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Books on the topic 'Immune related diseases'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

National Cancer Institute (U.S.), ed. Understanding cancer and related topics: Understanding gene testing. National Cancer Institute, 2011.

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2

Colloque, des "Cent gardes" (2nd 1987 Marnes-la-Coquette France). Retroviruses of human A.I.D.S. and related animal diseases: 2e collogue des "cent gardes", 28-30 octobre 1987, Marnes-la-Coquette/Paris, France. Pasteur vaccins, 1988.

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3

1929-, Newcombe David S., Rose Noel R, and Bloom John C, eds. Clinical immunotoxicology. Raven Press, 1992.

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4

L, Petrakis Peter, American Medical Society on Alcoholism and Other Drug Dependencies., National Council on Alcoholism, and AIDS and Chemical Dependency Forum (1986 : San Francisco, Calif.), eds. Acquired Immune Deficiency Syndrome and chemical dependency: Report of symposium. U.S. Dept. of Health and Human Services, Public Health Service, Alcohol, Drug Abuse, and Mental Health Administration, National Institute on Alcohol Abuse and Alcoholism, 1987.

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5

Okolelova, Tamara, Syergyey YEngashyev, and Ivan Yegorov. Poultry farming: current questions and answers. Publishing Center RIOR, 2020. http://dx.doi.org/10.29039/02023-4.

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In the book in the form of questions and answers considerable attention is paid to data on the needs of all types of poultry in nutritional, mineral and biologically active substances, taking into account age of poultry. The characteristic of the main feed products is given, and the rational norms for including them in mixed feed for poultry are indicated. The role of vitamins, macro- and microelements, enzyme preparations, probiotics, prebiotics, antibiotics, organic acids, antioxidants, emulsifiers and other sources of biologically active substances in poultry nutrition is shown. Both nutrit

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6

Peter, Bridge T., Mirsky Allan F, and Goodwin Frederick K. 1936-, eds. Psychological, neuropsychiatric, and substance abuse aspects of AIDS. Raven Press, 1988.

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7

1931-, Friedman Herman, and Symposium on AIDS, Drugs of Abuse, and the Neuroimmune Axis (3rd : 1995 : San Diego, Calif.), eds. AIDS, drugs of abuse, and the neuroimmune axis. Plenum Press, 1996.

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8

Barbouche, Mohamed-Ridha, and Brian Stephen Eley, eds. Immune-Related Non-Communicable Diseases in Africa. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88974-419-0.

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9

Lee, Kyung-Yil, Hiromichi Hamada, and Miika Kaleva Arvonen, eds. Infection-Related Immune-Mediated Diseases and Microbiota. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-744-7.

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10

Maghazachi, Azzam A., ed. Immunomodulatory Effects of Drugs for Treatment of Immune-Related Diseases. Frontiers Media SA, 2017. http://dx.doi.org/10.3389/978-2-88945-288-0.

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11

Konlee, Mark. How to Reverse Immune Dysfunction : A Nutrition Manual for Hiv, Chronic Fatigue Syndrome, Candidiasis and Other Immune Related Conditions. 7th ed. Keep Hope Alive, 1999.

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12

Konlee, Mark. How to Reverse Immune Dysfunction: A Nutrition Manual for Hiv, Chronic Fatigue Syndrome, Candidiasis and Other Immune Related Conditions. 7th ed. Keep Hope Alive, 1999.

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13

Mucke, Lennart. Astrocytes in Infectious and Immune-Related Diseases of the Cns (Neuroscience Intelligence Unit Series). R. G. Landes, 1999.

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14

Segall, Liviu, and Adrian Covic. Immune-mediated tubulointerstitial nephritis. Edited by Adrian Covic. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0093_update_001.

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Immune-mediated tubulointerstitial nephritides (TINs) are generally encountered in the context of systemic or extrarenal autoimmune diseases, such as sarcoidosis, Sjögren syndrome, systemic lupus erythematosus, inflammatory bowel disease, TIN and uveitis (TINU) syndrome, and immunoglobulin G4-related disease. The pathogenesis of these TINs is complex and more or less unclear; it usually involves leucocyte activation, autoantibodies, immune complex deposition, complement activation, and release of inflammatory cytokines and growth factors. Tubulointerstitial inflammation most commonly has a chr

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15

Jay, Taylor R., Shane M. Bemiller, Lee E. Neilson, Paul J. Cheng-Hathaway, and Bruce T. Lamb. Neuroinflammation and Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0004.

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Neuroinflammation has long been associated with many neurodegenerative diseases (NDDs). Immune-related genetic and environmental risk factors have recently been identified for NDDs, suggesting that neuroinflammation can play an active role in modifying NDD pathologies. Immune cells that underlie this neuroinflammatory response can have both beneficial and detrimental roles in NDDs. These cells can engage in clearance of debris and provide important survival factors to neighboring neurons. However, these cells can also release inflammatory molecules that promote oxidative stress and excitotoxic

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16

Friedman, Herman, Mauro Bendinelli, and Thomas W. Klein. Infectious Diseases and Substance Abuse. Springer, 2010.

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17

Friedman, Herman, Mauro Bendinelli, and Thomas W. Klein. Infectious Diseases and Substance Abuse. Springer London, Limited, 2006.

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18

(Editor), Herman Friedman, Thomas W. Klein (Editor), and Mauro Bendinelli (Editor), eds. Infectious Diseases and Substance Abuse (Infectious Agents and Pathogenesis). Springer, 2005.

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19

Jenney, Colin, and Angela Liegey Dougall. Immune System Functioning and Mental Health. Edited by Sara Maltzman. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780199739134.013.45.

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During the past few decades, research from multiple disciplines has elucidated the profound connections between the immune system and mental health. This chapter provides a review of this literature, placing emphasis on the connections between inflammation and cytokines, and stress, depression, posttraumatic stress disorder, schizophrenia, bipolar disorder, and social support. Additionally, brief overviews of the role of the natural immune system and adaptive immunity, as well as past research investigating stress are included. Further attention is focused on the physical health consequences o

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20

Santos, Dilvani Oliveira, and Paulo Renato Zuquim Antas, eds. Mitochondrial DNA and the Immuno-inflammatory Response: New Frontiers to Control Specific Microbial Diseases. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/97898150516981220301.

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Mitochondria are multifunctional organelles that actively participate in the immune-inflammatory response in various pathologies. This volume updates readers on knowledge about mitochondria function. The editors have compiled six chapters about inflammation in its broadest sense, with contributions from active groups of cell biologists, infectologists and pathologists. The chapters in this volume focus on research related to five notable diseases: (1) two diseases (one bacterial and one viral) in which the exacerbation of the inflammatory response can lead to neuropathies: leprosy (one of the

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21

Tyndall, Alan, and Jacob M. van Laar. Stem cell therapies. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0085.

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Since the start of the international project in 1997, over 1500 patients have received a haematopoietic stem cell transplant (HSCT), mostly autologous, as treatment for a severe autoimmune disease, with overall 85% 5-year survival and 43% progression-free survival. Around 30% of patients in all disease subgroups had a complete response, often durable despite full immune reconstitution. In many cases, e.g. systemic sclerosis, morphological improvement such as reduction of skin collagen and normalization of microvasculature was documented, beyond any predicted known effects of intense immunosupp

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22

Tyndall, Alan, and Jacob M. van Laar. Stem cell therapies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0085_update_003.

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Since the start of the international project in 1997, over 2000 patients have received a haematopoietic stem cell transplant (HSCT), mostly autologous, as treatment for a severe autoimmune disease, with overall 85% 5-year survival and 43% progression-free survival. Around 30% of patients in all disease subgroups had a complete response, often durable despite full immune reconstitution. In many cases, e.g. systemic sclerosis, morphological improvement such as reduction of skin collagen and normalization of microvasculature was documented, beyond any predicted known effects of intense immunosupp

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23

Kelly, Patricia, and Susan Holman. Primary Care of Women and Children with HIV Infection: A Multidisciplinary Approach (Jones and Bartlett Books on Oncology and Hiv-Related Illnesses). Jones & Bartlett Publishers, 1995.

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24

Dembic, Zlatko. Cytokines of the Immune System: The Role of Cytokines in Disease Related to Immune Response. Elsevier Science & Technology Books, 2015.

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25

Cytokines of the Immune System: The Role of Cytokines in Disease Related to Immune Response. Elsevier Science & Technology Books, 2015.

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26

Bielekova, Bibiana, Gary Birnbaum, and Robert P. Lisak, eds. Neuroimmunology. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190050801.001.0001.

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This book provides clinical and supporting scientific background on a diverse group of neurological disorders in an expanding field of neurology, that of neuroimmunology. It includes chapters on multiple sclerosis and related disorders in adults and children, neuromyelitis optica spectrum disorder, Guillain-Barré syndrome, chronic inflammatory deymyelinating polyradiculoneuropathy and variants, immune-mediated disorders of the neuromuscular junction, inflammatory myopathies, paraneoplastic disorders and autoimmune encephalitities, and neurologic manifestations of systemic immune-mediated disea

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27

Straub, Rainer H. Neuroendocrine system. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0022.

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Endocrine abnormalities are very common in patients with chronic autoimmune rheumatic diseases (CARDs) due to the systemic involvement of the central nervous system and endocrine glands. In recent years, the response of the endocrine (and also neuronal) system to peripheral inflammation has been linked to overall energy regulation of the diseased body and bioenergetics of immune cells. In CARDs, hormonal and neuronal pathways are outstandingly important in partitioning energy-rich fuels from muscle, brain, and fat tissue to the activated immune system. Neuroendocrine regulation of fuel allocat

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28

Straub, Rainer H. Neuroendocrine system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0022_update_002.

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Endocrine abnormalities are very common in patients with chronic autoimmune rheumatic diseases (CARDs) due to the systemic involvement of the central nervous system and endocrine glands. In recent years, the response of the endocrine (and also neuronal) system to peripheral inflammation has been linked to overall energy regulation of the diseased body and bioenergetics of immune cells. In CARDs, hormonal and neuronal pathways are outstandingly important in partitioning energy-rich fuels from muscle, brain, and fat tissue to the activated immune system. Neuroendocrine regulation of fuel allocat

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29

Straub, Rainer H. Neuroendocrine system. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199642489.003.0022_update_003.

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Endocrine abnormalities are very common in patients with chronic autoimmune rheumatic diseases (CARDs) due to the systemic involvement of the central nervous system and endocrine glands. In recent years, the response of the endocrine (and also neuronal) system to peripheral inflammation has been linked to overall energy regulation of the diseased body and bioenergetics of immune cells. In CARDs, hormonal and neuronal pathways are outstandingly important in partitioning energy-rich fuels from muscle, brain, and fat tissue to the activated immune system. Neuroendocrine regulation of fuel allocat

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30

Ogunbambi, Olabambo, and Yusuf I. Patel. Parasitic infection. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0105.

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Parasitic infections remain prevalent, particularly in the developing world. However, increasing global travel poses a risk of contracting parasitic infections while travelling through endemic areas and therefore all medical practitioners should remain aware of parasitic infections and investigate for them when appropriate. Increased understanding of the relationship of parasites with the immune system has led to some progress with therapeutics but this still lags behind other infectious diseases. In this chapter we outline the musculoskeletal manifestations of parasitic infection and updated

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31

Tillema, Jan-Mendelt, J. Graves, L. A. Benson, et al. Pediatric Multiple Sclerosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0022.

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Increased interest in pediatric-onset multiple sclerosis (MS) has contributed to improved knowledge of the presentation and evolution of central nervous system demyelinating diseases in childhood. This chapter reviews the unique features and challenges related to pediatric MS. The close proximity of pediatric MS to the biological onset of the disease provides a unique window into disease pathogenesis at stages of life when innate and adaptive immune pathways are still maturing. It is expected that the interplay between genetics, epigenetics, environmental exposures, and the maturing central ne

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32

Voll, Reinhard E., and Barbara M. Bröker. Innate vs acquired immunity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0048.

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The innate and the adaptive immune system efficiently cooperate to protect us from infections. The ancient innate immune system, dating back to the first multicellular organisms, utilizes phagocytic cells, soluble antimicrobial peptides, and the complement system for an immediate line of defence against pathogens. Using a limited number of germline-encoded pattern recognition receptors including the Toll-like, RIG-1-like, and NOD-like receptors, the innate immune system recognizes so-called pathogen-associated molecular patterns (PAMPs). PAMPs are specific for groups of related microorganisms

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33

Sousek, Alexandra, and Mehdi Tafti. The genetics of sleep. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0005.

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Although there is strong evidence for a genetic contribution to inter-individual variations in sleep, the underlying factors and their interaction remain largely elusive. Much effort has been expended in studying genetic variations contributing to circadian and sleep phenotypes, the individual pattern of the human sleep EEG, reactions to sleep loss, and the pathophysiology of sleep-related disorders. Certain sleep-related diseases may be caused by single genes, while the etiology of others seems to be variable and complex. This is especially the case when the immune system is involved. This ch

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34

Who Expert Committee on Drug Dependence: Thirty Third Report (Technical Report Series). World Health Organization, 2003.

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35

Coyle, Patricia K. Immune-mediated Disorders of the Central Nervous System. Edited by Emma Ciafaloni, Cheryl Bushnell, and Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0010.

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This chapter reviews pregnancy in multiple sclerosis (MS), neuromyelitis optica spectrum disorder (NMOSD), and acute transverse myelitis (ATM) syndrome. MS is a major acquired disease of young adults, with a rising female predominance. MS has no direct negative consequences on fertility or pregnancy. Pregnancy has a profound effect on MS, with decrease in disease activity during the last trimester counteracted by a three-month postpartum increase in disease activity. With the development of disease-modifying therapies, important questions arise about washout periods, the feasibility and risks

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36

Adam, Sheila, Sue Osborne, and John Welch. The immune system and the immunocompromised patient. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199696260.003.0013.

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This chapter discusses the physiology of the immune system and its related disorders in the critically ill patient. The causes, complications, and management of the immunocompromised patient is discussed at length, detailing the specific nursing management. It also discusses the management of autoimmune disease such as Wegener's granulomatosis and rheumatoid arthritis, and hypersensitivity reactions such as drug allergy and anaphylactic reaction. Cancer is explained in detail, including its complications, specific therapies such as stem cell transplant, and the role of the cancer patient withi

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37

Karaman, Sinem, Aleksanteri Aspelund, Michael Detmar, and Kari Alitalo. The lymphatic system. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0009.

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The lymphatic vascular system is an integral component of the circulatory system; it forms a one-way conduit that transports tissue interstitial components back to the venous circulation through lymph nodes. Lymphatic vessels extend to most tissues and contribute to the regulation of interstitial fluid homeostasis, trafficking of immune cells, and absorption of dietary fats from the gut. Developmentally, lymphatic vessels originate from embryonic veins and specialized angioblasts. A number of molecules have been identified in the commitment of endothelial cells to the lymphatic lineage, and th

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38

Miller, Aaron E., Tracy DeAngelis, Michelle Fabian, and Ilana Katz Sand. Neuroimmunology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190693190.001.0001.

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Part of the What Do I Do Now?” series, Neuroimmunology uses a case-based approach to cover common and important topics in the examination, investigation, and management of central and peripheral demyelinating diseases, vasculitis, and other immune system related neurological disorders. Each chapter provides a discussion of the diagnosis, key points to remember, and selected references for further reading. For this edition, all cases and references have been updated and new cases have been added, including POEMS, CASPR2 Antibody Syndrome, Isaac’s Syndrome, Histiocytosis, and Churg-Strauss. Neur

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39

Vinod, Nikhra. Living with COVID-19: The Nemesis, the Hubris, and the Elpis. Heighten Science Publications Inc., 2021. http://dx.doi.org/10.29328/ebook1004.

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The immuno-thrombo-inflammatory disease: COVID-19 is an acute immuno-thrombogenic inflammatory viral disease manifested by dysfunctions related to multiple organs involved in its pathogenesis. Its incidence and prevalence of is related to environmental viability of the virus, various transmission factors associated with the agent and the host, possible modes of transmission, period of infectiousness, and composition and susceptibility of the population. Whereas respiratory route is dominant mode of transmission, transmission through direct contact or fomite transmission do occur.

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40

Heeringa, Peter, and Coen A. Stegeman. The patient with vasculitis. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0158.

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Vasculitis is an inflammatory destructive process affecting blood vessels resulting in fibrinoid necrosis of the vessel wall that eventually can lead to occlusion of the vascular lumen and organ failure. Vasculitis may be the primary manifestation of a disease, or be a secondary manifestation of another underlying disease.The pathogenesis of vasculitis is complex, involving innate and adaptive immune effector mechanisms that range from cell-mediated inflammation, immune-complex-mediated inflammation, and inflammation triggered by autoantibodies. Here it is discussed with respect to general pat

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41

Berden, Jo H. M., and Jack F. M. Wetzels. Immunological investigation of the patient with renal disease. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0017.

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Laboratory techniques (electrophoresis, indirect immunofluorescence, ELISA, and immunoblotting) required for immunological investigation of the patient with renal disease are described. Renal disease-related aspects of immunoglobulins (immunoglobulin A, paraproteins, cryoglobulins), complement, antinuclear antibodies, anti-C1q antibodies, antineutrophil cytoplasmic antibodies, anti-glomerular basement membrane antibodies, antipodocyte antibodies, antiphospholipid antibodies, and antimicrobial responses (streptococci, hepatitis C, hepatitis B) are reviewed. Laboratory assays which evaluate the

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42

Stowell, Janet, and Ronan Breen. Pulmonary disease caused by non-tuberculous mycobacteria. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199657742.003.0014.

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This chapter describes a case of Mycobacterium malmoense in a male ex-smoker with chronic obstructive pulmonary disease. The approaches to a diagnosis of pulmonary non-tuberculous mycobacterial disease are discussed, including key laboratory features and associated radiological changes. The factors influencing the decision to treat and treatment regimen selected are reviewed, along with evidence from landmark trials regarding drug combinations and the role of surgery in managing non-tuberculous mycobacterial disease. This case was complicated by a secondary diagnosis of invasive aspergillosis,

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43

Gregory, Scott J. A Holistic Protocol for the Immune System: HIV/ARC/AIDS/Candidiasis/Epstein-Barr/Herpes and other opportunistic infections. 6th ed. Progressive Press, 1995.

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44

Scott, Gregory J., and Scott Gregory. A Holistic Protocol for the Immune System: HIV/ARC/AIDS, Candidiasis, Epstein-Barr, Herpes, and Other Opportunistic Infections. Progressive Press, 1991.

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45

Sprague, Stuart M., and Menaka Sarav. Chronic kidney disease-mineral and bone disorder. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0115_update_001.

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The kidneys play a critical role in maintaining normal serum calcium and phosphorus concentrations, under the regulation of three main hormones: parathyroid hormone, calcitriol, and fibroblast growth factor 23. With the progression of chronic kidney disease (CKD), most patients develop CKD–mineral and bone disorder (CKD-MBD), which is a systemic disorder involving derangement in mineral metabolism, renal osteodystrophy, and extraskeletal calcification. Disturbances in mineral metabolism develop early in CKD and include phosphate retention, hypocalcaemia, vitamin D deficiency, and hyperparathyr

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46

Pediatric ICD-10-CM. American Academy of Pediatrics, 2015. http://dx.doi.org/10.1542/9781581109016.

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In this first edition, Pediatric ICD-10-CM: A Manual for Provider-Based Coding strives to bring to the pediatric provider, coder, and biller the most accurate and easy-to use manual on ICD-10-CM yet. Composed entirely with a pediatrics focus, this manual exclusively features codes and guidelines for physician- and provider-based coding, all in a simplified yet familiar format. The full draft of the ICD-10-CM code set comes in at well over 1,000 pages. This book condenses that large and potentially cumbersome book into 400 pages of the most relevant,pediatrics-related codes and guidelines. It a

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47

Ajmal, Saira, and Zelalem Temesgen. Initiation of Antiretroviral Therapy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0020.

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The primary goal of therapy is to prevent HIV-associated morbidity and mortality. In addition to the dramatic decline in HIV-related illness and death that has been observed as a result of the introduction and expansion of combination antiretroviral therapy, evidence is emerging that uncontrolled HIV replication also has a deleterious impact on conditions that are not conventionally associated with immune deficiency. These conditions include cardiovascular disease, kidney disease, liver disease, neurologic complications, and malignancy. Studies have found an independent association between cum

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48

Rosh, Joel R., Leo A. Heitlinger, and Walter D. Rosenfeld, eds. AM:STARs: Clinical GI Challenges in the Adolescent, Vol. 27, No. 1. American Academy of Pediatrics, 2016. http://dx.doi.org/10.1542/9781581109382.

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It is now recognized that the prevalence of atopic disorders including (EoE) and immune based conditions such as celiac disease and inflammatory bowel disease are on the rise. This changing epidemiology coupled with advancements in the diagnosis of these conditions have led to greater numbers of adolescents needing treatment. Topics include: Swallowing disorders and eosinophilic esophagitis Celiac and gluten-related disorders Functional GI disorders Advances in inflammatory bowel disease Advances in hepatology Obesity Fad diets, FODMAPS Vitamin D and bone health Gut microbiome and probiotics G

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49

Acquired immune deficiency syndrome: Biological, medical, social, and legal issues. Prentice Hall, 1993.

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50

Acquired immune deficiency syndrome: Biological, medical, social, and legal issues. 2nd ed. Prentice Hall, 1996.

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