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Relevant bibliographies by topics / Hypercapnic

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Academic literature on the topic 'Hypercapnic'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Hypercapnic"

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Ketabchi, Farzaneh, Bakytbek Egemnazarov, Ralph T. Schermuly, et al. "Effects of hypercapnia with and without acidosis on hypoxic pulmonary vasoconstriction." American Journal of Physiology-Lung Cellular and Molecular Physiology 297, no. 5 (2009): L977—L983. http://dx.doi.org/10.1152/ajplung.00074.2009.

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Acute respiratory disorders and permissive hypercapnic strategy may lead to alveolar hypoxia and hypercapnic acidosis. However, the effects of hypercapnia with or without acidosis on hypoxic pulmonary vasoconstriction (HPV) and oxygen diffusion capacity of the lung are controversial. We investigated the effects of hypercapnic acidosis and hypercapnia with normal pH (pH corrected with sodium bicarbonate) on HPV, capillary permeability, gas exchange, and ventilation-perfusion matching in the isolated ventilated-perfused rabbit lung. No alteration in vascular tone was noted during normoxic hyperc

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Higgins, Brendan D., Joseph Costello, Maya Contreras, Patrick Hassett, Daniel O' Toole, and John G. Laffey. "Differential Effects of Buffered Hypercapnia versus Hypercapnic Acidosis on Shock and Lung Injury Induced by Systemic Sepsis." Anesthesiology 111, no. 6 (2009): 1317–26. http://dx.doi.org/10.1097/aln.0b013e3181ba3c11.

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Background Acute hypercapnic acidosis protects against lung injury caused by nonseptic insults and after both pulmonary and systemic sepsis. The authors wished to dissect the contribution of the acidosis versus hypercapnia per se to the effects of hypercapnic acidosis on the hemodynamic profile and severity of lung injury induced by systemic sepsis. Methods In the hypercapnic acidosis series, adult male Sprague-Dawley rats were randomized to normocapnia or hypercapnic acidosis-produced by adding 5% carbon dioxide to the inspired gas-and cecal ligation and puncture performed. In the buffered hy

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Talor, Z., W. C. Yang, J. Shuffield, E. Sack, and J. A. Arruda. "Chronic hypercapnia enhances Vmax of Na-H antiporter of renal brush-border membranes." American Journal of Physiology-Renal Physiology 253, no. 3 (1987): F394—F400. http://dx.doi.org/10.1152/ajprenal.1987.253.3.f394.

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Chronic hypercapnia is associated with increased proximal HCO3 reabsorption that is thought to be mediated by a Na-H antiporter. We hypothesized that chronic hypercapnia would be associated either with increased Vmax or with decreased Km of the Na-H antiporter. To test this hypothesis we made rabbits hypercapnic for 48 h by exposure to 10% CO2. In both control and hypercapnic animals, cortical luminal membranes were enriched over the homogenate 16-fold in alkaline phosphatase and 10-fold in maltase activity. The kinetic activity of the Na-H antiporter was measured by the dissipation of the que

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Caples, Sean M., Deborah L. Rasmussen, Won Y. Lee, Marla Z. Wolfert, and Rolf D. Hubmayr. "Impact of buffering hypercapnic acidosis on cell wounding in ventilator-injured rat lungs." American Journal of Physiology-Lung Cellular and Molecular Physiology 296, no. 1 (2009): L140—L144. http://dx.doi.org/10.1152/ajplung.90339.2008.

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We measured the effects of raising perfusate pH on ventilator-induced cell wounding and repair in ex vivo mechanically ventilated hypercapnic rat lungs. Lungs were randomized to one of three perfusate groups: 1) unbuffered hypercapnic acidosis, 2) bicarbonate-buffered hypercapnia, or 3) tris-hydroxymethyl aminomethane (THAM)-buffered hypercapnia. The membrane-impermeant label propidium iodide was added to the perfusate either during or after injurious ventilation providing a means to subsequently identify transiently wounded and permanently wounded cells in optical sections of subpleural alveo

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Raff, H., C. W. Kane, and C. E. Wood. "Arginine vasopressin responses to hypoxia and hypercapnia in late-gestation fetal sheep." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 260, no. 6 (1991): R1077—R1081. http://dx.doi.org/10.1152/ajpregu.1991.260.6.r1077.

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The purpose of this study was to determine the interaction of hypoxia and hypercapnia in the control of arginine vasopressin (AVP) secretion in fetal sheep and to determine the role of the peripheral arterial chemoreceptors in that response. We measured the plasma AVP response to hypercapnia and/or hypoxia in catheterized intact or sinoaortic-denervated fetal sheep between 123 and 144 days of gestation. Ewes were exposed to the following inspired gases: two successive 30-min periods of normocapnic normoxia, 30 min of normocapnic normoxia followed by 30 min of normocapnic hypoxia, two successiv

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Tregub, Pavel P., Vladimir P. Kulikov, Irada Ibrahimli, et al. "Molecular Mechanisms of Neuroprotection after the Intermittent Exposures of Hypercapnic Hypoxia." International Journal of Molecular Sciences 25, no. 7 (2024): 3665. http://dx.doi.org/10.3390/ijms25073665.

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The review introduces the stages of formation and experimental confirmation of the hypothesis regarding the mutual potentiation of neuroprotective effects of hypoxia and hypercapnia during their combined influence (hypercapnic hypoxia). The main focus is on the mechanisms and signaling pathways involved in the formation of ischemic tolerance in the brain during intermittent hypercapnic hypoxia. Importantly, the combined effect of hypoxia and hypercapnia exerts a more pronounced neuroprotective effect compared to their separate application. Some signaling systems are associated with the predomi

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Clayson, Maxwell S., Maiah E. M. Devereaux, and Matthew E. Pamenter. "Neurokinin-1 receptor activation is sufficient to restore the hypercapnic ventilatory response in the Substance P-deficient naked mole-rat." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 318, no. 4 (2020): R712—R721. http://dx.doi.org/10.1152/ajpregu.00251.2019.

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Naked mole-rats (NMRs) live in large colonies within densely populated underground burrows. Their collective respiration generates significant metabolic carbon dioxide (CO2) that diffuses slowly out of the burrow network, creating a hypercapnic environment. Currently, the physiological mechanisms that underlie the ability of NMRs to tolerate environmental hypercapnia are largely unknown. To address this, we used whole-body plethysmography and respirometry to elucidate the hypercapnic ventilatory and metabolic responses of awake, freely behaving NMRs to 0%–10% CO2. We found that NMRs have a blu

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Perry, S. F., S. Malone, and D. Ewing. "Hypercapnic acidosis in the rainbow trout (Salmo gairdneri). II. Renal ionic fluxes." Canadian Journal of Zoology 65, no. 4 (1987): 896–902. http://dx.doi.org/10.1139/z87-143.

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The involvement of the rainbow trout kidney in acid–base regulation was assessed before, during, and after 72 h of exposure to external hypercapnia (1% CO2). Hypercapnia caused a significant elevation of renal acid excretion throughout most of the hypercapnic period, which could account for 16% of the overall compensatory increase in plasma bicarbonate concentration. The two predominant urine buffers, phosphate and ammonia, both increased markedly during hypercapnia, thereby preventing reductions in urine pH during the period of enhanced urinary acidification. Stimulation of tubular H+ secreti

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Peng, Shin-Lei, Harshan Ravi, Min Sheng, Binu P. Thomas, and Hanzhang Lu. "Searching for a truly “iso-metabolic” gas challenge in physiological MRI." Journal of Cerebral Blood Flow & Metabolism 37, no. 2 (2016): 715–25. http://dx.doi.org/10.1177/0271678x16638103.

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Hypercapnia challenge (e.g. inhalation of CO2) has been used in calibrated fMRI as well as in the mapping of vascular reactivity in cerebrovascular diseases. An important assumption underlying these measurements is that CO2 is a pure vascular challenge but does not alter neural activity. However, recent reports have suggested that CO2 inhalation may suppress neural activity and brain metabolic rate. Therefore, the goal of this study is to propose and test a gas challenge that is truly “iso-metabolic,” by adding a hypoxic component to the hypercapnic challenge, since hypoxia has been shown to e

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Nichol, Alistair D., Donall F. O'Cronin, Finola Naughton, Natalie Hopkins, John Boylan, and Paul McLoughlin. "Hypercapnic Acidosis Reduces Oxidative Reactions in Endotoxin-induced Lung Injury." Anesthesiology 113, no. 1 (2010): 116–25. http://dx.doi.org/10.1097/aln.0b013e3181dfd2fe.

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Background Hypercapnic acidosis frequently occurs when patients with acute lung injury are initially ventilated with low tidal volume "protective" strategies. Hypercapnic acidosis per se, in the absence of any change in tidal volume or airway pressure, is protective when instituted before the onset of injury. However, the mechanisms by which hypercapnic acidosis confers this protection are incompletely understood, in particular, the effects on pulmonary oxidative reactions, which are potent mediators of tissue damage, have not been previously examined in vivo. Methods After anesthesia, tracheo

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Dissertations / Theses on the topic "Hypercapnic"

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Heinert, Gideon. "Hypoxic and hypercapnic cerebral vasodilatation." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.389003.

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Kelly, Julia Louise. "Autotitrating non-invasive ventilation (NIV) in patients with hypercapnic ventilatory failure." Thesis, Imperial College London, 2015. http://hdl.handle.net/10044/1/32008.

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Non-invasive ventilation (NIV) is an evidenced based treatment of alveolar hypoventilation in patients with hypercapnic respiratory failure (HRF). Volume assured pressure support (VAPS) is a new mode of NIV that aims to maintain alveolar ventilation (VA) by autotitration of the pressure support (PS) delivered to a patient in response to changes in respiratory physiology. The overall aim of this thesis was to investigate the use of VAPS ventilation in patients with chronic HRF during wakefulness and sleep, specifically in the detection and treatment of acute exacerbations. Specific aims were to

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Elhossaini, Hawraa. "Proliferation and Primitivity of Hematopoietic Progenitor Cells in Hypoxic Hypercapnic Conditions." Thesis, The University of Sydney, 2022. https://hdl.handle.net/2123/30010.

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This thesis aims to improve the in vitro cultivation of hematopoietic stem cells (HSCs) by exploring the effects of various biophysical and biochemical factors. The study uses KG-1a cells as a model for HSCs and evaluates the impact of different O2/CO2 conditions on apoptosis, proliferation, and primitivity. Additionally, the effect of differently-treated cuttlefish bone (CB) on mouse bone marrow hematopoietic cells is examined. The results show that combining hypoxia and hypercapnia is more effective in maintaining CD34+ cell count, survival, and primitivity compared to other gas combinations

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Tinworth, Kellie. "Arousal, Sleep and Cardiovascular Responses to Intermittent Hypercapnic Hypoxia in Piglets." Thesis, The University of Sydney, 2003. http://hdl.handle.net/2123/1116.

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Clinical studies have demonstrated an arousal deficit in infants suffering Obstructive Sleep Apnoea (OSA), and that treatment to alleviate the symptoms of OSA appears to reverse the deficit in arousability. Some sudden infant deaths are thought to be contingent upon such an arousal deficit. This research utilised young piglets during early postnatal development, and exposed them to intermittent hypercapnic hypoxia (IHH) as a model of clinical respiratory diseases. Arousal responses of control animals were compared to the animals exposed to IHH. Comparisons were also made between successive exp

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Tinworth, Kellie. "Arousal, Sleep and Cardiovascular Responses to Intermittent Hypercapnic Hypoxia in Piglets." University of Sydney, 2003. http://hdl.handle.net/2123/1116.

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Master of Science (Medicine)<br>Clinical studies have demonstrated an arousal deficit in infants suffering Obstructive Sleep Apnoea (OSA), and that treatment to alleviate the symptoms of OSA appears to reverse the deficit in arousability. Some sudden infant deaths are thought to be contingent upon such an arousal deficit. This research utilised young piglets during early postnatal development, and exposed them to intermittent hypercapnic hypoxia (IHH) as a model of clinical respiratory diseases. Arousal responses of control animals were compared to the animals exposed to IHH. Comparisons were

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Nickol, Annabel Henrietta. "Mechanism of action of nocturnal non-invasive ventilation in chronic hypercapnic respiratory failure." Thesis, Imperial College London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411971.

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Oscroft, Nicholas Stephen. "The effects of long-term non-invasive positive pressure ventilation in hypercapnic chronic obstructive pulmonary disease." Thesis, University of London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.542958.

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Ciarlone, Geoffrey Edward. "Hypercapnic Hyperoxia Increases Free Radical Production and Cellular Excitability in Rat Caudal Solitary Complex Brain Slice Neurons." Scholar Commons, 2016. http://scholarcommons.usf.edu/etd/6481.

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The caudal solitary complex (cSC) is a cardiorespiratory integrative center in the dorsal medulla oblongata that plays a vital role in the central CO2-chemoreceptive network. Neurons in this area respond to hypercapnic acidosis (HA) by a depolarization of the membrane potential and increase in firing rate, however a definitive mechanism for this response remains unknown. Likewise, CO2-chemoreceptive neurons in the cSC respond to hyperoxia in a similar fashion, but via a free radical mediated mechanism. It remains unknown if the response to increased pO2 is merely an increase in redox signaling

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Jeton, Florine. "Etude du rôle de l’Erythropoïétine et des systèmes de neurotransmission dans la mise en place des réponses ventilatoires à l’hypoxie et à l’hypercapnie." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCD078/document.

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Lors de variations de PO2 et PCO2, différents mécanismes se mettent en place afin demaintenir l’oxygénation des tissus, notamment au niveau du métabolisme et de la ventilation. En casde stimulation hypoxique ou hypercapnique, on observe alors une réponse ventilatoire, caractériséepar une augmentation progressive de la ventilation. Parmi les facteurs qui influencent la réponse àl’hypoxie, on trouve l’érythropoïétine (Epo) qui, en plus de son rôle dans l’érythropoïèse, possèded’autres rôles, notamment au sein du système nerveux central. Cette thèse présente l’étude del’implication de l’Epo et de

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Nanagas, Vivian C. "Clamping of Intracellular pH in Neurons from Neonatal Rat Brainstem during Hypercapnia." Wright State University / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=wright1246393988.

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Books on the topic "Hypercapnic"

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Piracha, Kashif. Hypercapnic Respiratory Failure. Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-36128-9.

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Al-Abdulmunem, M. A. Effects of hypoxia and hypercapnia associated withcontactlenswearon corneal epithelium. UMIST, 1994.

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Jarsky, Tim M. The effects of hypoxia and hypercapnia on hamster activity rhythms. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Peever, John H. Day-night differences in ventilation, metabolism, and body temperature during normoxia, hypoxia and hypercapnia in the awake adult rat. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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U, Sliwka, and United States. National Aeronautics and Space Administration., eds. Effects of sustained low-level elevations of carbon dioxide on cerebral blood flow and autoregulation of the intracerebral arteries in humans. National Aeronautics and Space Administration, 1996.

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United States. National Aeronautics and Space Administration., ed. "CO₂-O₂ interactions in extension of tolerance to acute hypoxia": Final report. National Aeronautics and Space Administration, 1995.

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United States. National Aeronautics and Space Administration., ed. "CO₂-O₂ interactions in extension of tolerance to acute hypoxia": Final report. National Aeronautics and Space Administration, 1995.

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Rochester, Dudley. Hypercapnic Respiratory Failure. Butterworth-Heinemann, 1997.

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Rochester, Dudley F. Hypercapnic Respiratory Failure. Butterworth-Heinemann, 1990.

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Junna, Mithri R., Bernardo J. Selim, and Timothy I. Morgenthaler. Central sleep apnea and hypoventilation syndromes. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0018.

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Sleep disordered breathing (SDB) may occur in a variety of ways. While obstructive sleep apnea is the most common of these, this chapter reviews the most common types of SDB that occur independently of upper airway obstruction. In many cases, there is concurrent upper airway obstruction and neurological respiratory dysregulation. Thus, along with attempts to correct the underlying etiologies (when present), stabilization of the upper airway is most often combined with flow generators (noninvasive positive pressure ventilation devices) that modulate the inadequate ventilatory pattern. Among the

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Book chapters on the topic "Hypercapnic"

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Aydin, Ani, Daniel Joseph, and Melissa Joseph. "Hypercapnic Respiratory Failure." In Simulation in EMS and Critical Care Transport. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-40090-2_18.

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Pogach, Melanie, and Robert Thomas. "Hypercapnic Obstructive Sleep Apnea." In Complex Sleep Breathing Disorders. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57942-5_1.

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Yerosimou, Fotis, Pagona Kamparoudi, and Nikolaos Barbetakis. "Hypercapnic Chronic Obstructive Pulmonary Disorder." In Upper Airway Disorders and Noninvasive Mechanical Ventilation. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-32487-1_27.

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Pearce, William J. "Mechanisms of Hypoxic and Hypercapnic Cerebral Vasodilatation." In The Human Brain Circulation. Humana Press, 1994. http://dx.doi.org/10.1007/978-1-4612-0303-2_15.

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Qasrawi, Shaden O., and Ahmed S. BaHammam. "NIV in Type 2 (Hypercapnic) Acute Respiratory Failure." In Mechanical Ventilation in the Critically Ill Obese Patient. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-49253-7_24.

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Nicolini, Antonello, Josef Lucazovitch, and Stephan Walterspacher. "High-Intensity Noninvasive Ventilation in Stable Hypercapnic COPD." In Noninvasive Ventilation in Sleep Medicine and Pulmonary Critical Care. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42998-0_5.

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Lun, Chung-Tat, and Chung-Ming Chu. "Noninvasive Ventilation Withdrawal Methodology After Hypercapnic Respiratory Failure." In Noninvasive Mechanical Ventilation and Difficult Weaning in Critical Care. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-04259-6_14.

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Kattih, Zein, Erica Altschul, and Bushra Mina. "Noninvasive Positive Pressure Ventilation (NIPPV) in Hypercapnic Respiratory Failure." In Noninvasive Ventilation in Sleep Medicine and Pulmonary Critical Care. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42998-0_34.

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Witt, Ashleigh, and Alistair Miller. "Neuromuscular Disease as a Cause of Hypercapnic Respiratory Failure." In Beyond Evidence-Based Medicine. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-4440-8_62.

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Zhou, Lan, Patrick Kwon, and Susan C. Shin. "A 58-Year-Old Man with Hypercapnic Respiratory Failure." In A Case-Based Guide to Neuromuscular Pathology. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-25682-1_16.

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Conference papers on the topic "Hypercapnic"

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Bräunlich, Jens, Kristin Turba, and Hubert Wirtz. "Predictors of persistent hypercapnia after an hypercapnic AECOPD." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa1777.

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Bräunlich, J., K. Turba, and H. Wirtz. "Predictors of persistent hypercapnia after a hypercapnic COPD exacerbation." In 61. Kongress der Deutschen Gesellschaft für Pneumologie und Beatmungsmedizin e.V. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1723341.

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Rötting, Matthias, Markus Polke, Katharina Kriegsmann, et al. "Impact of endoscopic valve therapy on hypercapnia in patients with chronic hypercapnic failure based on a severe lung emphysema." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3777.

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Sahin, Hulya, Ilknur Naz, Sevcan Guney, Nimet Aksel, and Fevziye Tuksavul. "Outcomes of pulmonary rehabilitation in hypercapnic COPD patients." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2217.

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El-Nayal, A., G. Raynes, C. Aday, S. Lutchegadoo, and S. Iyer. "P120 Outcomes from mild acute hypercapnic respiratory failure." In British Thoracic Society Winter Meeting 2024, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 27 to 29 November 2024, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2024. http://dx.doi.org/10.1136/thorax-2024-btsabstracts.281.

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Faverio, Paola, Anna Stainer, Francesca D'Arcangelo, et al. "Non-invasive ventilation weaning in acute hypercapnic respiratory failure." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2165.

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Killebrew, S., H. Jenad, and P. C. Gay. "Amyotrophic Lateral Sclerosis Disguised as Hypercapnic Hypoxemic Respiratory Failure." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6749.

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Bräunlich, Jens, and Hubert Wirtz. "Nasal high-flow in acute hypercapnic exacerbation of COPD." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4035.

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Braunlich, J., and H. R. Wirtz. "Nasal High-Flow in Acute Hypercapnic Exacerbation of COPD." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2736.

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Gilman, Sean A., Lewis Adams, Richard M. Schwartzstein, Carl R. O'Donnell, Robert W. Lansing, and Robert B. Banzett. "Morphine Alleviates Laboratory Dyspnea AND DIMINISHES HYPERCAPNIC VENTILATORY RESPONSE." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4804.

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Reports on the topic "Hypercapnic"

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Hariswar, Pari Thenmozhi, Ramanathan Venkateswaran, George Melvin, kshirsagar Shivani, and M. Rajeswari. Acetazolamide in weaning from mechanical ventilation in hypercapnic respiratory failure. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.7.0108.

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Selkirk, Alaleh, Barbara Shykoff, and James Briggs. Cognitive Effects of Hypercapnia on Immersed Working Divers. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada561029.

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Zhuo, Guifeng, Hengwang Yu, Ran Liao, et al. Auricular point pressing therapy for obstructive sleep apnea hypoventilation syndrome: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0015.

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Review question / Objective: Patients with obstructive sleep apnea hypoventilation syndrome (OSAHS) suffer from repeated hypoxemia, hypercapnia, and sleep structure disorders at night, leading to daytime lethargy and complications of heart, brain, lung, and blood vessel damage, which seriously affect their quality of life and life span. Clinical studies have shown that auricular point pressing therapy has an excellent therapeutic effect on OSAHS, and has the potential to be a complementary and alternative therapy for patients with OSAHS. Currently, systematic reviews and meta-analyses evaluati

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Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desatura

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