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Journal articles on the topic 'Autoimmune response'

Author: Grafiati

Published: 4 June 2021

Last updated: 27 July 2025

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1

VEGA, MIGUEL A., RODERIC GUIGó, and TEMPLEF SMITH. "Autoimmune response in AIDS." Nature 345, no. 6270 (1990): 26. http://dx.doi.org/10.1038/345026a0.

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2

Ridgway, William M., Howard L. Weiner, and C. Garrison Fathman. "Regulation of autoimmune response." Current Opinion in Immunology 6, no. 6 (1994): 946–55. http://dx.doi.org/10.1016/0952-7915(94)90018-3.

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3

Voelker, R. "Autoimmune Response and CHF." JAMA: The Journal of the American Medical Association 281, no. 10 (1999): 889—b—889. http://dx.doi.org/10.1001/jama.281.10.889-b.

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Voelker, Rebecca. "Autoimmune Response and CHF." JAMA 281, no. 10 (1999): 889. http://dx.doi.org/10.1001/jama.281.10.889-jwm90001-3-1.

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5

P, Mathew. "Autoimmune Hepatitis-Profile and Response to Treatment in Indian Patients." Gastroenterology & Hepatology International Journal 4, no. 2 (2019): 1–6. http://dx.doi.org/10.23880/ghij-16000159.

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Introduction: Autoimmune hepatitis (AIH) can have varied manifestations, commonest presentation being as chronic liver disease. The data on the disease profile in India is scanty compared to the West. Aim: To study the clinical, biochemical, histological profile and response to treatment in patients with Auto-immune Hepatitis in Indian population. Methods: This is a Retrospective analysis of the twenty one (12 M = 57.1%; 9 F = 42.9%) patients diagnosed with AIH according to simplified criteria for diagnosis of AIH, in the last three years (2017-2019) in the department of medical gastroenterolo

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Fedoseyeva, Eugenia V., Feng Zhang, Patricia L. Orr, David Levin, Harry J. Buncke, and Gilles Benichou. "De Novo Autoimmunity to Cardiac Myosin After Heart Transplantation and Its Contribution to the Rejection Process." Journal of Immunology 162, no. 11 (1999): 6836–42. http://dx.doi.org/10.4049/jimmunol.162.11.6836.

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Abstract Allograft rejection is initiated by an immune response to donor MHC proteins. We recently reported that this response can result in breakdown of immune tolerance to a recipient self Ag. However, the contribution of this autoimmune response to graft rejection has yet to be determined. Here, we found that after mouse allogeneic heart transplantation, de novo CD4+ T cell and B cell autoimmune response to cardiac myosin (CM), a major contractile protein of cardiac muscle, is elicited in recipients. Importantly, CM is the autoantigen that causes autoimmune myocarditis, a heart autoimmune d

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Stergioti, Eirini Maria, Theodora Manolakou, Dimitrios T. Boumpas, and Aggelos Banos. "Antiviral Innate Immune Responses in Autoimmunity: Receptors, Pathways, and Therapeutic Targeting." Biomedicines 10, no. 11 (2022): 2820. http://dx.doi.org/10.3390/biomedicines10112820.

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Innate immune receptors sense nucleic acids derived from viral pathogens or self-constituents and initiate an immune response, which involves, among other things, the secretion of cytokines including interferon (IFN) and the activation of IFN-stimulated genes (ISGs). This robust and well-coordinated immune response is mediated by the innate immune cells and is critical to preserving and restoring homeostasis. Like an antiviral response, during an autoimmune disease, aberrations of immune tolerance promote inflammatory responses to self-components, such as nucleic acids and immune complexes (IC

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Stys, Peter K. "Multiple Sclerosis: Autoimmune Disease or Autoimmune Reaction?" Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 37, S2 (2010): S16—S23. http://dx.doi.org/10.1017/s0317167100022393.

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ABSTRACT:Multiple sclerosis (MS) is traditionally considered an autoimmune inflammatory demyelinating disease of the central nervous system (CNS) with much knowledge available to support this view. However, this characterization implies that the primary event is an aberrant immune response directed at CNS antigens, promoting inflammation and later driving progressive axo-glial degeneration. Trials with potent anti-inflammatory agents and detailed neuropathological studies raise questions about this sequence of events. This hypothetical paper argues that MS may be primarily a “cytodegenerative”

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Rita Korponay-Szabo, Ilma, Zsofia Simon-Vecsei, Luigina De Leo, and Tarcisio Not. "Gluten-dependent Intestinal Autoimmune Response." Current Pharmaceutical Design 18, no. 35 (2012): 5753–58. http://dx.doi.org/10.2174/138161212803530826.

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10

Hassfeld, Wolfgang, GÜNter Steiner, Andrea Studnicka-Benke, et al. "Autoimmune response to the spliceosome." Arthritis & Rheumatism 38, no. 6 (1995): 777–85. http://dx.doi.org/10.1002/art.1780380610.

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11

Insignares, Iván, Luis E. Rodríguez, Óscar Correa-Jiménez, et al. "Autoimmunity against cytokines: Double strike in autoimmune disease, a historical perspective." Biomédica 44, Sp. 2 (2024): 209–19. https://doi.org/10.7705/biomedica.7570.

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Autoimmune responses are characterized by the development of antibodies and the activation of T lymphocytes against self-antigens. This leads to an effector immuneresponse against tissues expressing antigens, which are later recognized by the host immune system.Host antigens attacked by antibodies are called “autoantigens” and are of different kinds, including receptors, enzymes, and channel proteins. The autoimmune response is potentiated by cytokines that mediate the activation of Th1, Th2, or Th17 lymphocytes. The released cytokines can also be recognized as autoantigens, meaning they can b

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Rajic, Miodrag, Snezana Djurica, Dragoslav Milosevic, and Natasa Markovic. "Autoimmune haemopoietic disturbances simultaneous with autoimmune thyroid diseases." Srpski arhiv za celokupno lekarstvo 133, Suppl. 1 (2005): 52–54. http://dx.doi.org/10.2298/sarh05s1052r.

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Out of 362 patients with diagnosis of autoimmune haemopathy treated in eight-year period at the Hematology centre, concomitant autoimmune thyreoideopathy was confirmed in 22 (5.52%). The most frequent was simultaneous manifestation of pernicious anemia and autoimmune thyreoiditis- Phenomenon was less frequently observed in patients with immune thrombocytopenic purpura and Graves disease. In the subgroup of patients with autoimmune hemolytic anemia, there was no evidence of simultaneous autoimmune thyreoideopathy. General opinion is that etiopathogenesis of these immunological disorders does no

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13

Spencer, Chantal Y., Jennifer Millman, Keila Veiga, and Alfin G. Vicencio. "Airway Autoimmune Inflammatory Response (AAIR) Syndrome: An Asthma-Autoimmune Overlap Disorder?" Pediatrics 141, no. 3 (2018): e20170138. http://dx.doi.org/10.1542/peds.2017-0138.

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14

Seder, R. A., B. L. Kelsall, and D. Jankovic. "Differential roles for IL-12 in the maintenance of immune responses in infectious versus autoimmune disease." Journal of Immunology 157, no. 7 (1996): 2745–48. http://dx.doi.org/10.4049/jimmunol.157.7.2745.

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Abstract IL-12 has been shown to be important in the generation of a functional Th1 response in animal models of both infectious and autoimmune disease. Furthermore, the role of IL-12 in the maintenance of the immune responses in these diseases is now emerging. Herein, we discuss the idea that memory responses for certain infections may be IL-12 independent, whereas memory responses for specific autoimmune diseases still require IL-12 to maintain a pathogenic response.

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Rosloniec1, Edward F., David D. Brand, Linda K. Myers, et al. "Induction of Autoimmune Arthritis in HLA-DR4 (DRB1*0401) Transgenic Mice by Immunization with Human and Bovine Type II Collagen." Journal of Immunology 160, no. 6 (1998): 2573–78. http://dx.doi.org/10.4049/jimmunol.160.6.2573.

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Abstract Although associations between the expression of particular HLA genes and the susceptibility to specific autoimmune diseases has been known for some time, the role that these HLA molecules play in the autoimmune response is unclear. Through the establishment of a chimeric HLA-DR/I-E transgene, we have examined the function of the rheumatoid arthritis (RA) susceptibility allele HLA-DR4 (DRB1*0401) in presenting antigenic peptides derived from the model Ag, type II collagen (CII), and in mediating an autoimmune response. As a transgene, the chimeric DR4 molecule conferred susceptibility

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Nazneen, Shahela, Mohammad Imtiaj Mahbub, and Zulfia Zinat Choudhury. "Steroid Response in Autoimmune Hemolytic Anemia." Journal of Shaheed Suhrawardy Medical College 13, no. 1 (2022): 41–45. http://dx.doi.org/10.3329/jssmc.v13i1.60930.

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Background: Auto-immune hemolytic anemia is an uncommon but not rare disorder. Steroid is the best and commonly used drug for this disorder because it is cheap, easily available and less toxic. But there are a few studies on evaluation of response of steroids in auto-immune hemolytic anemia in our country. Objectives: The purpose of the study was to find out the mean duration for attaining response with prednisolone at the dose of 1 mg/kg body weight, to categorize the patients according to response criteria and to find out the adverse effects due to prednisolone. Methodology: This was a longi

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17

Waite, Janelle C., and Dimitris Skokos. "Th17 Response and Inflammatory Autoimmune Diseases." International Journal of Inflammation 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/819467.

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The proinflammatory activity of T helper 17 (Th17) cells can be beneficial to the host during infection. However, uncontrolled or inappropriate Th17 activation has been linked to several autoimmune and autoinflammatory pathologies. Indeed, preclinical and clinical data show that Th17 cells are associated with several autoimmune diseases such as arthritis, multiple sclerosis, psoriasis, and lupus. Furthermore, targeting the interleukin-17 (IL-17) pathway has attenuated disease severity in preclinical models of autoimmune diseases. Interestingly, a recent report brings to light a potential role

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18

Zhang, Ming, and Michael C. Carroll. "Natural antibody mediated innate autoimmune response." Molecular Immunology 44, no. 1-3 (2007): 103–10. http://dx.doi.org/10.1016/j.molimm.2006.06.022.

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19

Davies, A. J. S. "Immunological tolerance and the autoimmune response." Autoimmunity Reviews 7, no. 7 (2008): 538–43. http://dx.doi.org/10.1016/j.autrev.2008.04.007.

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20

IBORRA, A., J. R. PALACIO, P. MARTÍNEZ, and Z. ULCOVA-GALLOVA. "Autoimmune Response in Women with Endometriosis." American Journal of Reproductive Immunology 44, no. 4 (2000): 236–41. http://dx.doi.org/10.1111/j.8755-8920.2000.440408.x.

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21

Martynenko, A. V., S. V. Magaeva, L. A. Basharova, and M. V. Martynenko. "Autoimmune response in experimental hippocampal pathology." Bulletin of Experimental Biology and Medicine 102, no. 1 (1986): 878–80. http://dx.doi.org/10.1007/bf00839978.

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22

Russo, Pierre A., Pierre Brochu, Ernest G. Seidman, and Claude C. Roy. "Autoimmune Enteropathy." Pediatric and Developmental Pathology 2, no. 1 (1999): 65–71. http://dx.doi.org/10.1007/s100249900092.

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Autoimmune enteropathy (AIE) is an entity reported primarily in infancy, resulting in intractable diarrhea and associated with small bowel villous atrophy and the presence of circulating anti-enterocyte (AEA) antibodies. It is a multisystem disorder with a response, in many cases, to immunosuppressive therapy.

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23

Avent, Neil D. "Unraveling the immune response during AIHA." Blood 111, no. 2 (2008): 483. http://dx.doi.org/10.1182/blood-2007-09-111054.

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In this issue of Blood, Ward and colleagues make some novel fundamental observations on the nature of the immune response during autoimmune hemolytic anemia (AIHA). They show a key role for T regulatory cells (Tregs) in the pathogenesis of this autoimmune disease.

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24

Shoenfeld, Yehuda. "COVID-19 AND POST COVID-19 AND THE EMERGENCE OF SLE AND EXACERBATIONS." Journal of Rheumatology 52, Suppl 1 (2025): 42.2–42. https://doi.org/10.3899/jrheum.2025-0390.o046a.

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O046a / #565Topic:AS01 - Adaptive ImmunityABSTRACT CONCURRENT SESSION 08: RECENT ADVANCES IN LUPUS BIOMARKERS23-05-2025 1:40 PM - 2:40 PMBackground/PurposeCovid-19 virus is an autoimmune virus and more notorious than EBV. It induces autoimmune diseases by hyperstimulation combined with induction of autoimmune disease by molecular mimicry. There are many autoimmune conditions which have emerged following the COVID 19 infection. One of them is SLE. We will summarize the various publications discussing de novo eruption of SLE as well as induction of exacerbation.MethodsWe assume the possibility o

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Lapierre, Pascal, and Alain Lamarre. "Regulatory T Cells in Autoimmune and Viral Chronic Hepatitis." Journal of Immunology Research 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/479703.

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In both autoimmune liver disease and chronic viral hepatitis, the injury results from an immune-mediated cytotoxic T cell response to liver cells. As such, it is not surprising that CD4+regulatory T cells, a key regulatory population of T cells able to curb immune responses, could be involved in both autoimmune hepatitis and chronic viral hepatitis. The liver can induce the conversion of naïve CD4+T cells to CD4+regulatory T cells and induce tolerance to locally expressed antigens. This tolerance mechanism is carefully regulated in physiological conditions but any imbalance could be pathologic

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26

Perdan-Pirkmajer, K., GG Thallinger, N. Snoj, et al. "Autoimmune response following influenza vaccination in patients with autoimmune inflammatory rheumatic disease." Lupus 21, no. 2 (2012): 175–83. http://dx.doi.org/10.1177/0961203311429817.

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27

Nastasio, Silvia, Marco Sciveres, Paola Francalanci, and Giuseppe Maggiore. "Pediatric Autoimmune Hepatitis." Pediatric Reports 16, no. 1 (2024): 110–13. http://dx.doi.org/10.3390/pediatric16010011.

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Pediatric autoimmune hepatitis (PAIH) is a rare necro-inflammatory disease of the liver of unknown etiology thought to derive from the dysregulation of the immune response upon exposure to environmental triggers in genetically predisposed individuals [...]

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28

Mizrachi, Tehila, Livnat Brill, Malcolm Rabie, et al. "NMO-IgG and AQP4 Peptide Can Induce Aggravation of EAMG and Immune-Mediated Muscle Weakness." Journal of Immunology Research 2018 (2018): 1–10. http://dx.doi.org/10.1155/2018/5389282.

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Neuromyelitis optica (NMO) and myasthenia gravis (MG) are autoimmune diseases mediated by autoantibodies against either aquaporin 4 (AQP4) or acetylcholine receptor (AChR), respectively. Recently, we and others have reported an increased prevalence of NMO in patients with MG. To verify whether coexisting autoimmune disease may exacerbate experimental autoimmune MG, we tested whether active immunization with AQP4 peptides or passive transfer of NMO-Ig can affect the severity of EAMG. Injection of either AQP4 peptide or NMO-Ig to EAMG or to naive mice caused increased fatigability and aggravatio

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29

Kosiyakul, Punchika, Jiraporn Jitprapaikulsan, Ekdanai Uawithya, et al. "Real-world data on the Immunity Response to the COVID-19 Vaccine among Patients with Central Nervous System Immunological Diseases." Siriraj Medical Journal 76, no. 2 (2024): 69–79. http://dx.doi.org/10.33192/smj.v76i2.266638.

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Objective: The effects of immunotherapies on the immune response to various regimens of SARS-CoV-2 vaccines in patients with autoimmune neurological disease have been demonstrated in limited data. Thus, we evaluated the immune responses in each platform of COVID-19 vaccination between patients with autoimmune neurological disease and a healthy population. Materials and Methods: We conducted a prospective observational study. We collected serum from patients with autoimmune neurological diseases to perform serological methods using anti-RBD IgG assay, neutralizing antibodies assay, and interfer

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30

Zandanell, Stephan, Lorenz Balcar, Georg Semmler, et al. "Genetic Variants Determine Treatment Response in Autoimmune Hepatitis." Journal of Personalized Medicine 13, no. 3 (2023): 540. http://dx.doi.org/10.3390/jpm13030540.

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Background: Autoimmune hepatitis (AIH) is a rare entity; in addition, single-nucleotide polymorphisms (SNPs) may impact its course and outcome. We investigated liver-related SNPs regarding its activity, as well as in relation to its stage and treatment response in a Central European AIH cohort. Methods: A total of 113 AIH patients (i.e., 30 male/83 female, median 57.9 years) were identified. In 81, genotyping of PNPLA3-rs738409, MBOAT7-rs626238, TM6SF2-rs58542926, and HSD17B13-rs72613567:TA, as well as both biochemical and clinical data at baseline and follow-up, were available. Results: The m

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Algraittee, Satar Jabbar Rahi. "Autoimmune Hepatitis: Evaluation of Clinical Presentation, Diagnosis and Response to Treatment in Iraqi Patients." Journal of Communicable Diseases 53, no. 04 (2021): 104–13. http://dx.doi.org/10.24321/0019.5138.202180.

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Introduction: Autoimmune hepatitis (AIH) is a chronic heterogeneous inflammatory disease of the liver activated by unknown triggers. The details of its pathogenesis remain unclear. Sufficient data on AIH in terms of epidemiology as well as disease phenotype amongst the Iraqi population is not available. Objectives: To evaluate and document the experience of Iraqi AIH patients with respect to clinical presentation, diagnosis, treatment and response to treatment in accordance with the IAIHG guidelines. Method: A prospective study was conducted on patients who attended the Gastroenterology Hepato

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32

Pistoresi-Palencia, M. C., P. Rodrigues, S. Gea, and E. Vottero-Cima. "IgE Dependent Autoimmune Response. Effect ofTrypanosoma CruziInfection." Autoimmunity 8, no. 1 (1990): 53–58. http://dx.doi.org/10.3109/08916939008998432.

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33

Simons, Madison, Jack Loesch, Eyad Hamza, et al. "S2276 Response of Autoimmune Gastroparesis to Immunomodulation." American Journal of Gastroenterology 119, no. 10S (2024): S1630—S1631. http://dx.doi.org/10.14309/01.ajg.0001038472.07898.80.

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34

Axelrad, J. "An autoimmune response causes transmissible spongiform encephalopathies." Medical Hypotheses 50, no. 3 (1998): 259–64. http://dx.doi.org/10.1016/s0306-9877(98)90027-5.

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35

Nachman, Ralph L. "Refractory autoimmune thrombocytopenia: response to antiplatelet therapy." Lancet 366, no. 9494 (2005): 1410. http://dx.doi.org/10.1016/s0140-6736(05)67571-x.

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36

Klote, Mary M., and Renata JM Engler. "Response to sex differences in autoimmune disease." Lupus 16, no. 6 (2007): 457. http://dx.doi.org/10.1177/0961203307078732.

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37

Schramm, Christoph, Christina Weiler-Normann, Christiane Wiegard, Stefanie Hellweg, Susanne Müller, and Ansgar W. Lohse. "Treatment response in patients with autoimmune hepatitis." Hepatology 52, no. 6 (2010): 2247–48. http://dx.doi.org/10.1002/hep.23840.

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38

Falcone, Marika, Jae Lee, Gail Patstone, Brian Yeung, and Nora Sarvetnick. "B Lymphocytes Are Crucial Antigen-Presenting Cells in the Pathogenic Autoimmune Response to GAD65 Antigen in Nonobese Diabetic Mice." Journal of Immunology 161, no. 3 (1998): 1163–68. http://dx.doi.org/10.4049/jimmunol.161.3.1163.

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Abstract Recent reports have shown that B cells play a key role in the pathogenesis of T cell-mediated autoimmune diseases such as insulin-dependent diabetes mellitus (IDDM) in nonobese diabetic mice (NOD). We have investigated the role of B lymphocytes as APCs in the generation of autoreactive T cell responses by comparing spontaneous responses to self Ags in B cell-deficient and wild-type NOD mice. We determined that B cell-deficient mice had no spontaneous responses to 65-kDa glutamate decarboxylase (GAD65), its immunodominant peptides, and the 60-kDa heat shock protein. In contrast, these

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Tukaj, Stefan, and Krzysztof Sitko. "Heat Shock Protein 90 (Hsp90) and Hsp70 as Potential Therapeutic Targets in Autoimmune Skin Diseases." Biomolecules 12, no. 8 (2022): 1153. http://dx.doi.org/10.3390/biom12081153.

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Over a hundred different autoimmune diseases have been described to date, which can affect every organ in the body, including the largest one, the skin. In fact, up to one-fifth of the world’s population suffers from chronic, noninfectious inflammatory skin diseases, the development of which is significantly influenced by an autoimmune response. One of the hallmarks of autoimmune diseases is the loss of immune tolerance, which leads to the formation of autoreactive lymphocytes or autoantibodies and, consequently, to chronic inflammation and tissue damage. The treatment of autoimmune skin disea

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Saunders, Karin A., Tim Raine, Anne Cooke, and Catherine E. Lawrence. "Inhibition of Autoimmune Type 1 Diabetes by Gastrointestinal Helminth Infection." Infection and Immunity 75, no. 1 (2006): 397–407. http://dx.doi.org/10.1128/iai.00664-06.

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ABSTRACT Gastrointestinal nematode infections are prevalent worldwide and are potent inducers of T helper 2 responses with the capacity to modulate the immune response to heterologous antigens. Parasitic helminth infection has even been shown to modulate the immune response associated with autoimmune diseases. Nonobese diabetic (NOD) mice provide a model for studying human autoimmune diabetes; as in humans, the development of diabetes in NOD mice has been linked to the loss of self-tolerance to beta cell autoantigens. Previous studies with the NOD mouse have shown that helminth and bacterial i

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Nevárez-Lechuga, Christian Irene, Sandra Sánchez-Barbosa, Carla Landa-Saldívar, et al. "Development of a murine model resembling human lupus in mice parasitized with Trichinella spiralis." Journal of Immunology 200, no. 1_Supplement (2018): 175.16. http://dx.doi.org/10.4049/jimmunol.200.supp.175.16.

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Abstract Deregulation of Th1 and Th17 immune responses is fundamental in the development of autoimmune diseases, like Systemic Lupus Erythematosus that is a chronic inflammatory autoimmune disease characterized by the production of autoantibodies that cause inflammation and damage to different organs. New therapeutic approaches for autoimmune diseases have been based on the fact that nematodes such as Trichinella spiralis decrease the inflammatory response by inducing the production of anti-inflammatory cytokines, which are potent modulators of T cell function. Recent studies have shown that t

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Merello, Marcelo, Martin Nogues, Ramon Leiguarda, C. L�pez Saubidet, and Alejo Florin. "Abnormal sympathetic skin response in patients with autoimmune vitiligo and primary autoimmune hypothyroidism." Journal of Neurology 240, no. 2 (1993): 72–74. http://dx.doi.org/10.1007/bf00858719.

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Arthur Blair, Eric. "Immune deficiency derive a favorable response to IVIg in PANDAS." Psychology and Mental Health Care 2, no. 3 (2018): 01–03. http://dx.doi.org/10.31579/2637-8892/032.

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For two decades, pediatric autoimmune neuropsychiatric disorder associated with group a beta hemolytic streptococcal infection (PANDAS) has been treated with high-dose intravenous immune globulin (IVIg) therapy based upon the understanding that the disorder is partly due to post-infectious dysimmunity.

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Graham, Alice, Crystal Fong, Asghar Naqvi, and Jian-Qiang Lu. "Brain Toxoplasmosis Comorbid with Autoimmune Disease: Complicated Immune Response And Case Demonstration." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 48, s2 (2021): S8. http://dx.doi.org/10.1017/cjn.2021.164.

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Toxoplasmosis is an opportunistic infection caused by Toxoplasma gondii (TG), commonly involving the brain. Symptomatic clinical disease of TG infection is much more commonly associated with immunodeficiency; clinicopathological manifestations of brain toxoplasmosis are linked to individual immune responses including brain infiltration of T-cells that are thought to fight against toxoplasmosis. In patients with autoimmune diseases, immune status is typically characterized by T-cell infiltration and complicated mainly by immunosuppressant and/or immunomodulatory treatment. In this study, we dem

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Antony, Irene Rose, Brandon Han Siang Wong, Dermot Kelleher, and Navin Kumar Verma. "Maladaptive T-Cell Metabolic Fitness in Autoimmune Diseases." Cells 12, no. 21 (2023): 2541. http://dx.doi.org/10.3390/cells12212541.

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Immune surveillance and adaptive immune responses, involving continuously circulating and tissue-resident T-lymphocytes, provide host defense against infectious agents and possible malignant transformation while avoiding autoimmune tissue damage. Activation, migration, and deployment of T-cells to affected tissue sites are crucial for mounting an adaptive immune response. An effective adaptive immune defense depends on the ability of T-cells to dynamically reprogram their metabolic requirements in response to environmental cues. Inability of the T-cells to adapt to specific metabolic demands m

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46

Yeste, Ada, and Francisco J. Quintana. "Antigen Microarrays for the Study of Autoimmune Diseases." Clinical Chemistry 59, no. 7 (2013): 1036–44. http://dx.doi.org/10.1373/clinchem.2012.194423.

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BACKGROUND The immune response involves the activation of heterogeneous populations of T cells and B cells that show different degrees of affinity and specificity for target antigens. Although several techniques have been developed to study the molecular pathways that control immunity, there is a need for high-throughput assays to monitor the specificity of the immune response. CONTENT Antigen microarrays provide a new tool to study the immune response. We reviewed the literature on antigen microarrays and their advantages and limitations, and we evaluated their use for the study of autoimmune

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47

Bareke, Halin, Pablo Juanes-Velasco, Alicia Landeira-Viñuela, et al. "Autoimmune Responses in Oncology: Causes and Significance." International Journal of Molecular Sciences 22, no. 15 (2021): 8030. http://dx.doi.org/10.3390/ijms22158030.

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Specific anti-tumor immune responses have proven to be pivotal in shaping tumorigenesis and tumor progression in solid cancers. These responses can also be of an autoimmune nature, and autoantibodies can sometimes be present even before the onset of clinically overt disease. Autoantibodies can be generated due to mutated gene products, aberrant expression and post-transcriptional modification of proteins, a pro-immunogenic milieu, anti-cancer treatments, cross-reactivity of tumor-specific lymphocytes, epitope spreading, and microbiota-related and genetic factors. Understanding these responses

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Alatas, Fatima Safira, Gryselda Hanafi, Lestari Kanti Wilujeng, and Nielda Kezia Sumbung. "Autoimmune Hepatitis." Archives of Pediatric Gastroenterology, Hepatology, and Nutrition 1, no. 1 (2022): 17–27. http://dx.doi.org/10.58427/apghn.1.1.2022.17-27.

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Autoimmune hepatitis (AIH) is a condition caused by self-perpetuating immune response towards hepatocytes in liver. In children, AIH may progressed more rapidly compared to adults. Thus, early diagnosis and prompt treatment are the key for successful management of AIH. Five main characteristics of AIH include female predominance, increased IgG or hypergammaglobulinemia, circulatory autoantibody seropositivity, and hepatitis interface from the histological finding. Liver biopsy is needed to evaluate the degree of damage and to confirm the diagnosis. The standard regiment for AIH include prednis

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Zhang, Zijing. "Immunotherapy Toward Autoimmune Disease and Cancer." SHS Web of Conferences 174 (2023): 03007. http://dx.doi.org/10.1051/shsconf/202317403007.

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Specific immunity refers to the ability of the human body to resist infection obtained by acquiring infection. It is generally formed after microbial and other antigenic material stimulation and the activation of lymphocyte and the production of immunoglobin is typical of this process. If the specific immune response is not correctly triggered, some neoplastic diseases (such as cancer) may develop as the tumor cell can escape the immune response. If the specific immune response is wrongly triggered, autoimmune diseases may be developed as the immunoglobin and active lymphocyte may accumulate a

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50

Ur Rehman, Jahanzeb, Asghar Ali Kerio, Raheel Iftikhar, Uzma Rahim, and Sahla Riaz. "Autoimmune Hemolytic Anemia (AIHA): Response to First Line Therapy in Patients Treated at Tertiary Care Centre." Medical Journal of South Punjab 5, no. 2 (2024): 33–40. https://doi.org/10.61581/mjsp.vol05/02/05.

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Objective: To determine the response of First line therapy in patients with AIHA. Methods: Eighty individuals of various age groups with AIHA were included. Prednisolone was administered as first line treatment in maximal dose of 1-2mg/kg/day. The initial response of hemoglobin was assessed on day 14 and subsequently every two weeks until the full response was attained. Numerical variables i-e age, duration of response, hemoglobin (Hb), lactate dehydrogenase (LDH), indirect bilirubin were assessed by mean ±SD. Categorical variables i-e direct antiglobulin test (DAT), gender, response (complete

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