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1

Michael, Glick, ed. Infections, infectious diseases and dentistry. W.B. Saunders Co., 2003.

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2

Christine, Bechtel-Boenning, Boland Mary, and Grady Christine, eds. HIV infection. Perinatally transmitted HIV infection. W.B. Saunders Co., 1996.

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3

Thiemann, Lillian. Double jeopardy: The HIV/HCV co-infection handbook. Community Prescription Service, 1999.

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4

Social Policy Research Group (Zambia). Orphans, widows, and widowers in Zambia: A situation analysis and options for HIV/AIDS survival assistance. Institute for African Studies, University of Zambia, 1993.

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5

Schifter, Jacobo. Las gavetas sexuales del costarricense y el riesgo de infección con el VIH. Editorial Imediex, 1996.

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6

Symposium on HIV/AIDS (2003 Addis Ababa, Ethiopia). Proceedings: Symposium on HIV/AIDS : Addis Ababa, 19 & 20 July 2003 : committed leadership and popular participation--keys in the fight against HIV/AIDS. Walta Information Center, 2003.

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7

National AIDS Control Council (Kenya). M&E Division. Inventory of HIV and AIDS research and evaluation studies in Kenya. National AIDS Control Council, M&E Division, 2006.

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8

Berthelot, Pierre. Jeunes homosexuels masculins: Rapport d'une recension d'écrits. Centre de santé publique de Québec, 1995.

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9

Ross, Hazel. Nutrition & HIV infection. AVERT, 1994.

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10

1957-, Friis Henrik, ed. Micronutrients & HIV infection. CRC Press, 2002.

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11

Musharraf, Husain, and Population Council (Bangladesh), eds. Prevalence of HIV, HBV, HCV and syphilis markers in pregnant women of Bangladesh. Population Council, 1997.

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12

Bird, A. G., ed. Immunology of HIV Infection. Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2980-0.

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13

Fauci, Anthony S., and Giuseppe Pantaleo, eds. Immunopathogenesis of HIV Infection. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60867-4.

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14

Schönnesson, Lena Nilsson, and Michael W. Ross. Coping with HIV Infection. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-4681-8.

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15

Gupta, Sudhir, ed. Immunology of HIV Infection. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0191-0.

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16

Dammacco, Franco, ed. HCV Infection and Cryoglobulinemia. Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-1705-4.

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17

M, Grimes Richard, ed. AIDS and HIV infection. Mosby, 1994.

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18

M, Murphy Siobhan, ed. HIV infection and AIDS. Churchill Livingstone, 1992.

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19

Graham, Bird Angus, ed. Immunology of HIV infection. Kluwer Academic Publishers, 1992.

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20

Murphy, Siobhan M. HIV infection and AIDS. 2nd ed. Churchill Livingstone, 2000.

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21

Vithaysai, Parkong. Atlas of HIV infection. Allergy and Clinical Immunology Unit, Dept. of Medicine, Faculty of Medicine, Chiang Mai University, 1994.

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22

Scottish Committee on HIV Infection and Intravenous Drug Misuse. HIV infection in Scotland. Scottish Home and Health Department, 1986.

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23

United States. Agency for Health Care Policy and Research., ed. Managing early HIV infection. U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1994.

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24

National Institute of Allergy and Infectious Diseases (U.S.), ed. HIV infection and AIDS. National Institute of Allergy and Infectious Diseases, 1992.

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25

Nair, Madhavan P. N., and Stanley Schwartz. Immunology of HIV infection. Edited by Research Signpost (Trivandrum India). Research Signpost, 2003.

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26

Dammacco, Franco. HCV Infection and Cryoglobulinemia. Springer-Verlag Italia, 2012.

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27

1940-, Fauci Anthony S., and Pantaleo G, eds. Immunopathogenesis of HIV infection. Springer, 1997.

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28

J, Pinching A., ed. AIDS and HIV infection. W.B. Saunders, 1986.

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29

A, Johnson Judith. Women with HIV infection. Congressional Research Service, Library of Congress, 1992.

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30

United States. Agency for Health Care Policy and Research, ed. Managing early HIV infection. U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1994.

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31

National Institute of Allergy and Infectious Diseases (U.S.), ed. HIV infection and AIDS. National Institute of Allergy and Infectious Diseases, 1992.

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32

Johnson, Margaret A., M.D. and Johnstone Frank D, eds. HIV infection in women. Churchill Livingstone, 1993.

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33

1942-, Grant Igor, and Martin Alex 1949-, eds. Neuropsychology of HIV infection. Oxford University Press, 1994.

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34

Royal College of Midwives (Great Britain), ed. HIV infection in pregnancy. Books for Midwives Press, 1994.

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35

Sudhir, Gupta, ed. Immunology of HIV infection. Plenum Medical Book Co., 1996.

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36

L, Minkoff Howard, DeHovitz Jack A, and Duerr Ann, eds. HIV infection in women. Raven Press, 1995.

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37

Bernard, Edwin J. Criminal HIV transmission. NAM, 2007.

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38

Policy, American Hospital Association Special Committee on AIDS/HIV Infection. AIDS/HIV infection policy: Ensuring a safe hospital environment. American Hospital Association, 1987.

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39

M, Fanning Mary, ed. HIV infection: A clinical appoach. 2nd ed. Saunders, 1997.

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40

Friedman, Herman, Thomas W. Klein, and John J. Madden. Neuroimmune circuits, drugs of abuse, and infectious diseases. Kluwer Academic, 2002.

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41

Keshav, Satish, and Palak Trivedi. Viral hepatitis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0212.

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Hepatitis means ‘inflammation of the liver’ and is manifest with symptoms that include malaise, anorexia, fever, flu-like symptoms, and pain in the right upper quadrant of the abdomen, with the pain being caused by swelling of the liver and its capsule. Elevations in circulating hepatic enzymes, particularly aspartate transaminase and alanine transaminase, are common, with jaundice occurring some time after the onset of other symptoms and signs. There are five viruses that primarily cause viral hepatitis: hepatitis A, B, C, D, and E viruses, abbreviated HAV, HBV, HCV, HDV, and HEV, respectivel
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42

Wilson, Deanna. Hepatitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0035.

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Hepatitis A (HAV) and E (HEV) viruses are spread via the fecal-oral route. Hepatitis B virus (HBV) exposure is via occupational or recreational activities. Hepatitis D virus (HDV; also spread parentally) can only coinfect or superinfect those with chronic HBV. Hepatitis C (HCV) transmission is predominantly parenteral; the highest risk group is injection drug users. Prodromal-period patients with acute hepatitis present with vague constitutional symptoms when serum transaminases peak, with elevated serum bilirubin and varying levels of hepatic protein synthesis impairment; during the icteric p
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43

Bulterys, Marc, Julia Brotherton, and Ding-Shinn Chen. Prevention of Infection-Related Cancers. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0066.

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This chapter discusses primary prevention measures that disrupt transmission of oncogenic infections. It begins by discussing vaccination against hepatitis B virus (HBV) and human papillomavirus (HPV), two major causes of cancer for which safe and effective vaccines are currently available. It briefly discusses the importance of treatment and prophylaxis against human immunodeficiency virus type 1 (HIV-1), which potentiates the virulence of other viral infections as well as directly increasing the incidence of non-Hodgkin lymphoma. It does not discuss the treatment of HBV or hepatitis C virus
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44

Jadoul, Michel, Laura Labriola, and Eric Goffin. Viral infections in patients on dialysis. Edited by Jonathan Himmelfarb. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0271.

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From the early days of hemodialysis, viral hepatitis has been recognized as common in dialyzed patients.The prevalence and incidence of HBV infection have decreased markedly over the last decades in HD units. Still, the infectivity of HBV is very high. Vaccinating HD patients, preferably prior to starting dialysis, together with the strict application of hygienic precautions and adequate screening of blood donors remains required, together with the segregation of infective (HBV+) patients in a separate dialysis ward. The level of aminotransferases is markedly lower in HD patients than in the g
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45

Jolly, Elaine, Andrew Fry, and Afzal Chaudhry, eds. Infectious diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0012.

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Chapter 12 covers the basic science and clinical topics relating to infectious disease which trainees are required to learn as part of their basic training and demonstrate in the MRCP. It begins with an overview, before covering diagnostic techniques, sepsis, antibiotics, needlestick injury, nosocomial infection, travel-related infection, immunocompromised hosts, pyrexia of unknown origin, infection in injecting drug users, bioterrorism, viral infection, HIV and AIDS, bacterial infections, mycobacterial infections, rickettsial infections, systemic fungal infections, protozoal infections, and h
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46

Barnett, Ben J., and Margaret Hoffman-Terry. HIV/Hepatitis Co-infection. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0039.

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Hepatitis B virus (HBV) infection is common in people living with HIV, and all patients with HIV should be screened for HBV infection. The most common route of transmission worldwide is through perinatal or early childhood exposure, but adult transmission of HBV is often by routes similar to those for HIV, including sexual contact and injection drug use. Although it varies by exposure route, approximately 10% of HIV-positive patients also have chronic HBV infection, and up to 90% have serologic evidence of past exposure to HBV. Long-term complications of HBV infection can include cirrhosis, en
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47

Price, Jennifer Cohen, Priyanka Amin, and Antoine Douaihy. Hepatitis C and HIV Co-Infection. Edited by Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding, and Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0043.

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Chronic infection with hepatitis C virus (HCV) is a leading cause of end-stage liver disease and is the most common indication for liver transplantation in the United States. Because of shared risk factors, individuals living with HIV infection are disproportionately affected by HCV. Moreover, co-infection with HIV accelerates the natural history of chronic HCV infection, increasing the risk of cirrhosis, hepatocellular carcinoma, hepatic decompensation, and death. Highly effective medications such as direct-acting antivirals (DAA) to cure HCV are now available and have the potential to profou
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48

Decision-Making in HIV/HCV Co-Infection. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2016.

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49

Wilson, John W., and Lynn L. Estes. Infectious Syndromes in Adults. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696924.003.0005.

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This section contains tables and text covering an exhaustive group of infectious syndromes including respiratory tract infections, infective endocarditis, intravascular catheter-related infections, central nervous system infections, urinary tract infections, soft-tissue infections, osteomyelitis, gastrointestinal infections, tick-borne infections, tuberculosis, sexually transmitted diseases, HIV, hepatitis, and fungal and zoonotic infections. Vaccination schedules, travel medicine, and bioterrorism are also reviewed.
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50

Kannan, Meena, Harrison Taylor, and William Tyor. HIV Infection. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0148.

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This chapter focuses on four common opportunistic infections of the nervous system associated with HIV infection, namely cryptococcal infection, cytomegalovirus infection, progressive multifocal leukoencephalitis, and toxoplasmosis. Essential features of neurobiology, clinical presentation, differential diagnosis, diagnostic workup, clinical management, and outcome are discussed for each condition. Although combined antiretroviral therapy for HIV has generally reduced the incidence of these complications of HIV infection, they remain important considerations, especially in areas in which antir
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