Relevant bibliographies by topics / Cancer of mammary gland

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cancer of mammary gland'

Author: Grafiati
Published: 28 May 2022
Last updated: 30 July 2025

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Journal articles on the topic "Cancer of mammary gland"

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Batan, Sonia, Jabunnesa Khanom, Sabarish Ramachandran, et al. "Abstract P3-04-04: The Butyrate Transporter SLC5A8 Selectively Inhibits Breast Tumor Metastasis." Clinical Cancer Research 31, no. 12_Supplement (2025): P3–04–04—P3–04–04. https://doi.org/10.1158/1557-3265.sabcs24-p3-04-04.

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Abstract Introduction: The mammary gland is a dynamic organ that undergoes significant developmental changes during pregnancy, lactation, and involution. The process of involution is a highly orchestrated series of molecular and physical events that can be divided into two distinct phases. (Lund et al., 1996). Accumulation of milk in the alveolar lumen (milk stasis) is required to initiate the first phase during which the secretory cells begin to enter apoptosis. Here we provide genetic and molecular biological evidence to shows that the short-chain fatty acid Butyrate (BTR), a significant com
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Mogus, Joshua Philip. "Exposure to the Endocrine Disruptor, Propylparaben, During Pregnancy and Lactation, Alters Typical Parity-Induced Reorganization of the Mouse Mammary Gland." Journal of the Endocrine Society 5, Supplement_1 (2021): A487—A488. http://dx.doi.org/10.1210/jendso/bvab048.997.

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Abstract The mammary gland is a hormone sensitive organ that is susceptible to endocrine disrupting chemicals (EDCs) during several vulnerable periods, including pregnancy and lactation. Mammary gland reorganization during pregnancy and lactation is hormone driven and provides long-term protection against breast cancer risk. It is unknown if EDC exposures during these sensitive windows can alter mammary reorganization to either enhance or offset parity-induced protection against breast cancer. Here, we examined effects of propylparaben (PP), a common preservative used in personal care products
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Atashgaran, Vahid, Pallave Dasari, Leigh J. Hodson, Andreas Evdokiou, Simon C. Barry, and Wendy V. Ingman. "Foxp3 heterozygosity does not overtly affect mammary gland development during puberty or the oestrous cycle in mice." Reproduction, Fertility and Development 32, no. 8 (2020): 774. http://dx.doi.org/10.1071/rd19378.

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Female mice heterozygous for a genetic mutation in transcription factor forkhead box p3 (Foxp3) spontaneously develop mammary cancers; however, the underlying mechanism is not well understood. We hypothesised that increased cancer susceptibility is associated with an underlying perturbation in mammary gland development. The role of Foxp3 in mammary ductal morphogenesis was investigated in heterozygous Foxp3Sf/+ and wildtype Foxp3+/+ mice during puberty and at specific stages of the oestrous cycle. No differences in mammary ductal branching morphogenesis, terminal end bud formation or ductal el
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Avagliano, Angelica, Giuseppe Fiume, Maria Rosaria Ruocco, et al. "Influence of Fibroblasts on Mammary Gland Development, Breast Cancer Microenvironment Remodeling, and Cancer Cell Dissemination." Cancers 12, no. 6 (2020): 1697. http://dx.doi.org/10.3390/cancers12061697.

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The stromal microenvironment regulates mammary gland development and tumorigenesis. In normal mammary glands, the stromal microenvironment encompasses the ducts and contains fibroblasts, the main regulators of branching morphogenesis. Understanding the way fibroblast signaling pathways regulate mammary gland development may offer insights into the mechanisms of breast cancer (BC) biology. In fact, the unregulated mammary fibroblast signaling pathways, associated with alterations in extracellular matrix (ECM) remodeling and branching morphogenesis, drive breast cancer microenvironment (BCM) rem
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GARIFULLOVA, YU V., L. I. MALTSEVA, and M. G. KALINKINA. "Digital mammography in clinical practice of a gynecologist: highlighting the key points." Practical medicine 21, no. 1 (2023): 17–23. http://dx.doi.org/10.32000/2072-1757-2023-1-17-23.

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High density of the mammary gland, determined with the digital mammography method, is an independent risk factor for mammary gland cancer (MGC). Prescription of combined oral contraceptives, menopausal hormone therapy drugs, intrauterine system with levonorgestrel, and ovulation induction may lead to an increase of mammary gland density (MGD). The latest research showed that the insulin-like growth factor is also a risk factor for mammary gland density. In the Republic of Tatarstan, out of 265,217 examined women, a high mammary gland density was diagnosed in 7%, including in patients older tha
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Mustafi, Devkumar, Abby Leinroth, Xiaobing Fan, et al. "Magnetic Resonance Angiography Shows Increased Arterial Blood Supply Associated with Murine Mammary Cancer." International Journal of Biomedical Imaging 2019 (January 17, 2019): 1–6. http://dx.doi.org/10.1155/2019/5987425.

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Breast cancer is a major cause of morbidity and mortality in Western women. Tumor neoangiogenesis, the formation of new blood vessels from pre-existing ones, may be used as a prognostic marker for cancer progression. Clinical practice uses dynamic contrast enhanced magnetic resonance imaging (DCE-MRI) to detect cancers based on increased blood flow and capillary permeability. However, DCE-MRI requires repeated injections of contrast media. Therefore we explored the use of noninvasive time-of-flight (TOF) MR angiography for serial studies of mouse mammary glands to measure the number and size o
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Ma, Zhiyuan, Dumin Yuan, Xiaoming Cheng, Biguang Tuo, Xuemei Liu, and Taolang Li. "Function of ion transporters in maintaining acid-base homeostasis of the mammary gland and the pathophysiological role in breast cancer." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 318, no. 1 (2020): R98—R111. http://dx.doi.org/10.1152/ajpregu.00202.2019.

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The incidence of breast cancer is increasing year by year, and the pathogenesis is still unclear. Studies have shown that the high metabolism of solid tumors leads to an increase in hypoxia, glycolysis, production of lactic acid and carbonic acid, and extracellular acidification; a harsh microenvironment; and ultimately to tumor cell death. Approximately 50% of locally advanced breast cancers exhibit hypoxia and/or local hypoxia, and acid-base regulatory proteins play an important role in regulating milk secretion and maintaining mammary gland physiological function. Therefore, ion transporter
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Wu, Diana, Lilian U. Thompson, and Elena M. Comelli. "MicroRNAs: A Link between Mammary Gland Development and Breast Cancer." International Journal of Molecular Sciences 23, no. 24 (2022): 15978. http://dx.doi.org/10.3390/ijms232415978.

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Breast cancer is among the most common cancers in women, second to skin cancer. Mammary gland development can influence breast cancer development in later life. Processes such as proliferation, invasion, and migration during mammary gland development can often mirror processes found in breast cancer. MicroRNAs (miRNAs), small, non-coding RNAs, can repress post-transcriptional RNA expression and can regulate up to 80% of all genes. Expression of miRNAs play a key role in mammary gland development, and aberrant expression can initiate or promote breast cancer. Here, we review the role of miRNAs
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Anderson, Breanne M., Mira B. MacLennan, Lyn M. Hillyer, and David W. L. Ma. "Lifelong exposure to n-3 PUFA affects pubertal mammary gland development." Applied Physiology, Nutrition, and Metabolism 39, no. 6 (2014): 699–706. http://dx.doi.org/10.1139/apnm-2013-0365.

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There is growing evidence that early developmental periods may importantly influence future breast cancer risk. Also, there is great interest in the role of dietary fat in breast cancer risk, but the role of dietary fat during pubertal mammary gland development remains poorly understood. This study investigated the effect of n-3 polyunsaturated fatty acids (PUFA) using complementary dietary and genetic approaches to examine the effect of lifelong exposure of n-3 PUFA or n-6 PUFA (control) on mammary gland development and fatty acid composition. n-3 PUFA from both diet and genetics were enriche
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Chakraborty, Moumita, and Michal Hershfinkel. "Zinc Signaling in the Mammary Gland: For Better and for Worse." Biomedicines 9, no. 9 (2021): 1204. http://dx.doi.org/10.3390/biomedicines9091204.

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Zinc (Zn2+) plays an essential role in epithelial physiology. Among its many effects, most prominent is its action to accelerate cell proliferation, thereby modulating wound healing. It also mediates affects in the gastrointestinal system, in the testes, and in secretory organs, including the pancreas, salivary, and prostate glands. On the cellular level, Zn2+ is involved in protein folding, DNA, and RNA synthesis, and in the function of numerous enzymes. In the mammary gland, Zn2+ accumulation in maternal milk is essential for supporting infant growth during the neonatal period. Importantly,
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Dissertations / Theses on the topic "Cancer of mammary gland"

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Pascual, Domingo Rosa. "CPEB2 in mammary gland homeostasis and breast cancer." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/586313.

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The mammary gland develops postnatally and is remodeled, during each estrous cycle and pregnancy, through very dynamic expansions and involutions of its polarized epithelial tree. Moreover, the mammary gland is hierarchically organized, where the two main epithelial populations may arise from a common bipotent mammary stem cell (MaSC). The principal epithelial cell types in the mammary gland are luminal cells and myoepithelial cells (also named basal cells), which are found surrounding luminal cells and in contact with the extracellular matrix. Further, the luminal compartment has two main li
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Artibani, Mara. "WT1 role in mammary gland and breast cancer biology." Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/21036.

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The Wilms' Tumour Suppressor gene 1, WT1, encodes for a complex protein which is essential in mammals throughout life. Its roles vary with the developmental stages: in the embryo, it regulates the epithelial-mesenchymal balance required for a correct organogenesis and acts as a tumour suppressor; in the adult, it is involved in the maintenance of tissue homeostasis and has been controversially considered as an oncogene. Breast cancer is one of the adult tumours in which WT1 oncogenic function was first hypothesised. This malignancy is the most common in women, with more than one million cases
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Dawson, Simon Paul. "Genes expressed in the lactating rabbit mammary gland." Thesis, University of Nottingham, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335293.

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Kutanzi, Kristy, and University of Lethbridge Faculty of Arts and Science. "The role of epigenetics in the rat mammary gland." Thesis, Lethbridge, Alta. : University of Lethbridge, Dept. of Biological Sciences, c2010, 2010. http://hdl.handle.net/10133/2492.

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Epigenetics plays an important role in carcinogenesis with heritable changes in DNA methylation and histone modifications intricately linked to the initiation, promotion, and progression of cancer. Evidence shows that a number of chemical and physical agents can induce epigenetic changes during carcinogenesis. Two such agents, estrogen and ionizing radiation, are generally recognized as being carcinogenic. Yet the epigenetic repercussions of these carcinogens remain relatively unknown. More importantly, the combined effect of these carcinogens has never been addressed in vivo from an epigeneti
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Knostman, Katherine A. B. "Sodium/iodide symporter regulation by oncogenes in the mammary gland and thyroid gland using mouse models." The Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=osu1181659993.

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Staniszewska, Anna Dominika. "Roles of Stat3 in mammary gland development, involution and breast cancer." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610277.

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Ferrari, Nicola. "Investigating RUNX transcription factors in mammary gland development and breast cancer." Thesis, University of Glasgow, 2013. http://theses.gla.ac.uk/4790/.

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Breast cancer is the third most common cause of cancer death in the UK, accountable for more than 11000 deaths in 2010 alone (www.cancerresearchuk. org). Developmental pathways commonly required for normal development are often hijacked during tumour progression, so a better understanding of mammary gland development is necessary to fully understand the roots of breast cancer. The Runx gene family are known to be important regulators of development in different lineages. In particular RUNX1 and RUNX2 have been widely studied in the context of haematopoiesis and osteogenesis respectively, but t
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Charifou, Elsa. "Characterization and impact of cellular senescence during mammary gland involution." Electronic Thesis or Diss., Sorbonne université, 2022. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2022SORUS559V2.pdf.

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La sénescence est une réponse à un stress biologique, caractérisée par un arrêt stable du cycle cellulaire. Néanmoins, les cellules restent métabolliquement actives et acquièrent un phenotype sécrétoire associé à la sénescence, avec la production d’un sécrétome complexe composé de cytokines, chémiokines, facteurs de croissance et modulateurs du remodelage de la matrice extracellulaire. La sénescence est associée à de nombreux processus pathologiques, comme la tumorigénèse et le vieillessement. Cependant, où, quand et comment la sénescence contribue aux processus physiologiques reste méconnu. P
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Williams, Rebecca. "The role of the Edar signalling pathway in mammary gland development and tumourigenesis." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-the-edar-signalling-pathway-in-mammary-gland-development-and-tumourigenesis(2cfad486-e1ad-4f70-9c39-212e2c6a0c6c).html.

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Edar is a member of the death receptor subfamily of TNF receptors that plays an important role in the development of ectodermal appendages. Mutations in this signalling pathway cause the developmental disorder hypohidrotic ectodermal dysplasia (HED). HED has a similar phenotype in a number of mammals, and includes improper development of ectodermal appendages such as hair, teeth and glands. Edar signalling has not previously been linked with tumourigenesis, however, increased NFĸB signalling has been associated with a number of cancers, including breast cancer, and is known to be downstream of
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Bernardo, Gina M. "Discerning the Role of FOXA1 in Mammary Gland Development and Breast Cancer." Case Western Reserve University School of Graduate Studies / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=case1315607905.

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Books on the topic "Cancer of mammary gland"

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Tavassoli, Fattaneh A. Tumors of the mammary gland. American Registry of Pathology in collaboration with the Armed Forces Institute of Pathology, 2009.

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Ip, Margot M., and Bonnie B. Asch, eds. Methods in Mammary Gland Biology and Breast Cancer Research. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4295-7.

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O'Connell, Fiona Claire. Morphology and gene expression in the postnatal mouse mammary gland. University College Dublin, 1997.

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Carlyle, Jones Thomas, Mohr U, and Hunt Ronald Duncan, eds. Integument and mammary glands. Springer-Verlag, 1989.

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Tan, Kah Poh. Early mammary gland development and cancer risk in rats exposed to flaxseed or its major lignan during suckling. National Library of Canada, 2002.

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Rinzler, Carol Ann. Estrogen and breast cancer: Awarning to women. Macmillan Pub. Co., 1993.

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Rinzler, Carol Ann. Estrogen and breast cancer: A warning to women. Macmillan Pub. Co., 1993.

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Rinzler, Carol Ann. Estrogen and breast cancer: A warning to women. 2nd ed. Hunter House Publishers, 1996.

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Neville, Margaret C., and Charles W. Daniel, eds. The Mammary Gland. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4899-5043-7.

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Martin, Finian, Torsten Stein, and Jillian Howlin, eds. Mammary Gland Development. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6475-8.

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Book chapters on the topic "Cancer of mammary gland"

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Vonderhaar, Barbara K., and Maitreyi Bhattacharjee. "The Mammary Gland." In Biological Responses in Cancer. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1236-9_6.

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Sampayo, Rocío, Sol Recouvreux, María Inés Diaz Bessone, and Marina Simian. "Mammary Gland Organoids." In Cancer Drug Discovery and Development. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-60511-1_3.

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Snyderwine, E. G. "Diet and Mammary Gland Carcinogenesis." In Recent Results in Cancer Research. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-45769-2_1.

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Pike, M. C., A. H. Wu, D. V. Spicer, S. Lee, and C. L. Pearce. "Estrogens, Progestins, and Risk of Breast Cancer." In Progestins and the Mammary Gland. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/2789_2007_059.

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Medina, Daniel. "Mouse Models for Mammary Cancer." In Methods in Mammary Gland Biology and Breast Cancer Research. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4295-7_1.

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Welsch, Clifford W. "Dietary Fat, Calories, and Mammary Gland Tumorigenesis." In Exercise, Calories, Fat and Cancer. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4684-7953-9_16.

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Fernig, David G., Roger Barraclough, Youqiang Ke, et al. "Fibroblast Growth Factors in Mammary Development and Cancer." In Intercellular Signalling in the Mammary Gland. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1973-7_14.

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Medina, Daniel, and Frances Kittrell. "Hormonal Stimulation of the Mouse Mammary Gland." In Methods in Mammary Gland Biology and Breast Cancer Research. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4295-7_10.

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Nguyen, Duy-Ai, Neal Beeman, Michael Lewis, Jerome Schaack, and Margaret C. Neville. "Intraductal Injection into the Mouse Mammary Gland." In Methods in Mammary Gland Biology and Breast Cancer Research. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4295-7_23.

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Rasmussen, Susan B., Lawrence J. T. Young, and Gilbert H. Smith. "Preparing Mammary Gland Whole Mounts from Mice." In Methods in Mammary Gland Biology and Breast Cancer Research. Springer US, 2000. http://dx.doi.org/10.1007/978-1-4615-4295-7_7.

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Conference papers on the topic "Cancer of mammary gland"

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Mazumdar, Abhijit, Jamal L. Hill, Yun Zhang, Frances S. Kittrell, Daniel Medina, and Powel Brown. "Abstract A101: Metformin prevents mammary tumors in p53-null mammary gland mice." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-a101.

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Nair, S., and R. Li. "Critical Role of a Transcription Elongation Regulator in Mammary Gland Development and Mammary Tumorigenesis." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-1152.

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Brown, Powel, Jamal Hill, Francis Kittrell, et al. "Abstract B56: Rexinoid LG100268 and tamoxifen prevents mammary tumors in p53-null mammary gland mice." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Nov 7-10, 2010; Philadelphia, PA. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-10-b56.

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Ibrahim HUSSAIN, Hussain, Ruqayah Ali SALMAN, Fahim M. MAHMOOD, and Ayad H. IBRAHIM. "HISTOLOGICAL AND PHYSIOLOGICAL ASSESSMENT OF ENDOTHELIN-1 AND CHOLESTEROL IN BREAST CANCER OF WOMEN." In VI.International Scientific Congress of Pure,Applied and Technological Sciences. Rimar Academy, 2022. http://dx.doi.org/10.47832/minarcongress6-25.

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Both histological and physiological factors were examined in this investigation. From May 1 to October 2, 2019, researchers collected blood samples from 75 women with breast cancer and 15 healthy women as controls, as well as tissue biopsy from the mammary glands of women whose blood samples had already been collected. The women in the study, who ranged in age from 38 to 68 and had been diagnosed with breast cancer, took part in the study. Gland tissue and the owner of those tumors swelling of the axillary lymph nodes. Another cancer that was found was papillary carcinoma, where epithelial hyp
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Visbal, A., A. Visbal, H. Villanueva, et al. "The Role of Smoothened in Mammary Gland Development and Breast Cancer." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-2164.

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Brady, Nicholas J., Michael A. Farrar, and Kathryn L. Schwertfeger. "Abstract B64: Macrophage-specific deletion of STAT5 disrupts normal mammary gland development and accelerates mammary tumorigenesis." In Abstracts: AACR Special Conference: Advances in Breast Cancer; October 17-20, 2015; Bellevue, WA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.advbc15-b64.

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Moreno, Marcelo, Amauri de Oliveira, Tália Cássia Boff, Gabriela Nogueira Matschinski, and Izadora Czarnobai. "SQUAMOUS CELL CARCINOMA METASTASIS OF THE MAMMARY GLAND: CASE REPORT." In Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1007.

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Introduction: Primary squamous cell carcinoma (SCC) of the breast is a rare neoplasm, which represents less than 0.1% of invasive breast cancers. Therefore, it is essential to discriminate between a primary SCC and a metastatic SCC. In order to be considered a primary carcinoma of the breast, a histological examination of the lesion must show more than 90% of squamous neoplastic cells, in addition to the absence of cutaneous SCC or other anatomical sites. Extra-mammary neoplasm metastases are uncommon, representing 0.5% to 2% of breast malignancies. Metastatic SCC in the mammary gland is an un
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Carter, Matthew, Melissa Troester, Melissa Johnson, D. Joseph Jerry, and Sallie Schneider. "Abstract B115: Parity alters responses to ionizing radiation in the human mammary gland." In Abstracts: Frontiers in Cancer Prevention Research 2008. American Association for Cancer Research, 2008. http://dx.doi.org/10.1158/1940-6207.prev-08-b115.

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Gyorki, D., G. Lindeman, and J. Visvader. "Resident Macrophages Influence Stem Cell Activity in the Mammary Gland." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-49.

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Zhang, Lixing, Louise Howe, Richard Kolesnick, and Anthony Brown. "Abstract B22: Analysis of Lgr5-positive cells in mouse mammary gland." In Abstracts: AACR Special Conference on Developmental Biology and Cancer; November 30 - December 3, 2015; Boston, Massachusetts. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.devbiolca15-b22.

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Reports on the topic "Cancer of mammary gland"

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Bissell, Mina J. Training in Mammary Gland Biology and Breast Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada442975.

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Bissell, Mina J. Training in Mammary Gland Biology and Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398979.

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Bissell, Mina J. Training in Mammary Gland Biology and Breast Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada430411.

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Bissell, Mina J., Mary H. Barcellos-Hoff, Kunxin Luo, G. Shyamala, and Matha R. Stampfer. Training in Mammary Gland Biology and Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada411464.

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Bissell, Mina J. Training in Mammary Gland Biology and Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada421765.

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Xian, Wa, and Jeffrey M. Rosen. Hormonal Regulation of Mammary Gland Development and Breast Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada444687.

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Bissell, Mina J. Undergraduate Training in Mammary Gland Biology and Breast Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada427171.

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Xian, Wa, and Jeffrey M. Rosen. Hormonal Regulation of Mammary Gland Development and Breast Cancer. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada435276.

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Bissell, Mina J. Undergraduate Training in Mammary Gland Biology and Breast Cancer. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada459182.

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10

Chen, Mercy, and Jeffrey Rosen. The Role of Beta-Catenin in Mammary Gland Development and Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada415787.

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