Relevant bibliographies by topics / Hepato toxicity

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Hepato toxicity'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Hepato toxicity"

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Deglmann, C. J., R. Metzger, M. Stickel, S. Hoerrlein, F. W. Schildberg, and H. G. Koebe. "A New Bioassay Including a Small Scale Hepatocyte Bioreactor for Hepato-Mediated Toxicity Testing in a Target Cell Line." International Journal of Artificial Organs 25, no. 10 (2002): 975–84. http://dx.doi.org/10.1177/039139880202501012.

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New approaches for in vitro testing of hepato-mediated toxicity are undertaken to offer alternatives to in vivo animal testing. The described bioassay for hepato-mediated toxicity testing is based on a small scale hepatocyte-bioreactor with pig hepatocytes connected to a silicon sensor based microphysiometer system for monitoring of the extracellular acidification rate (EAR) of cells and the microphysiometer alone. EAR represents the metabolic activity of tested cells (hepatocytes and ZR 751 cells) under the influence of perfused media, compared to controls, which were set to 100%. Cyclophosph
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Udavant, Pavan, Pragati Gurav, Gayatri Kanade та ін. "Evaluation of Hepatoprotective and Nephroprotective Effect of Α- Pinene on Wistar Albino Rat". Biomedical and Pharmacology Journal 16, № 1 (2023): 103–12. http://dx.doi.org/10.13005/bpj/2592.

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Introduction: Hepato-renal toxicity is a devastating, non-communicable disease. Because of a lack of information on low-cost management to combat the disease, this study postulates the ameliorative effect of selected phytoconstituents against toxicity. Aim and Objective: The current study reveals an active phytoconstituent, α- Pinene, that has the ability to combat the degenerative effects of CCl4. Methodology: Carbon tetrachloride (CCl4) is an organic xenobiotic molecule as well as the most potent hepatotoxic agent used (1200 mg/kg body weight; i.p.) to induce hepato-renal toxicity in experim
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Yuliana, Ida, Triawanti, Muhammad Darwin Prenggono, Ika Kustiyah Oktaviyanti, Irfan Maulana, and Fahrina Ulfah. "Effect of Mercury Administration as an Oxidative Stress Trigger in Hepato-Renal Injuries." JURNAL KESEHATAN LINGKUNGAN 17, no. 2 (2025): 159–67. https://doi.org/10.20473/jkl.v17i2.2025.159-167.

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Introduction: Mercury as the source of free radicals can trigger the activation of oxidative stress pathways. With its high toxicity, it can cause hepato-renal injuries. There have been many studies on mercury toxicity in various organs, but there are still few scientific studies that examine the hepato-renal injuries caused by mercury through the oxidative stress pathway. This study was conducted to investigate the triggering of the oxidative stress pathway due to mercury exposure in hepato-renal injuries. Methods: Research using randomized true laboratory experiment method with post-test con
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Abdullah, A. H., and S. A. Althanoon. "The Immunohistochemical Study of Doxorubicin on Liver and Kidney Tissues in albino Rats." IOP Conference Series: Earth and Environmental Science 1449, no. 1 (2025): 012025. https://doi.org/10.1088/1755-1315/1449/1/012025.

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Abstract The current study was conducted to check the influence of DOX on the liver and kidney of male albino rats. The current work was carried out in a research setting, on characterizing the DOX-induced hepato-renal toxicity model in terms of immunochemical evaluations to provide a methodological reference for studying potential therapeutic drugs with hepato-renal effects. The experiences were performed using two groups of healthful male albino rats (12 rats/group). The control group was given normal saline while the experimental group received a singular dosage of DOX at a concentration of
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Ebito, Gabriel, Adelaja Akinlolu, Mubarak Ameen, et al. "In vivo anti-inflammatory effects of anti-toxic principles from Moringa oleifera(MO11) and Musa sapientum (MS06) via NF-KB/IL-4/IL-10- mediated pathway in Cadmium Chloride-induced hepato-toxicity." Anatomy Journal of Africa 12, no. 1 (2023): 2269–78. http://dx.doi.org/10.4314/aja.v12i1.3.

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Cadmium-induction of hepato-toxicity and inflammation in animal models have been reported. The mechanism underlying hepatic inflammation is poorly understood. This study evaluated antiinflammatory potentials of MO11 (isolated from Moringa oleifera leaves) and MS06 (isolated from Musa sapientum suckers) in Cadmium Chloride (CdCl)-induced hepato-toxicity and inflammation in rats.Twenty-eight adult male wistar rats (average weight of 155 g) were randomly divided into 7 groups (n = 4). Group 1 was control. Groups 2 - 4 and 7 received single intraperitoneal administration of 1.5 mg/Kg bodyweight of
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AKINLOLU, ADELAJA A., TEMITOPE OMOHIMORIA, ADEOYE OYEWOPO, RISIKAT E. KADIR, and MUBARAK O. AMEEN. "Anti-Toxic Principles from Morinda lucida and Annona muricata Down-Regulated Ki67 and Multi-Drug Resistance1 Genes in Lead-Induced Hepato-Toxicity in Rats." Malaysian Journal of Pharmaceutical Sciences 20, no. 2 (2022): 135–46. http://dx.doi.org/10.21315/mjps2022.20.2.11.

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Morinda lucida (ML) and Annona muricata (AM) are ethno-medicinal plants with antioxidant potentials. In addition, lead is a toxic pollutant of global health concerns. This study evaluated the effects of column chromatography-extracted ethanolic fractions of ML and AM leaves on immuno-modulations of Ki67 and multi-drug resistance1 (MDR1) proteins in the liver of rats in lead acetate (LA)-induced hepato-toxicity in-order to determine their hepato-protective, anti-proliferation, anti-drug resistance and anti-cancer potentials. Sixty adult female rats were randomly divided into 12 groups (n = 5).
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Waly, Hanan Salah Ahmed, Mohamed Bassam Al-Salahy, Wafaa Mohammed Hassan El-Arably, Magdy Wilson, and Khaled Mohamed Ahmed Hassanein. "Hepato- and nephro-toxicity of coumatetralyl rodenticide in some wild rat species." Journal of Multidisciplinary Sciences 2, no. 2 (2020): 49–55. http://dx.doi.org/10.33888/jms.2020.226.

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Coumatetralyl (COM) is an anticoagulant rodenticide absorbed quickly after oral dosing. It causes rodent’s death due to internal or external bleeding after complete depletion of plasma vitamin K-dependent coagulation factors. The present study aimed was to shed light on metabolic alterations as the potential hepato- and nephrotoxicity caused by ¼ LD50 COM in Rattus rattus, Arvicanthis niloticus, and Geribellus geribellus wild rat species. Elevation in plasma alanine aminotransferase (ALT), aspartate aminotransferase (AST), albumin, total free amino acids, and erythrocyte lysate glucose-6-phosp
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Dar, AA, A. Fehaid, S. Alkhatani, et al. "The protective role of luteolin against the methotrexate-induced hepato-renal toxicity via its antioxidative, anti-inflammatory, and anti-apoptotic effects in rats." Human & Experimental Toxicology 40, no. 7 (2021): 1194–207. http://dx.doi.org/10.1177/0960327121991905.

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Methotrexate (MTX) is frequently used drug in treatment of cancer and autoimmune diseases. Unfortunately, MTX has many side effects including the hepato-renal toxicity. In this study, we hypothesized that Luteolin (Lut) exhibits protective effect against the MTX-induced hepato-renal toxicity. In order to investigate our hypothesis, the experiment was designed to examine the effect of exposure of male rats to MTX (20 mg/kg, i.p., at day 9) alone or together with Lut (50 mg/kg, oral for 14 days) compared to the control rats (received saline). The findings demonstrated that MTX treatment induced
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Sumathi Rajamani, Gobinath Kalyanasundaram, Tamizharasi Sengodan, Sivakumar Thangavelu, Nikhitha K Shanmukhan, and Arun Radhakrishnan. "Hepato & nephro protective effects of naringenin-loaded tpgs polymeric nanosuspension against cisplatin-induced toxicity." International Journal of Research in Pharmaceutical Sciences 10, no. 4 (2019): 2755–64. http://dx.doi.org/10.26452/ijrps.v10i4.1544.

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Cisplatin (Cis-Diammineplatinum (II) dichloride/CIS) is one of the most potent chemotherapeutic agents widely used in treatment of various cancers. Naringenin (NAR), a natural flavonoid, protect against CIS-induced injury in rats without hampering CIS beneficial cytotoxic activity. Even though NAR exhibits therapeutic potency, clinical evolution of the molecule is embarrassed because of very less aqueous solubility which corresponds to low availability at the site of the tumor. In our former analysis, nanosuspension of naringenin (NARNS) was developed by the method of high-pressure homogenizat
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Rana, Suresh V. S., and Nidhi Rastogi. "Effects of Cadmium On Liver Function in Diabetic Rats." Toxicology and Industrial Health 14, no. 3 (1998): 473–77. http://dx.doi.org/10.1177/074823379801400306.

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Hepato-toxicity of cadmium in alloxan induced diabetic rats has been studied by estimating a few enzymes viz serum glutamic oxalacetic transaminase, glutamic pyruvic transaminase, alkaline phosphatase and γ-glutamyltranspeptidase. Present results suggest that cadmium manifests difterent effects in normal and diabetic rats. Insulin therapy helps in restoring the liver function. It is suggested that an isozyme of cytochrome P450 that appears in diabetic rats might be responsible for altered toxicity of cadmium.
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Dissertations / Theses on the topic "Hepato toxicity"

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Bonfim, Daniel José Pimentel. "INFLUÊNCIA DO pH DA ÁGUA NA HEPATO E NEFROTOXICIDADE NA INTOXICAÇÃO CRÔNICA POR CÁDMIO EM RATOS WISTAR." Universidade do Oeste Paulista, 2014. http://bdtd.unoeste.br:8080/tede/handle/tede/298.

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Made available in DSpace on 2016-01-26T18:55:40Z (GMT). No. of bitstreams: 1 Daniel Jose Pimentel Bonfim.pdf: 627142 bytes, checksum: c21af76961f71071fd84a086e01d5155 (MD5) Previous issue date: 2014-08-22<br>Introduction: Cadmium is a heavy metal found in the environment and used industrially and can cause hepato-and nephrotoxic effects. Objective: To evaluate the effect of pH of drinking water in the hepato-and nephrotoxicity caused by chronic cadmium poisoning. Methods: We used 90 Wistar albino rats, adult, male, divided into 6 groups (n = 15): GC5 - solution of cadmium chloride in drinkin
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Al-Attrache, Houssein. "Etude de la toxicité idiosyncratique de médicaments sur cellules HepaRG et levure : influence du stress inflammatoire et de la biotransformation." Thesis, Rennes 1, 2016. http://www.theses.fr/2016REN1B049/document.

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Chez l’homme, de nombreux médicaments ne sont toxiques que chez un petit nombre de patients traités. Divers facteurs de susceptibilité, génétiques et autres (doses quotidiennes, stress inflammatoire, réaction immune, maladie hépatique,…), favoriseraient la survenue de ces toxicités de type idiosyncratique, non prévisibles par des études chez l’animal. Leur prédiction et la caractérisation des mécanismes impliqués représentent donc un challenge majeur. Dans ce travail, nous avons étudié in vitro l’influence d’un stress inflammatoire sur le potentiel cytotoxique, cholestatique et stéatosique de
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Wild, Stacie Lynn. "Pyrrolizidine alkaloids: Hepatic metabolism and extrahepatic toxicity." Diss., The University of Arizona, 1994. http://hdl.handle.net/10150/186599.

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Pyrrolizidine alkaloids are proposed to be metabolized in the liver to reactive pyrrole species, or dehydroalkaloids. These reactive pyrroles are hypothesized to be responsible for pyrrolizidine alkaloid toxicity. This dissertation research has established that dehydroalkaloids are, in fact, metabolites of pyrrolizidine alkaloids. It was first determined that dehydromonocrotaline is produced during hepatic microsomal metabolism of monocrotaline and that it has the ability to bind in vitro with a synthetic thiol-containing resin, Thiopropyl Sepharose 6B. Similarly, synthetic dehydromonocrotalin
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Savary, Camille. "Étude de la toxicité chronique et du potentiel cancérogène de contaminants de l’environnement séparément et en mélange sur les cellules HepaRG." Thesis, Rennes 1, 2014. http://www.theses.fr/2014REN1B007/document.

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L’Homme est exposé tout au long de sa vie à de nombreux contaminants présents dans l’environnement et l’alimentation généralement à faibles doses et en mélanges. L’évaluation des risques pose problème dans la mesure où il est bien établi qu’il existe des différences de réponse entre l’homme et l’animal. Quelle que soit la voie d’exposition, de par son rôle majeur dans la biotransformation des xénobiotiques, le foie est considéré comme un organe cible pour de nombreuses classes de produits chimiques potentiellement cytotoxiques, génotoxiques voire cancérogènes. Nous avons utilisé la lignée de c
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Smith, Marie-Caroline. "Incidence de la multi-contamination aux mycotoxines de Fusarium sur cellules humaines : évaluation de la cytotoxicité et approche toxico-protéomique." Thesis, Brest, 2017. http://www.theses.fr/2017BRES0084/document.

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Les céréales et les produits issus de leur transformation sont susceptibles d’être contaminés par des espèces fongiques capables de produire des mycotoxines. L’Homme est ainsi exposé tout au long de sa vie à travers son alimentation à ces contaminants naturels, généralement à de faibles doses et en mélange. Cependant, l’incidence de la présence simultanée de ces toxines sur notre santé, à court terme comme à plus long terme, ainsi que les mécanismes responsables de leur toxicité sont encore peu ou mal caractérisés. L’utilisation de modèles cellulaires humains pertinents et adaptés est particul
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Powell, Christine Louise Rusyn Ivan. "Improving linkage of hepatic toxicity and pathology endpoints with toxicogenomics." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,997.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2007.<br>Title from electronic title page (viewed Dec. 18, 2007). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Curriculum in Toxicology." Discipline: Toxicology; Department/School: Medicine.
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Weyers, Carli. "Evaluating metabolism-induced toxicity using a non-hepatic cell line." Thesis, Rhodes University, 2018. http://hdl.handle.net/10962/61950.

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Sauer, John-Michael. "All- trans-retinol modulation of chemically-induced pulmonary and hepatic toxicity." Diss., The University of Arizona, 1996. http://hdl.handle.net/10150/187501.

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It has been previously reported that acute hypervitaminosis A in rats dramatically increases the hepatotoxicity of a number of chemicals. This potentiation appears to be mediated by the enhanced release of reactive oxygen species from retinol-primed Kupffer cells. However, in the lung it has been shown that retinol can protect against many of the inflammatory effects caused by bleomycin. Whether or not retinol pretreatment can modulate chemical-induced injury of compounds that cause both liver and lung toxicity is unknown. Therefore, the studies presented here were designed to test the hypothe
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Benlashehr, Imad. "Fumonisin toxicity in ducks and turkeys." Thesis, Toulouse, INPT, 2013. http://www.theses.fr/2013INPT0070/document.

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Les fumonisines (FBs) sont les principales mycotoxines produites par Fusarium verticillioides et Fusarium proliferatum, qui se retrouvent partout dans le monde dans le maïs et ses produits dérivés. Les doses toxiques et les signes cliniques de toxicité provoqués par les FBs varient dune espèce à lautre. La toxicité des FBs est généralement liée à leur capacité à bloquer le métabolisme des sphingolipides chez les espèces animales, y compris chez les espèces aviaires. De précédentes études ont démontré que les canards présentent une plus grande sensibilité à la toxicité des FBs que les dindes, a
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Scott, Maya Millicent. "Canine hepatic slices as a model for studying drug toxicity and metabolism." Diss., Texas A&M University, 2005. http://hdl.handle.net/1969.1/3731.

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Tissue slices can be made from organs, such as liver, kidney, brain, and heart, and from various species including humans, dogs, non-human primates, rats and mice. It has been demonstrated that human and rat liver slices are viable for up to 2 days, and liver slices have been extensively used as an in vitro method to study hepatic drug metabolism and toxicity in humans. The objective of this study was to determine the utility of canine hepatic slices as an in vitro model for studying drug metabolism and hepatotoxicity in dogs. Canine hepatic slices were incubated in media containing various dr
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Books on the topic "Hepato toxicity"

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Felipo, Vicente, and Santiago Grisolia, eds. Hepatic Encephalopathy, Hyperammonemia, and Ammonia Toxicity. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-1989-8.

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Grisolía, Santiago, Vicente Felipo, and María-Dolores Miñana, eds. Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5826-8.

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International Symposium on Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity, Molecular Basis and Clinical Aspects (1989 Valencia, Spain). Cirrhosis, hepatic encephalopathy, and ammonium toxicity. Plenum Press, 1990.

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Vicente, Felipo, and Grisolía Santiago 1923-, eds. Hepatic encephalopathy, hyperammonemia, and ammonia toxicity. Plenum Press, 1994.

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Redegeld, Franciscus Antonius Maria. Hepatic and renal toxicity of xenobiotics: The role of metabolism and transport. s.n.], 1989.

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Felipo, Vicente, and Santiago Grisolía. Hepatic Encephalopathy, Hyperammonemia, and Ammonia Toxicity. Springer London, Limited, 2012.

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Felipo, Vicente, and Santiago Grisolía. Hepatic Encephalopathy, Hyperammonemia, and Ammonia Toxicity. Springer, 2012.

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Felipo, Vicente, Santiago Grisolía, and Maria-Dolores Miñana. Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity. Springer London, Limited, 2013.

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Hepatic Encephalopathy, Hyperammonemia, and Ammonia Toxicity. Island Press, 1995.

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Felipo, Vicente, Santiago Grisolía, and Maria-Dolores Miñana. Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity. Springer, 2012.

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Book chapters on the topic "Hepato toxicity"

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Xu, Jinghai James, and Keith Hoffmaster. "Hepatic Toxicity." In ADMET for Medicinal Chemists. John Wiley & Sons, Inc., 2010. http://dx.doi.org/10.1002/9780470915110.ch8.

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Rainsford, K. D. "Hepatic Toxicity." In Ibuprofen: Pharmacology, Therapeutics and Side Effects. Springer Basel, 2012. http://dx.doi.org/10.1007/978-3-0348-0496-7_10.

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Thurman, Ronald G., Frederick C. Kauffman, and Jeffrey Baron. "Biotransformation and Zonal Toxicity." In Regulation of Hepatic Metabolism. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5041-5_13.

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Cooper, Arthur J. L., James C. K. Lai, and Alan S. Gelbard. "Ammonia in Liver and Extrahepatic Tissues: An Overview of Metabolism and Toxicity in Mammals." In Hepatic Encephalopathy. Humana Press, 1989. http://dx.doi.org/10.1007/978-1-4612-4506-3_2.

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Ferro, Margherita. "Hepatoma Cell Cultures as In Vitro Models for Hepatotoxicity." In In Vitro Toxicity Testing Protocols. Humana Press, 1995. http://dx.doi.org/10.1385/0-89603-282-5:51.

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Dancygier, Henryk, and Christian P. Strassburg. "Hepatic Drug Metabolism and Drug Toxicity." In Clinical Hepatology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-04519-6_38.

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Coe, John E., Kamal G. Ishak, and M. J. Ross. "Hepatic Toxicity of Estrogen in Armenian and Chinese Hamsters." In Hormonal Carcinogenesis III. Springer New York, 2001. http://dx.doi.org/10.1007/978-1-4612-2092-3_52.

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Viña, Juan R., Federico V. Pallardo, Tadayasu Furukawa, and Jose Viña. "Oral glutathione increases hepatic glutathione and prevents acetaminophen toxicity." In Amino Acids. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-011-2262-7_87.

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Felipo, Vicente, Eugenio Grau, Maria-Dolores Miñana, and Santiago Grisolia. "Activation of NMDA Receptor Mediates the Toxicity of Ammonia and the Effects of Ammonia on the Microtubule-Associated Protein MAP-2." In Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2484-7_8.

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Pan, Guoyu. "Roles of Hepatic Drug Transporters in Drug Disposition and Liver Toxicity." In Advances in Experimental Medicine and Biology. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-7647-4_6.

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Conference papers on the topic "Hepato toxicity"

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Franich, Andjela, Ivana Vasić, Snežana Rajković, et al. "CYTOTOXICITY OF CATIONIC DINUCLEAR PLATINUM(II) COMPLEXES IN AN EXPERIMENTAL MODEL OF MOUSE COLON CANCER." In 1st INTERNATIONAL Conference on Chemo and BioInformatics. Institute for Information Technologies, University of Kragujevac, 2021. http://dx.doi.org/10.46793/iccbi21.293f.

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The series of nine dinuclear platinum(II) complexes, [{Pt(L)Cl}2(μ-X)]2+ (where L is two NH3 or bidentantly coordinated diamine ligand – ethylenediamine, en; (±)-1,2-propylenediamine, 1,2-pn; isobutylenediamine, ibn; trans-(±)-1,2-diaminocyclohexane, dach; 1,3-propylenediamine, 1,3-pd; 2,2- dimethyl-1,3-propylenediamine, 2,2-diMe-1,3-pd; (±)-1,3-pentanediamine,1,3-pnd, and X is a bridging pyrazine (pz) or pyridazine (pydz) ligand) have been synthesized and characterized. The antitumor potential of these complexes against CT26 cells were determined by in vitro and in vivo assays. A murine model
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Malia, R. G., H. J. Kennedy, D. R. Triger, and F. E. Preston. "PROTECTIVE EFFECT OF VITAMIN K AGAINST ACETAMINOPHEN (PARACETAMOL) TOXICITY IN THE HAMSTER." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644341.

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We have previously reported that paracetamol may interfere with the metabolism of vitamin K via the vitamin K 2, 3-epoxide cycle. (Thrombosis and Haemostasis, 54,205,1985). In this study we have examined the effects of large doses of vitamin K on experimentally induced paracetamol hepatic necrosis in a hamster model. Paracetamol (1.2g/Kg) when given by gavage to 24 hamsters (Group I) resulted in 10 deaths (42%) at 24 hours. Simultaneous administration of lmg vitamin K intraperitoneally (Group II) reduced mortality to 3/24 (12%); mortality was 2/24 (8%) if vitamin K was given 4 hours after the
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"Hepatoprotective Effect of Typhonium flagelliforme against Thioacetamide Induced Liver Cirrhosis in Rats." In 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.643.

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Typhonium flagelliforme (T. flagelliforme) was utilized in outdated medication for handling numerous syndromes. This study aimed to investigate hepatoprotection effects of T. flagelliforme against thioacetamide (TAA) hepatotoxicity in rodents.Thirty rodents arbitrarily separated five clusters. Collection 1 was intraperitoneally injected distilled water thrice /week and fed (p.o) daily with 10% Tween 20 to eight weeks. Collection 2-5 i.p. injected with 200 mg/kg TAA three times thrice per week for 8 weeks and fed 10% Tween 20, 50 mg/kg silymarin, 250 and 500 mg/kg of T. flagelliforme extract da
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Al-dosari, Aldana, Nadin Younes, and Gheyath Nasrallah. "Ecotoxicological assessment of two surfactant on the emryonic development." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0149.

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In this study, zebrafish (Danio rerio) embryos was served as a model for marine fauna to determine if there is any potential of organ-specific toxicity (neuromuscular, hepatic, cytotoxic, and cardiac) caused by Silicone-Q-22 and Ploy-Q-47. as both surfactants are considered eco-friendly corrosion inhibitors. The calculated LC50 of Silicon-Q-22 and Poly-Q-47 was 22.36 and 8.28 mg/L, respectively. At NOEC both surfactants had resulted in teratogenic defects and cardiotoxicity, but only poly Q-47 resulted in neurotoxicity.
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Fatima, S., N. Sheikh, and A. Tayyeb. "Investigation of hepatic and renal toxicity induced by omeprazole in CCl4 injury mouse model." In 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677070.

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Zhao Yong-Mei, Chen Ai-Xia, and Du Xi-Chun. "Effects of the battery liquid on chromosomal aberration, Hepatic and Nephric toxicity in Mice." In 2011 International Symposium on Water Resource and Environmental Protection (ISWREP). IEEE, 2011. http://dx.doi.org/10.1109/iswrep.2011.5893487.

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Tsakonas, Georgios, Rodopi Stamatiou, Anastasios Kontses, et al. "Exhaust Emission Toxicity Assessment for Two Different Modern Gasoline Vehicle Technologies." In 16th International Conference on Engines & Vehicles. SAE International, 2023. http://dx.doi.org/10.4271/2023-24-0117.

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&lt;div class="section abstract"&gt;&lt;div class="htmlview paragraph"&gt;Cellular exposure to diluted exhaust gas is a promising method to assess the adverse effects of road traffic on human health. To fully understand the potential correlation between emission patterns, vehicle technologies and cellular toxicity in real-world scenarios, further research is needed. This study evaluates the toxicity of exhaust emissions from two advanced technology vehicles in real-world driving conditions. One vehicle is a gasoline direct injection (GDI) with a particle filter (GPF), while the other is a gaso
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Harvey, Shalon L., Jessica L. Beers, and Klarissa D. Jackson. "Evaluation of the Time- and Dose-Dependent Toxicity of Sunitinib Using a 3-D Human Hepatic Spheroid Model." In ASPET 2023 Annual Meeting Abstracts. American Society for Pharmacology and Experimental Therapeutics, 2023. http://dx.doi.org/10.1124/jpet.122.241590.

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Parisi, S., M. C. Ditto, M. Scarati, M. Priora, and F. Enrico. "AB0499 Evaluation of the effectiveness of metaxodine in the hepatic toxicity due to methotrexate in patients with rheumatoid arthritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.6253.

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Ma, Liang, Jeremy Barker, Changchun Zhou, Biaoyang Lin, and Wei Li. "A Perfused Two-Chamber System for Anticancer Drug Screening." In ASME 2010 International Manufacturing Science and Engineering Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/msec2010-34326.

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A cell culture microfluidic device has been developed to test the cytotoxicity of anticancer drugs while reproducing multi-organ interactions in vitro. Cells were cultured in separate chambers representing the liver and tumor. The two chambers were connected through a channel to mimick the blood flow. Glioblastoma (GBM) cancer cells (M059K) and hepatoma cells (HepG2) were cultured in the tumor and the liver chambers, respectively. The cytotoxic effect of cancer treatment drug Temolozomide (TMZ) was tested using this two chamber system. The experimental results showed that with the liver cells,
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Reports on the topic "Hepato toxicity"

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Thevenin, E., and J. McMillian. Trichloroethylene toxicity in a human hepatoma cell line. Office of Scientific and Technical Information (OSTI), 1994. http://dx.doi.org/10.2172/121308.

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Lawanprasert, Somsong, Chaiyo Chaichantipyuth, Supatra Srichairat, Nuansri Niwattisaiwong, and Laddawal Phivthong-ngam. Subchronic exposure of Pueraria mirifica in normal - and high cholesterol diet fed rats : influence on hepatic cytochrome P450, lipid profile and toxicity. Chulalongkorn University, 2004. https://doi.org/10.58837/chula.res.2004.28.

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Pueraria mirifica airy shaw and suvatabandhu, know locally as Kwao Keur, is a plant in family Leguminosae. In this study, effects of P.mirifica on hepatic cytochrome P450 (CYP), serum lipid profile and subchronic toxicity were investigated in male Wistar rats. Rats were randomly divided into four treatment groups as following: normal diet-fed group; normal diet-fed supplemented with P.mirifica group; high cholesterol diet-fed group; high cholesterol diet-fed supplemented with P.mirifica group. Each group comprised 10 rats. P.mirifica was administered orally at a dosage of 100 mg/kg/day for 90
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