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCC141.

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Les anticorps sont des protéines en forme de Y, trouvées comme composant du sérum circulant, qui aident le système immunitaire à cibler et à répondre aux agents pathogènes et aux molécules étrangères, mais peuvent aussi contribuer à la maladie en réagissant aux protéines constitutives. Les anticorps sont produits par des Plasmocytes, qui les sécrètent dans la circulation. Parce qu'il n'y a pas de lien physique entre les plasmocytes et leurs anticorps sécrétés, la détection d'anticorps spécifiques d’un antigène est problématique. Dans cette thèse, j'explore l'utilisation de la microfluidique en

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22

Forni, D. "HOW NATURAL SELECTION SHAPED DIVERSITY AT IMMUNE RESPONSE GENES AND AUTOIMMUNE RISK ALLELES DURING MAMMALIAN EVOLUTION." Doctoral thesis, Università degli Studi di Milano, 2015. http://hdl.handle.net/2434/279118.

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ABSTRACT INTRODUCTION Genetic diversity is generated by a combination of different evolutionary processes, including mutation, genetic drift, migration, and natural selection.It is well known that natural selection acts on a specific locus\variant, whereas demographic effects act on all loci in the same way; also the selection is expected to be focused on genomic positions that have a functional role. Importantly, the selected variants targeted by selection may not only have a functional role but can correlate with predisposition or protection to some specific diseases. They can therefore

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23

Buhler, Marc McWilliam. "Genetics of the immune cell receptors TCRB and CCR5 in human disease /." Connect to full text, 2003. http://setis.library.usyd.edu.au/adt/public_html/adt-NU/public/adt-NU20040405.141449/index.html.

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24

Ronkainen, M. (Matti). "Characterization of the humoral immune response to the beta-cell antigens insulin and glutamic acid decarboxylase in preclinical and clinical type 1 diabetes." Doctoral thesis, University of Oulu, 2005. http://urn.fi/urn:isbn:9514277805.

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Abstract The characteristics of humoral immunity have been proposed to reflect the bias between two T helper (Th) lymphocyte subsets: Th1 cells, which activate cell-mediated immunity, and Th2 cells, which mediate humoral immunity. The present study aimed to characterize the humoral immunity to beta-cell autoantigens insulin and glutamic acid decarboxylase (GAD65) in preclinical and clinical type 1 diabetes. Insulin antibodies were analyzed in pregnant women with or without type 1 diabetes and their newborn infants and in prediabetic children. Epitope or/and isotype-specific GAD65 antibodies (

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25

Groom, Joanna Ruth School of Medicine UNSW. "Loss of immune regulatory checkpoints in BAFF transgenic mice." Awarded by:University of New South Wales. School of Medicine, 2006. http://handle.unsw.edu.au/1959.4/27281.

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Multiple checkpoints control the survival and activation of auto-reactive B cells. The discovery of the TNF family cytokine BAFF has been crucial to understanding peripheral B cell tolerance mechanisms. Homeostatic levels of BAFF are tightly regulated to maintain tolerance in the periphery. Chronically increased levels of BAFF lead to the survival of autoreactive B cells. Autoimmune patients display elevated serum BAFF levels. BAFF Tg mice model this situation with systemically high levels of BAFF and the subsequent development of two separate but related autoimmune syndromes; systemic lupus e

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26

Sisay, Sofia. "GPR55 as a novel target in disease control of multiple sclerosis." Thesis, Queen Mary, University of London, 2013. http://qmro.qmul.ac.uk/xmlui/handle/123456789/27210.

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Multiple sclerosis (MS) is a neurodegenerative disease associated with immune attack of the central nervous system (CNS) leading to neuronal and axonal loss. This affects neurotransmission accumulating residual disability and the development of neurological signs such as spasticity. Numerous studies have reported a beneficial role of cannabinoids in alleviating symptoms associated with neurological damage. The endocannabinoid system has been shown to control experimental spasticity in experimental autoimmune encephalomyelitis (EAE) an animal model of multiple sclerosis (MS). The orphan G-prote

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27

Sherman, Samantha. "The Response of the Glycerophosphocholine Metabolite Lipidome to Experimental Autoimmune Encephalomyelitis and Cycling Female Sex Hormones in the Hippocampus and Temporal-Parietal-Entorhinal Cortex of Female Mice." Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/34588.

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Recently, several glycerophosphocholine biomarkers for multiple sclerosis were discovered in serum, plasma, and cerebrospinal fluid; little is known, however, about brain glycerophosphocholine metabolism during multiple sclerosis despite evidence that lysophosphocholines can elicit demyelination experimentally. Using a lipidomics approach, glycerophosphocholine metabolites in the hippocampus and temporal-parietal-entorhinal cortex of female C57BL/6J mice subjected to experimental autoimmune encephalomyelitis (a mouse model of multiple sclerosis) were quantified and compared to metabolite level

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28

Powell, Nicole Damico. "Immunomodulation of experimental autoimmune encephalomyelitis targeting the autoreactive T cell and the cytokine macrophage migration inhibitory factor /." Columbus, Ohio : Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1141052089.

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29

Hecker, Michael [Verfasser], Reinhard [Akademischer Betreuer] Guthke, Raimund W. [Akademischer Betreuer] Kinne, and Ronald [Akademischer Betreuer] Westra. "Integrative Modeling of Transcriptional Regulation in Response to Autoimmune Desease Therapies / Michael Hecker. Gutachter: Reinhard Guthke ; Raimund W. Kinne ; Ronald Westra." Jena : Thüringer Universitäts- und Landesbibliothek Jena, 2011. http://d-nb.info/1016367775/34.

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30

Scarpa, Carlotta. "The immunological response to breast implant: the role of cells and cytokines in the periprosthetic capsule and their involvement in the onset of the autoimmune diseases." Doctoral thesis, Università degli studi di Padova, 2013. http://hdl.handle.net/11577/3426173.

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Since their discovery breast prostheses have been criticized for being responsible for triggering systemic autoimmune disease. The presence of breast implants causes a natural foreign body reaction characterized by the infiltration of macrophages and T-cells. In order to understand which immunological pathways could be responsible for giving rise to, and the development of, connective tissue disease such as systemic sclerosis, I considered the cells and cytokines involved, focusing on the relationship between tissue growth factor-β, interleukin (IL)-1, IL-6 and T helper 17 and/or T regulatory

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31

Campos, Ana Margarida Ferreira. "Lipidomic analysis of mesenchymal cells candidates for cell therapy." Master's thesis, Universidade de Aveiro, 2015. http://hdl.handle.net/10773/15275.

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Mestrado em Bioquímica - Métodos Biomoleculares<br>Mesenchymal stromal cells are adult stem cells found mostly in the bone marrow. They have immunosuppressive properties and they have been successfully applied as biological therapy in several clinical trials regarding autoimmune diseases. Despite the great number of clinical trials, MSCs’ action is not fully understand and there are no identified markers that correlate themselves with the immunomodulatory power. A lipidomic approach can solve some of these problems once lipids are one of the major cells’ components. Therefore, in this study

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32

Buhler, Marc McWilliams. "Genetics of the immune cell receptors TCRB and CCR5 in human disease." University of Sydney, 2003. http://hdl.handle.net/2123/601.

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Abstract Early in the evolution of the vertebrates it is thought that two genomic duplications occurred, providing a basis for the evolution in body plan and neural crest of very early vertebrates and substantive material for further evolution of various gene families such as those making up a number of components of the adaptive vertebrate immune system. While the bony fish possibly had another, genome duplications are not generally a feature of vertebrate evolution and indeed the appearance of an antigen-adaptive immune recognition system may have served to limit the size that various verteb

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33

Mathieson, Peter William. "Role of T lymphocytes in autoimmune responses." Thesis, University of Cambridge, 1992. https://www.repository.cam.ac.uk/handle/1810/251527.

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34

Zhao, Ming-Hui. "Characterisation of autoimmune responses in systemic vasculitis." Thesis, Anglia Ruskin University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.259510.

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35

Wållberg, Maja. "Modulation of immune responses in experimental autoimmune encephalomyelitis /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-335-3/.

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36

Deng, Jun, and 鄧軍. "Leptin modulates T cells responses in autoimmune arthritis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208601.

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Leptin, a protein hormone encoded by obese (ob) gene, is mainly produced by adipocytes. Leptin plays an important role in regulating neuroendocrine function and energy metabolism. As a cytokine, leptin is involved in modulating the hematopoiesis and lymphopoiesis. Although leptin has been found to promote T cells activation, it is largely unclear whether and how leptin regulates T cell differentiation and function. Leptin has been associated with disease severity in rheumatoid arthritis (RA). Elevated leptin levels have been detected in the sera and synovial fluid of active RA patients. Th1

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37

Richards, Ian. "Autologous mixed lymphocyte reaction in myasthenia gravis." Thesis, University of Bath, 1987. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.379569.

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38

Murera, Uwanyirigira Diane. "Study of lymphocyte autophagy in normal and autoimmune responses." Thesis, Strasbourg, 2016. http://www.theses.fr/2016STRAJ068.

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L’autophagie est un processus catabolique lié aux lysosomes, essentiel à la l’homéostasie cellulaire notamment dans les lymphocytes. Elle est impliquée dans la pathogenèse de nombreuses maladies et pourrais jouer un rôle dans le développement de maladies auto-immunes. Nous avons voulu étudier son rôle in vivo dans les lymphocytes B et T. Nous avons généré des souris déficientes en autophagie spécifiquement dans ces cellules et montré que l’autophagie n’est pas essentielle au développement des LB, mais que dans un contexte auto-immun la persistance de plasmocytes et la production d’autoanticorp

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39

Cunha, Andreia. "Regulation of autoimmune neuroinflammation by stress-responsive genes." Doctoral thesis, Universidade Nova de Lisboa. Instituto de Tecnologia Química e Biológica, 2012. http://hdl.handle.net/10362/8588.

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Dissertation presented to obtain the Ph.D degree in Biology<br>Inflammation is a protective response generated by innate immune cells, upon infection and/or tissue injury, and aims at clearing the source of infection or noxious stimuli. It is required for activation of adaptive immunity. Inflammation has several layers of regulation in order to minimize the degree of tissue damage. Antigen-presenting cells (APC) constitute one layer of regulation as they initiate the activation of T helper (TH) cells, bridging innate and adaptive immunity. The TH cell response generated is usually protective,

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40

Hall, Håkan. "T cell responses and NK cell function in experimental autoimmune diabetes /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-152-0/.

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41

Narendran, Partheepan. "Immune responses to proinsulin in type 1 diabetes mellitus." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325704.

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42

Walker, Lucy S. K. "Control of T lymphocyte responses by CD95-mediated apoptosis." Thesis, University of Bath, 1997. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.389953.

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43

Schulz, Kerstin Ingrid. "Modulation of Th1 and Th2 type immune responses." Thesis, University of Glasgow, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390690.

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44

Lundholm, Peter. "Immune and autoimmune responses to HIV-1 in mucosa and other tissues /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3679-X/.

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45

Evans, Teresa Ann. "In Vivo Observations of Resident Microglia and Blood Derived Macrophages in the Brain and Spinal Cord." Case Western Reserve University School of Graduate Studies / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=case1396399768.

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46

Young, A. "The regulation of T cell responses by interleukin-27 : implications for autoimmune disease." Thesis, Queen's University Belfast, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.677857.

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Here presented is a detailed account of two novel findings, as well as further clarification of another known effect of the heterodimeric cytokine IL-27 on T cell biology. The first novel finding describes a potent anti-inflammatory mechanism through the suppression of T cell derived GM-CSF production via IL-27 stimulation. While IL-27 suppressed GM-CSF production by Th1 cells in vitro, it did not influence the production of GM-CSF in committed Th17 cells. il2T'- T cells produced more GM-CSF in response to Toxoplasma gondii infection, suggesting IL-27 signalling regulates T cell derived GM-CSF

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47

BENEDETTI, Sabrina. "HHV6 INFECTION OF THYROID CELLS MAY PROMOTE AUTOIMMUNE RESPONSES LEADING TO HASHIMOTO'S THYROIDITIS." Doctoral thesis, Università degli studi di Ferrara, 2012. http://hdl.handle.net/11392/2388766.

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Chronic lymphocytic thyroiditis, also known as Hashimoto's thyroiditis (HT) is the most common cause of hypothyroidism in the Western world. It is an autoimmune disease, where thyroid follicles are gradually destroyed by a variety of cell and antibody mediated immune processes. The involvement of viral infections has been frequently suggested as a major environmental factor, but no conclusive data are available. We analyzed fine needle biopsies from 34 HT patients for the presence and the transcriptional state of human herpesvirus 6 (HHV6). Controls, represented by 28 patients with benign foll

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48

Yanamandra, Kiran. "Studies of in vivo prostate amyloidosis and autoimmune responses towards amyloid structures in neurodegeneration." Doctoral thesis, Umeå universitet, Institutionen för medicinsk kemi och biofysik, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-37561.

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By using multidisciplinary analysis of CA inclusions in prostate glands of patients diagnosed with prostate cancer, we have revealed that their major components are the amyloid forms of S100A8 and S100A9 proteins associated with numerous inflammatory conditions and types of cancer. We have demonstrated that material closely resembling CA can be produced from S100A8/A9 in vitro and shows the characters of amyloids. This process is facilitated by calcium or zinc, both of which are abundant in ex vivo inclusions. These observations were supported by computational analysis of the S100A8/A9 calcium

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49

Ma, Liang. "Induction and regulation of autoimmune responses by dendritic cells upon interaction with dying cells in murine models." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B3554756X.

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50

Muller, Ilaria. "Autoimmune responses to thyroid/breast shared antigens to develop novel and specific therapies and diagnostics." Thesis, Cardiff University, 2016. http://orca.cf.ac.uk/91002/.

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An association between breast cancer (BC) and thyroid autoimmunity (TA) has been frequently observed and several small-scale studies correlated the presence of thyroid peroxidase (TPO) autoantibodies (TPOAb) with an improved BC outcome. The presence of an immune response to shared thyroid/breast antigens has been hypothesized: both tissues express the sodium iodide symporter (NIS) and have a peroxidase activity: TPO in thyroid and lactoperoxidase (LPO) in breast. I have identified 3 possibilities: i) BC patients with TPOAb may also have autoantibodies to NIS (NISAb) ii) BC may express TPO iii)

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