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Journal articles on the topic 'Immune related diseases'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Weksler, Marc E., Graham Pawelec, and Claudio Franceschi. "Immune therapy for age-related diseases." Trends in Immunology 30, no. 7 (2009): 344–50. http://dx.doi.org/10.1016/j.it.2009.03.011.

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2

Yang, Yaxu, Xue Han, Lijun Sun, Fangyu Shao, Yue Yin, and Weizhen Zhang. "ETS Transcription Factors in Immune Cells and Immune-Related Diseases." International Journal of Molecular Sciences 25, no. 18 (2024): 10004. http://dx.doi.org/10.3390/ijms251810004.

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The development, differentiation, and function of immune cells are precisely regulated by transcription factors. The E26 transformation-specific (ETS) transcription factor family is involved in various physiological and pathological processes by regulating cell proliferation, differentiation, and apoptosis. Emerging evidence has suggested that ETS family proteins are intimately involved in the development and function of immune cells. This review summarizes the role of the ETS family in immune cells and immune-related disorders. Seven transcription factors within the ETS family, including PU.1

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3

Singh, Dhirendra P., Prathyusha Bagam, Malaya K. Sahoo, and Sanjay Batra. "Immune-related gene polymorphisms in pulmonary diseases." Toxicology 383 (May 2017): 24–39. http://dx.doi.org/10.1016/j.tox.2017.03.020.

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4

Zeng, Jianrui, Zhaochen Ning, Yuzhong Wang, and Huabao Xiong. "Implications of CD39 in immune-related diseases." International Immunopharmacology 89 (December 2020): 107055. http://dx.doi.org/10.1016/j.intimp.2020.107055.

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5

Castiglione, F., S. Motta, F. Pappalardo, and M. Pennisi. "A Modeling Framework For Immune-related Diseases." Mathematical Modelling of Natural Phenomena 7, no. 3 (2012): 40–48. http://dx.doi.org/10.1051/mmnp/20127304.

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6

Candore, Giuseppina, Carmela Rita Balistreri, Giuseppina Colonna-Romano, et al. "Gender-Related Immune-Inflammatory Factors, Age-Related Diseases, and Longevity." Rejuvenation Research 13, no. 2-3 (2010): 292–97. http://dx.doi.org/10.1089/rej.2009.0942.

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7

de Freitas, Fábio Alessandro, Débora Levy, Cadiele Oliana Reichert, Edecio Cunha-Neto, Jorge Kalil, and Sérgio Paulo Bydlowski. "Effects of Oxysterols on Immune Cells and Related Diseases." Cells 11, no. 8 (2022): 1251. http://dx.doi.org/10.3390/cells11081251.

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Oxysterols are the products of cholesterol oxidation. They have a wide range of effects on several cells, organs, and systems in the body. Oxysterols also have an influence on the physiology of the immune system, from immune cell maturation and migration to innate and humoral immune responses. In this regard, oxysterols have been involved in several diseases that have an immune component, from autoimmune and neurodegenerative diseases to inflammatory diseases, atherosclerosis, and cancer. Here, we review data on the participation of oxysterols, mainly 25-hydroxycholesterol and 7α,25-dihydroxyc

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8

Souza, Felipe. "Neurological Diseases Associated to Immune Checkpoint Inhibitors." Neuroscience and Neurological Surgery 8, no. 6 (2021): 01–08. http://dx.doi.org/10.31579/2578-8868/177.

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The treatment for cancer has been more widespread and new therapies appear as alternatives in the area to contain the advance of the tumor, having with the immune mechanisms one of the main sources of research and study for a possible advance in the treatment. Checkpoint inhibitors (ICI) are monoclonal therapy, which act by blocking the PD-1, PD-L1 and CTLA-4 molecules, responsible for immune control. However, among the effects caused by therapy, the use of medications is associated with neurological diseases reported as an adverse effect. Neurological complications can affect both the central

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9

Yang, Dan, Shangxue Lei, Keran Pan, et al. "Application of photodynamic therapy in immune-related diseases." Photodiagnosis and Photodynamic Therapy 34 (June 2021): 102318. http://dx.doi.org/10.1016/j.pdpdt.2021.102318.

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10

Xu, Joella, Guannan Huang, and Tai Guo. "Developmental Bisphenol A Exposure Modulates Immune-Related Diseases." Toxics 4, no. 4 (2016): 23. http://dx.doi.org/10.3390/toxics4040023.

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11

Skerka, Christine, and Peter F. Zipfel. "Complement factor H related proteins in immune diseases." Vaccine 26 (December 2008): I9—I14. http://dx.doi.org/10.1016/j.vaccine.2008.11.021.

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12

Gao, Jie, Hanting Zhang, and Fang Zhang. "Research progress of TIPE2 in immune-related diseases." International Immunopharmacology 121 (August 2023): 110514. http://dx.doi.org/10.1016/j.intimp.2023.110514.

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13

Bottini, Fulvia Gloria, Neri A, Saccucci P, et al. "IMMUNE RELATED DISEASES AND THEIR RELATIONSHIP WITH THE GENETIC VARIABILITY WITHIN THE ADENOSINE DEAMINASE GENE." Journal of Advances In Allergy & Immunologic Diseases 3, no. 1 (2018): 1–8. http://dx.doi.org/10.25177/jaaid.3.1.1.

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14

Zhang, Xiao-Lian. "Roles of Glycans and Glycopeptides in Immune System and Immune-Related Diseases." Current Medicinal Chemistry 13, no. 10 (2006): 1141–47. http://dx.doi.org/10.2174/092986706776360897.

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15

Kasaie, Zahra. "The Mechanisms of Immune System Regulation by Probiotics in Immune-Related Diseases." Journal of Pharmacy and Nutrition Sciences 6, no. 3 (2016): 105–11. http://dx.doi.org/10.6000/1927-5951.2016.06.03.4.

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16

Ye, Xin, Xu-jie Zhou, and Hong Zhang. "Autophagy in Immune-Related Renal Disease." Journal of Immunology Research 2019 (November 7, 2019): 1–10. http://dx.doi.org/10.1155/2019/5071687.

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Autophagy is an important biology process, central to the maintenance of biology process in both physiological and pathological situations. It is regarded as a “double-edged sword”—exerting both protective and/or detrimental effects. These two-way effects are observed in immune cells as well as renal resident cells, including podocytes, mesangial cells, tubular epithelial cells, and endothelial cells of the glomerular capillaries. Mounting evidence suggests that autophagy is implicated in the pathological process of various immune-related renal diseases (IRRDs) as well as the kidney that under

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17

Wang, Min, Tian Tian, Shuang Yu, Na He, and Daoxin Ma. "Th17 and Treg Cells in Bone Related Diseases." Clinical and Developmental Immunology 2013 (2013): 1–10. http://dx.doi.org/10.1155/2013/203705.

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Bone-related diseases share the process of immune response that targets bone tissue and bone marrow and then induce adverse effects on structure and function. In recent years, reciprocal relationship between immune cells and bone systems has been uncovered gradually. Regulatory T (Treg) and T helper 17 (Th17) cells are newly identified subsets of CD4+ T cells, and the balance between them is particularly essential for maintaining immune homeostasis. Accumulated data have demonstrated quantitative or functional imbalance between Th17 and Treg in bone related diseases, suggesting that Th17 and T

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18

Li, Cong, Yuao Hu, Shizhen Xu, Shiyu Ouyang, and Canpeng Luo. "Lactic acid bacteria and immune system regulation and impact on related diseases." Journal of Food Science, Nutrition and Health 1, no. 1 (2023): None. http://dx.doi.org/10.54254/3029-0821/1/2023004.

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Lactobacillus is a common food-grade microorganism with good bioactivity, which has been maturely used in the food industry. The significant role of Lactobacilli in immunomodulation has gradually become a research hotspot, and their significant impact on immune diseases through the modulation of the immune system gives them great potential to become materials for the treatment of immune diseases. This paper describes the interaction between Lactobacilli and the immune system, and the effect of immunomodulation of Lactobacilli on immune diseases, with a view to providing a theoretical basis for

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19

Liu, Chi, Lei Gu, Jie Ding, et al. "Autophagy in skin barrier and immune‐related skin diseases." Journal of Dermatology 48, no. 12 (2021): 1827–37. http://dx.doi.org/10.1111/1346-8138.16185.

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20

Liu, Bingnan, Yuanyuan Shao, and Rong Fu. "Current research status of HLA in immune‐related diseases." Immunity, Inflammation and Disease 9, no. 2 (2021): 340–50. http://dx.doi.org/10.1002/iid3.416.

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21

Vingerhoets, A. J. J. M., J. Assies, K. Goodkin, G. L. Van Heck, and M. H. Bekker. "Prenatal diethylstilbestrol exposure and self-reported immune-related diseases." European Journal of Obstetrics & Gynecology and Reproductive Biology 77, no. 2 (1998): 205–9. http://dx.doi.org/10.1016/s0301-2115(97)00274-1.

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22

Homayouni Rad, Aziz, Reza Torab, Morad Ghalibaf, Sharareh Norouzi, and Elnaz Vaghef Mehrabany. "Might patients with immune-related diseases benefit from probiotics?" Nutrition 29, no. 3 (2013): 583–86. http://dx.doi.org/10.1016/j.nut.2012.10.008.

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23

Wang, Jing, Zhenyu Li, Li Gao, Yanshuang Qi, Haibo Zhu, and Xuemei Qin. "The regulation effect of AMPK in immune related diseases." Science China Life Sciences 61, no. 5 (2017): 523–33. http://dx.doi.org/10.1007/s11427-017-9169-6.

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24

Zhang, Junfeng, Yunsheng Zhang, Qingpeng Wang, et al. "Interleukin-35 in immune-related diseases: protection or destruction." Immunology 157, no. 1 (2019): 13–20. http://dx.doi.org/10.1111/imm.13044.

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25

Effendi, Raden Mohamad Rendy Ariezal, Muhammad Anshory, Handono Kalim, et al. "Akkermansia muciniphila and Faecalibacterium prausnitzii in Immune-Related Diseases." Microorganisms 10, no. 12 (2022): 2382. http://dx.doi.org/10.3390/microorganisms10122382.

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Probiotics and synbiotics are used to treat chronic illnesses due to their roles in immune system modulation and anti-inflammatory response. They have been shown to reduce inflammation in a number of immune-related disorders, including systemic lupus erythematosus (SLE), human immunodeficiency virus (HIV), and chronic inflammatory skin conditions such as psoriasis and atopic dermatitis (AD). Akkermansia muciniphila (A. muciniphila) and Faecalibacterium prausnitzii (F. prausnitzii) are two different types of bacteria that play a significant part in this function. It has been established that Ak

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26

Konno, Satoshi. "The Concept of Management of Immune-related Pulmonary Diseases." Nihon Naika Gakkai Zasshi 111, no. 10 (2022): 2075–77. http://dx.doi.org/10.2169/naika.111.2075.

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27

Funchain, Pauline. "Reincarnating autoimmunity: Immune-related adverse events as new diseases." Cleveland Clinic Journal of Medicine 90, no. 5 (2023): 318–19. http://dx.doi.org/10.3949/ccjm.90a.23013.

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28

Anshory, Muhammad, Raden Mohamad Rendy Ariezal Effendi, Handono Kalim, et al. "Butyrate Properties in Immune-Related Diseases: Friend or Foe?" Fermentation 9, no. 3 (2023): 205. http://dx.doi.org/10.3390/fermentation9030205.

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Butyrate is a short-chain fatty acid (SCFA) created within the intestinal lumen by bacterial fermentation of largely undigested dietary carbohydrates. Its beneficial effects on cellular energy metabolism and intestinal homeostasis have garnered significant attention among SCFAs. Butyrate also has systemic effects and is known to regulate the immune system. Most of the butyrate and other SCFAs are produced in the human colon, through the fermentation of dietary fiber or resistant starch. However, the modern diet often lacks sufficient intake of fermentable dietary fiber, which can lead to low b

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29

BARANZINI, Sergio E. "Symposium 2-1 The autoimmunome: Similarities and differences among genetic susceptibility to common immune-related diseases." Japanese Journal of Clinical Immunology 37, no. 4 (2014): 261. http://dx.doi.org/10.2177/jsci.37.261.

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30

African, Research Journal of Medical Sciences. "Knowledge representation of age-related diseases under innate and adaptive-immunosenescence valued contexts." African Research Journal of Medical Sciences 1, no. 1 (2024): 33–41. https://doi.org/10.62587/AFRJMS.1.1.2024.33-41.

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Aging is a major risk factor for the higher incidence and prevalence of chronic conditions, such as cancer, cardiovascular and metabolic diseases, and neurodegenerative diseases. Closely linked with the onset of all these agerelated diseases is immunosenescence, a condition defined as progressive reconfiguration of the immune functions leading to an increased  susceptibility to infectious diseases, poorer response to vaccination, increased prevalence of cancer, rheumatoid arthritis, and other chronic diseases characterized by a pro-inflammatory state named inflammaging. Given that immunos

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31

Huang, Hai, Bing Liu, Na Qu, et al. "Research progress of opioid growth factor in immune-related diseases and cancer diseases." International Immunopharmacology 99 (October 2021): 107713. http://dx.doi.org/10.1016/j.intimp.2021.107713.

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32

Shao, Zonghong, Yuanyuan Shao, and Rong Fu. "IgG Subclasses Abnormal in Immune-Related Pancytopenia." Blood 124, no. 21 (2014): 5158. http://dx.doi.org/10.1182/blood.v124.21.5158.5158.

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Abstract In recent years, we found some patients with idiopathic cytopenia of undetermined significance (ICUS)responded well to corticosteroid and/or high-dose intravenous immunoglobulin treatment. We had detected both IgG and IgM antibodies by bone marrow mononuclear-cell-Coombs test, flow cytometric and immunofluorescence analysis on the various bone marrow cell membrane. We also detected IgG in the BM supernatant of these ICUS patients, which can target several antigens on hematopoietic cells membranes by western blot. Then We termed this group of ICUS cases with autoantibodies as immuno-re

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33

Peng, Linan, Yingying Wei, Yijia Shao, Yi Li, Na Liu, and Lihua Duan. "The Emerging Roles of CCN3 Protein in Immune-Related Diseases." Mediators of Inflammation 2021 (May 18, 2021): 1–8. http://dx.doi.org/10.1155/2021/5576059.

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The CCN proteins are a family of extracellular matrix- (ECM-) associated proteins which currently consist of six secreted proteins (CCN1-6). CCN3 protein, also known as nephroblastoma overexpressed protein (NOV), is a member of the CCN family with multiple biological functions, implicated in major cellular processes such as cell growth, migration, and differentiation. Recently, CCN3 has emerged as a critical regulator in a variety of diseases, including immune-related diseases, including rheumatology arthritis, osteoarthritis, and systemic sclerosis. In this review, we will briefly introduce t

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34

Liu, Guo, and Feng Liu. "Bach2: A Key Regulator in Th2-Related Immune Cells and Th2 Immune Response." Journal of Immunology Research 2022 (March 12, 2022): 1–10. http://dx.doi.org/10.1155/2022/2814510.

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Th2 immune response is essential for providing protection against pathogens and orchestrating humoral immunity. However, excessive Th2 immune response leads to the pathogenesis of Th2 inflammation diseases, including asthma, allergic rhinitis, and atopic dermatitis. Emerging evidence suggest a critical role of the transcription factor Bach2 in regulating Th2 immune responses. Bach2 serves as a super enhancer and transcriptional repressor to control the differentiation and maturation of Th2-related immune cells such as B cell lineages and T cell lineages. In B cells, Bach2 is required for every

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35

Patil, C. S., and K. L. Kirkwood. "p38 MAPK Signaling in Oral-related Diseases." Journal of Dental Research 86, no. 9 (2007): 812–25. http://dx.doi.org/10.1177/154405910708600903.

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Multiple dental diseases are characterized by chronic inflammation, due to the production of cytokines, chemokines, and prostanoids by immune and non-immune cells. Membrane-bound receptors provide a link between the extracellular environment and the initiation of intracellular signaling events that activate common signaling components, including p38 mitogen-activated protein kinase (MAPK), extracellular signal-regulated kinase (ERK), c-Jun N-terminal kinase (JNK), and nuclear factor (NF)-κB. Although ERK pathways regulate cell survival and are responsive to extracellular mitogens, p38 MAPK, JN

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36

Mammadova-Bach, Elmina, and Attila Braun. "Zinc Homeostasis in Platelet-Related Diseases." International Journal of Molecular Sciences 20, no. 21 (2019): 5258. http://dx.doi.org/10.3390/ijms20215258.

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Zn2+ deficiency in the human population is frequent in underdeveloped countries. Worldwide, approximatively 2 billion people consume Zn2+-deficient diets, accounting for 1–4% of deaths each year, mainly in infants with a compromised immune system. Depending on the severity of Zn2+ deficiency, clinical symptoms are associated with impaired wound healing, alopecia, diarrhea, poor growth, dysfunction of the immune and nervous system with congenital abnormalities and bleeding disorders. Poor nutritional Zn2+ status in patients with metastatic squamous cell carcinoma or with advanced non-Hodgkin ly

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37

Gediz, Fusun, and Senol Kobak. "Immune Checkpoint Inhibitors-related Rheumatic Diseases: What Rheumatologist Should Know?" Current Rheumatology Reviews 15, no. 3 (2019): 201–8. http://dx.doi.org/10.2174/1573397115666190119094736.

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: Immune checkpoint inhibitors are revolutionized drugs for cancer immunotherapy in the last years. The mechanism of action of CPIs including the limitation of the activation of Tcells, and thus enhancing the self-immune response against tumour cells. Checkpointinhibitors( CPIs) may dysregulate the immune system, resulting in some toxicities. These toxicities or side effects are called Immune-related Adverse Events (IRAEs) that can potentially affect any organ and tissue. Rheumatic diseases due to checkpoint inhibitors are also reported in the literature. The spectrum of rheumatic manifestatio

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38

Hui, Lu, Xiaolin Chen, Mengke Huang, Yongmei Jiang, and Ting Liu. "TANK-Binding Kinase 1 in the Pathogenesis and Treatment of Inflammation-Related Diseases." International Journal of Molecular Sciences 26, no. 5 (2025): 1941. https://doi.org/10.3390/ijms26051941.

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TANK-binding kinase 1 (TBK1) is a key signaling kinase involved in innate immune and inflammatory responses. TBK1 drives immune cells to participate in the inflammatory response by activating the NF-κB and interferon regulatory factor signaling pathways in immune cells, promoting the expression of pro-inflammatory genes, and regulating immune cell function. Thus, it plays a crucial role in initiating a signaling cascade that establishes an inflammatory environment. In inflammation-related diseases, TBK1 acts as a bridge linking inflammation to immunity, metabolism, or tumorigenesis, playing an

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39

Marenholz, Ingo, Jorge Esparza-Gordillo, and Young-Ae Lee. "Shared genetic determinants between eczema and other immune-related diseases." Current Opinion in Allergy and Clinical Immunology 13, no. 5 (2013): 478–86. http://dx.doi.org/10.1097/aci.0b013e328364e8f7.

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40

Xi, Dan, TianTian Luo, Haowei Xiong, et al. "SAP: structure, function, and its roles in immune-related diseases." International Journal of Cardiology 187 (May 2015): 20–26. http://dx.doi.org/10.1016/j.ijcard.2015.03.179.

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41

Franceschi, Claudio, Paolo Garagnani, Paolo Parini, Cristina Giuliani, and Aurelia Santoro. "Inflammaging: a new immune–metabolic viewpoint for age-related diseases." Nature Reviews Endocrinology 14, no. 10 (2018): 576–90. http://dx.doi.org/10.1038/s41574-018-0059-4.

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42

Akbar, A., and T. Fülöp. "CIRCULATING IMMUNE CELLS AND THEIR CONTRIBUTION TO AGE-RELATED DISEASES." Innovation in Aging 1, suppl_1 (2017): 1253–54. http://dx.doi.org/10.1093/geroni/igx004.4556.

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43

Sansonno, Domenico. "Immune-Related Disorders and Extrahepatic Diseases in Chronic HCV Infection." Clinical and Developmental Immunology 2012 (2012): 1–2. http://dx.doi.org/10.1155/2012/509309.

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44

Suarez-Alvarez, Beatriz, Ramon M. Rodriguez, Mario F. Fraga, and Carlos López-Larrea. "DNA methylation: a promising landscape for immune system-related diseases." Trends in Genetics 28, no. 10 (2012): 506–14. http://dx.doi.org/10.1016/j.tig.2012.06.005.

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45

Sahu, Kamal Kant, Azra Borogovac, and Jan Cerny. "COVID‐19 related immune hemolysis and thrombocytopenia." Journal of Medical Virology 93, no. 2 (2020): 1164–70. http://dx.doi.org/10.1002/jmv.26402.

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46

Liu, Xianqiang, Dingchang Li, Yue Zhang, et al. "Identifying Common Genetic Etiologies Between Inflammatory Bowel Disease and Related Immune-Mediated Diseases." Biomedicines 12, no. 11 (2024): 2562. http://dx.doi.org/10.3390/biomedicines12112562.

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Background: Patients with inflammatory bowel disease (IBD) have an increased risk of developing immune-mediated diseases. However, the genetic basis of IBD is complex, and an integrated approach should be used to elucidate the complex genetic relationship between IBD and immune-mediated diseases. Methods: The genetic relationship between IBD and 16 immune-mediated diseases was examined using linkage disequilibrium score regression. GWAS data were synthesized from two IBD databases using the METAL, and multi-trait analysis of genome-wide association studies was performed to enhance statistical

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47

Ghanemi, Abdelaziz, and Besma Boubertakh. "Spotlight on Neuroimmunology: Illustrations from Neurodegenerative Diseases." International Journal of Public Health Science (IJPHS) 6, no. 2 (2017): 146. http://dx.doi.org/10.11591/ijphs.v6i2.6645.

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The immune system plays key roles in the defense of the organism. However, the effects of the immune system are not limited to the immune functions and have impacts beyond the anti-pathogenes role. Indeed, neuroimmunology is a representative field of how the immune system affects non-immune biological and physio-pathological functions. Herein, we have selected a number of neurodegenerative diseases as illustrative examples to put a spotlight on this important field. Importantly, clarifying the links and interactions between the immune system and the nervous system represents key elements for t

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48

"Immune checkpoint inhibitor-related autoimmune diseases." Reactions Weekly 1861, no. 1 (2021): 10. http://dx.doi.org/10.1007/s40278-021-97810-0.

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49

"Virus Infection and Immune Related Diseases." Clinical and Experimental Pharmacology and Physiology 40 (October 2013): 31–36. http://dx.doi.org/10.1111/1440-1681.12170_7.

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50

Huang, Ning, Hao Dong, and Bin Shao. "Phase separation in immune regulation and immune-related diseases." Journal of Molecular Medicine, September 9, 2022. http://dx.doi.org/10.1007/s00109-022-02253-9.

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