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Relevant bibliographies by topics / Osteogenesis bone

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Academic literature on the topic 'Osteogenesis bone'

Author: Grafiati

Published: 28 May 2022

Last updated: 30 July 2025

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Journal articles on the topic "Osteogenesis bone"

1

Zhang, Bo, Yali Li, Yang Yu, et al. "MicroRNA-378 Promotes Osteogenesis-Angiogenesis Coupling in BMMSCs for Potential Bone Regeneration." Analytical Cellular Pathology 2018 (2018): 1–9. http://dx.doi.org/10.1155/2018/8402390.

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Bone tissue regeneration was closely associated with osteogenesis and angiogenesis. The harmonious regulation of osteogenetic and angiogenic growth factors would enhance bone regeneration, while the imbalance of that would lead to local excessive bone formation or vascular mass due to exogenous delivery. Therefore, microRNA is believed to regulate multiple metabolism progress through endogenous signaling pathways on the gene level. In this work, we identified microRNA 378 as a positive regulator of osteogenesis and angiogenesis simultaneously and also observed an increase of microRNA 378 than

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Mantsou, Aglaia, Eleni Papachristou, Panagiotis Keramidas, et al. "A Novel Drastic Peptide Genetically Adapted to Biomimetic Scaffolds “Delivers” Osteogenic Signals to Human Mesenchymal Stem Cells." Nanomaterials 13, no. 7 (2023): 1236. http://dx.doi.org/10.3390/nano13071236.

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This work describes the design, preparation, and deep investigation of “intelligent nanobiomaterials” that fulfill the safety rules and aim to serve as “signal deliverers” for osteogenesis, harboring a specific peptide that promotes and enhances osteogenesis at the end of their hydrogel fibers. The de novo synthesized protein fibers, besides their mechanical properties owed to their protein constituents from elastin, silk fibroin and mussel-foot adhesive protein-1 as well as to cell-attachment peptides from extracellular matrix glycoproteins, incorporate the Bone Morphogenetic Protein-2 (BMP2)

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Feng, Guiyu, Wei Liu, Yao Yu, et al. "Angiogenesis coupled with osteogenesis in a bone tissue engineering scaffold enhances bone repair in osteoporotic bone defects." Biomedical Materials 18, no. 4 (2023): 045002. http://dx.doi.org/10.1088/1748-605x/accf55.

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Abstract Increased life expectancy has resulted in an increase in osteoporosis incidence worldwide. The coupling of angiogenesis and osteogenesis is indispensable for bone repair. Although traditional Chinese medicine (TCM) exerts therapeutic effects on osteoporosis, TCM-related scaffolds, which focus on the coupling of angiogenesis and osteogenesis, have not yet been used for the treatment of osteoporotic bone defects. Panax notoginseng saponin (PNS), the active ingredient of Panax notoginseng, was added to a poly (L-lactic acid) (PLLA) matrix. Osteopractic total flavone (OTF), the active ing

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Masurkar, Deepika, Priyanka Jaiswal, and Diksha Agrawal. "Alveolar Distraction Osteogenesis." Journal of Evolution of Medical and Dental Sciences 10, no. 38 (2021): 3462–65. http://dx.doi.org/10.14260/jemds/2021/701.

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Resorbed alveolar ridges, which can be horizontal or vertical in size, are one of the most prevalent issues addressed by dental experts. One of the therapy options for addressing the weak sections of the alveolar ridges is alveolar distraction osteogenesis. Alveolar distraction osteogenesis is a biological process that includes gradual traction between vascularized bone segments in order to generate new bone. Typically, an osteotomy is done, followed by the placement of an osteodistraction device to manage the separating process. This approach eliminates the need for bone grafts by producing t

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Łukowicz, Krzysztof, Karolina Fijał, Aleksandra Nowak, and Anna M. Osyczka. "Connexin 43 in osteogenesis." Postępy Higieny i Medycyny Doświadczalnej 74 (September 25, 2020): 406–15. http://dx.doi.org/10.5604/01.3001.0014.4153.

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Skeleton formation and its proper functioning is possible thanks to specialized bone tissue cells: bone forming osteoblasts, bone resorbing osteoclasts and osteocytes located in bone cavities. Gap junctions are transmembrane channels connecting neighboring cell. Thanks to gap junctions it is possible for signals to be directly transmitted by cells. Gap junction type channels, and more specifically the connexin proteins that build them, have a key impacton the bone turnover process, and thus on both bone building and remodeling. A particularly important connexin in bone tissue is connexin43 (Cx

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Kim, Jung-Eun. "Osteoclastogenesis and Osteogenesis." International Journal of Molecular Sciences 23, no. 12 (2022): 6659. http://dx.doi.org/10.3390/ijms23126659.

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Stewart, Sarah, Alastair Darwood, Spyros Masouros, Claire Higgins, and Arul Ramasamy. "Mechanotransduction in osteogenesis." Bone & Joint Research 9, no. 1 (2020): 1–14. http://dx.doi.org/10.1302/2046-3758.91.bjr-2019-0043.r2.

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Bone is one of the most highly adaptive tissues in the body, possessing the capability to alter its morphology and function in response to stimuli in its surrounding environment. The ability of bone to sense and convert external mechanical stimuli into a biochemical response, which ultimately alters the phenotype and function of the cell, is described as mechanotransduction. This review aims to describe the fundamental physiology and biomechanisms that occur to induce osteogenic adaptation of a cell following application of a physical stimulus. Considerable developments have been made in recen

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Wang, Junjie, Bo Yuan, Ruixue Yin, and Hongbo Zhang. "Inflammation Responses to Bone Scaffolds under Mechanical Stimuli in Bone Regeneration." Journal of Functional Biomaterials 14, no. 3 (2023): 169. http://dx.doi.org/10.3390/jfb14030169.

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Physical stimuli play an important role in one tissue engineering. Mechanical stimuli, such as ultrasound with cyclic loading, are widely used to promote bone osteogenesis; however, the inflammatory response under physical stimuli has not been well studied. In this paper, the signaling pathways related to inflammatory responses in bone tissue engineering are evaluated, and the application of physical stimulation to promote osteogenesis and its related mechanisms are reviewed in detail; in particular, how physical stimulation alleviates inflammatory responses during transplantation when employi

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Palumbo, Carla, Francesca Paganelli, and Marzia Ferretti. "Ossification in Normal and Pathological Contexts: The Key Role of Static Osteogenesis vs. Dynamic Osteogenesis in the Etiopathology of Some Skeletal Alterations." Biomolecules 15, no. 5 (2025): 733. https://doi.org/10.3390/biom15050733.

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This Commentary is intended to start a discussion in the field of calcification/ossification related to osteogenesis. It highlights that two types of bone formation, static osteogenesis (SO) and dynamic osteogenesis (DO), are temporally followed by each other in bone histogenesis and bone lesion repair. Moreover, they also represent the common denominator in the pathological processes of both calcification and peculiar ossifications, such as heterotopic ossification and the formation of supernumerary skeletal segments. The final objective is to propose a different interpretation of certain bon

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Yan, Wei Qi, Jie Feng, Q. Chen, Di Sheng Yang, Ying Zhao, and Shi Gui Yan. "The Osteogenic Potential of Stem Cell- and BMP2 Gene-Activating Bioceramic In Vivo." Key Engineering Materials 284-286 (April 2005): 941–44. http://dx.doi.org/10.4028/www.scientific.net/kem.284-286.941.

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Stimulation of bone healing through local application of growth factors from implants may improve the clinical outcome in fracture treatments. However, the growth factors in reconstructive application require supraphysiologic dosing and considerable expense while hampering their clinical application. Genetic modification of mesenchymal stem cells (MSCs) to both produce and respond to osteogenic factors may have potential for use in enhancing bone healing. In this study, MSCs were genetically modified by a recombinant adenoviral containing the gene for human bone morphogenetic protein 2 (hBMP-2

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Dissertations / Theses on the topic "Osteogenesis bone"

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Midha, Swati. "Osteogenesis in porous biomaterials for bone regeneration." Thesis, Ulster University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.674920.

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Baigrie, Carolyn Frances. "Collagen genes in osteogenesis imperfecta." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.291073.

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Ma, Li. "The influence of nicotine on angiogenesis and osteogenesis in bone regeneration." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41508440.

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Ma, Li, and 马丽. "The influence of nicotine on angiogenesis and osteogenesis in bone regeneration." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41508440.

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Hrit, Manuela. "Acceleration of bone formation in distraction osteogenesis by bone morphogenetic protein-7." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=101142.

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The manipulation of the molecular mechanisms that govern distraction osteogenesis (DO) in order to increase the biomechanical strength of new bone and to accelerate its synthesis has been the topic of intense research during the past decades.<br>Bone morphogenetic proteins (BMPs) play an important role in bone formation. In this study, using a rabbit model of DO, the expressions of BMP's major intracellular signalling molecules, Smad proteins, was analyzed and correlated with the expression of BMP ligands and receptors. Based on these results, which confirmed post-receptor activity for the BMP

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Barragan-Adjemian, Maria del Cielo Bonewald Lynda F. "Mechanisms of mineralization in bone." Diss., UMK access, 2006.

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Thesis (Ph. D.)--School of Dentistry. University of Missouri--Kansas City, 2006.<br>"A dissertation in oral biology and cell biology and biophysics." Advisor: Lynda F. Bonewald. Typescript. Vita. Title from "catalog record" of the print edition Description based on contents viewed Nov. 12, 2007. Includes bibliographical references (leaves 121-139). Online version of the print edition.

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Chay, Siew Han. "Vascular endothelial growth pattern during demineralized bone matrix (intramembranous bone origin) induced osteogenesis." Click to view the E-thesis via HKUTO, 1999. http://sunzi.lib.hku.hk/HKUTO/record/B38628417.

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謝秀嫻 and Siew Han Chay. "Vascular endothelial growth pattern during demineralized bone matrix (intramembranous bone origin) induced osteogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B38628417.

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Alfayez, Eman Saud. "Synergizing angiogenesis and osteogenesis in a smart bone substitute." Thesis, King's College London (University of London), 2016. https://kclpure.kcl.ac.uk/portal/en/theses/synergizing-angiogenesis-and-osteogenesis-in-a-smart-bone-substitute(ad38b2b9-e1e3-42ce-88a0-91a764bc14e6).html.

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The major aim of this project was to develop a biologically active bone scaffold that could induce vascularization in critical-size defects (CSD) and hence bone formation. In this study, functionalization of three-dimensional (3D) printed biphasic calcium phosphate (BCP) scaffolds was investigated. The first functionalization approach involved printing scaffolds with two different pore geometries and sizes; square (400μ) and round (800μ). The second was by coating scaffolds with DAR16-II; a self-assembly peptide that forms a hydrogel nanostructure mimicking extracellular matrix (ECM). A rabbit

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Hamade, Fares. "Enhanced bone formation during distraction osteogenesis in FGFR3 deficient mice." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112630.

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Distraction Osteogenesis (DO) is a technique for bone lengthening and filling of bone defects following trauma, infection or resection of tumors. DO consists of an osteotomy of the bone to be lengthened, followed by controlled distraction of the bone segments with an external fixator until the desired lengthening is obtained (distraction phase). This is followed by the consolidation phase, during which the external fixator is kept in place until the newly formed bone in the distracted zone consolidates. This phase is long and may cause numerous problems. Ongoing research aims at finding a meth

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Books on the topic "Osteogenesis bone"

1

Lim, Jeremy Hon Lee. Beyond bone breaking. MindChamps Publishing, 2011.

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2

International, Workshop on Calcified Tissues (6th 1984 Kiryat ʻAnavim Israel). Current advances in skeletogenesis: Induction, biomineralization, bone seeking hormones, congenital and metabolic bone diseases : proceedings of the Sixth International Workshop on Calcified Tissues, Kiryat-Anavim, Israel, 18-23 March 1984. Excerpta Medica, 1985.

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3

Wiseman, Lorne. The effect of lyophilized demineralized bone on osteogenesis in vitro: A preliminary study. Faculty of Dentistry, University of Toronto], 1988.

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4

Culbert, Ainsley Amanda. Studies of the molecular basis of bone fragility in individuals with osteogenesis imperfecta. University of Manchester, 1996.

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Dietz, Georg. Calcium hydroxide and bone regeneration: Odontological aspects of induced osteogenesis in experiment and clinical practice. G. Dietz, 1998.

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6

Whitfield, James F. The parathyroid hormone: An unexpected bone builder for treating osteoporosis. R.G. Landes, 1998.

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Lin, Yunfeng. Osteogenesis. InTech, 2012.

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8

Maniatopoulos, Constantine. Development and characterization of an in vitro system permitting osteogenesis by stromal cells isolated from bone marrow of young adult rat(sic). Faculty of Dentistry, University of Toronto, 1988.

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9

Todescan, Reynaldo. The influence of a collagen matrix on osteogenesis by bone cells in vitro and in vivo and its effects on implant osseointegration. University of Toronto, Graduate Dept. of Dentistry], 1993.

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10

Davidson, Audrey Ekdahl. Osteogenesis imperfecta: Living with brittle bones. Logres, 2004.

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Book chapters on the topic "Osteogenesis bone"

1

Patel, Ruchita, and Pauline M. Camacho. "Osteogenesis Imperfecta." In Metabolic Bone Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-03694-2_10.

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Arundel, Paul, and Nicolas J. Bishop. "Osteogenesis Imperfecta." In Bone and Development. Springer London, 2010. http://dx.doi.org/10.1007/978-1-84882-822-3_13.

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Dawson, Paul A., and Joan C. Marini. "Osteogenesis Imperfecta." In The Genetics of Osteoporosis and Metabolic Bone Disease. Humana Press, 2000. http://dx.doi.org/10.1007/978-1-59259-033-9_5.

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Rosenberg, Nahum. "Distraction Osteogenesis." In Biophysical Osteoblast Stimulation for Bone Grafting and Regeneration. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-06920-8_8.

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Biggin, Andrew, and Craig F. Munns. "Bisphosphonates in Osteogenesis Imperfecta." In Bone Drugs in Pediatrics. Springer US, 2014. http://dx.doi.org/10.1007/978-1-4899-7436-5_5.

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Brookes, Murray, and William J. Revell. "Vascular control of osteogenesis." In Blood Supply of Bone. Springer London, 1998. http://dx.doi.org/10.1007/978-1-4471-1543-4_23.

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Hemprich, Alexander, and Thomas Hierl. "Craniomaxillofacial Distraction Osteogenesis." In Craniomaxillofacial Reconstructive and Corrective Bone Surgery. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-1529-3_51.

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Bartl, R., and B. Frisch. "Osteogenesis imperfecta." In Biopsy of Bone in Internal Medicine: An Atlas and Sourcebook. Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-2222-1_10.

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Shapiro, J. R., and D. W. Rowe. "Osteogenesis Imperfecta: Current Concepts." In Current Concepts of Bone Fragility. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70709-4_18.

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Birch, J. G. "Osteogenesis Imperfecta: Orthopedic Aspects." In Current Concepts of Bone Fragility. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70709-4_20.

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Conference papers on the topic "Osteogenesis bone"

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Shrivas, Nikhil V., Abhishek Kumar Tiwari, Rakesh Kumar, Dharmendra Tripathi, and Vasu Raman Sharma. "Investigation on Loading-Induced Fluid Flow in Osteogenesis Imperfecta Bone." In ASME 2018 5th Joint US-European Fluids Engineering Division Summer Meeting. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/fedsm2018-83496.

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Osteogenesis Imperfecta (OI) is a genetic bone disorder which is typically characterized by brittle bones with frequent fractures. It is also known as brittle bone disease. Surgical procedure is one of the ways adopted by clinicians for the management of OI. In recent years, it has however become clear that physical activity is equally important for managing OI in both children and adults. Exogenous mechanical stimulation e.g. prophylactic exercises may be useful in improving the bone mass and strength of OI bones as loading-induced mechanical components e.g. normal strain and canalicular flui

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Morgan, Elise F., Billy Andre, Daniel E. Hogan, Bader A. Al-Awadhi, and Louis C. Gerstenfeld. "Quantitative, 3-D Imaging to Co-Localize Bone and Vasculature Tissues During Bone Healing." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19100.

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Successful bone repair requires reestablishment of a blood supply at the injury site [1]. However, despite convincing evidence of the close relationship between angiogenesis and osteogenesis during bone healing, little is known about the spatial correspondence between these two processes. This paucity of information hampers understanding of the mechanisms regulating osteogenesis during bone healing and of how molecular communication between endothelial cells and osteoblasts mediates the restoration of mechanical function.

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Gurkan, Umut A., Adam Krueger, and Ozan Akkus. "Mechanical Stimulation Enhances the Production of BMP-2 in Ossifying Rat Bone Marrow Organ Cultures." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206745.

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Bone marrow is a reservoir of mesenchymal stem cells and osteoprogenitors. It was previously shown [1, 2] and we have recently verified that in vitro cultures of bone marrow undergo osteogenesis without addition of osteoinductive stimulants. We hypothesized that the in vitro ossifying bone marrow organ culture system can be used as a model to investigate the dynamics of the osteogenesis process and its mechanoresponsiveness in terms of expression of key osteoinductive factors. The outcomes of these studies can be used to develop more effective tissue engineered substitutes for bone regeneratio

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Dong, X. Neil, Mahyar Zoghi, Qitao Ran, and Xiaodu Wang. "Less Diffuse Damage was Observed in Osteogenesis Imperfecta Mice Femurs Than Wild-Type Controls." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19401.

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Accumulation of microdamage contributes to the deterioration of bone quality with aging among the elderly [1]. Microdamage formation in bone is dependent on its ultrastructural constituents. At the ultrastructural level, bone is a fiber-reinforced composite material in which mineral crystals are responsible for its stiffness and collagen fibrils offer its toughness [2]. Using animal models with distinct mineralization levels (BL6 and C3H mice), a previous study has demonstrated that highly mineralized bone tend to form linear microcracks whereas less mineralized bone is more likely to have dif

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Jameson, John, Carolyne Albert, Peter Smith, Robert Molthen, and Gerald Harris. "Micro-CT characterization of human trabecular bone in osteogenesis imperfecta." In SPIE Medical Imaging, edited by John B. Weaver and Robert C. Molthen. SPIE, 2011. http://dx.doi.org/10.1117/12.877586.

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Florez, H., A. Muxi, E. Gonzalez, A. Monegal, N. Guañabens, and P. Peris. "SAT0361 Trabecular bone score in osteogenesis imperfecta. is it useful?" In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.4883.

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Richards, Mark, Jeffrey A. Weiss, Eben Alsberg, et al. "Mechanical Strain Environment Controls New Bone Formation During Distraction Osteogenesis." In ASME 1996 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1996. http://dx.doi.org/10.1115/imece1996-1272.

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Abstract An animal model of distraction osteogenesis was used to investigate the effects of altered stresses and strains on tissue development, differentiation, and repair. Osteotomies were performed at 0 and 45 degrees to create different mechanical environments within the distraction gap. Finite element simulations of functional loading and actual distractions indicated significant changes in the gap deformation field. These differences correlated with significant decreases in new bone volume and changes in trabecular architecture for the 45-degree case.

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Chen, Jing, and Sihong Wang. "Thermal Effects on Osteogenesis of Human Mesenchymal Stem Cells." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80885.

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Intensive studies were reported on the osteogenesis of mesenchymal stem cells (MSC) using chemicals and mechanical loading. However, the maturity of differentiated osteoblasts is not same as that of isolated adult osteoblasts. Thermal treatment could be a missing factor in stem cell differentiation. It was reported that mild heat stimulated bone growth in animal experiments [1–2]. Thermal treatment is also used as a therapy to promote bone repair after injury [3]. In addition, hot shower daily is recommended to osteoarthritis patients. However, the mechanisms for the heat-induced osteogenesis

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Askew, Michael J., Gary B. Schneider, Kristina J. Grecco, Jason Hsu, Emily Mugler, and Donald A. Noe. "Effect of Pharmaceutical Bone Growth Stimulation With Novel Anabolic Peptides: Biomechanical and Bone Density Measurements in a Rat Model." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43044.

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Pharmaceutical bone growth stimulation holds promise for prevention and treatment bone disorders, and the enhancement of fracture healing. Bone growth hormones have begun to have limited clinical use, but can illicit adverse side effects. Recent studies have shown that short peptides (less than 15 amino acids) derived from the protein sequence of Vitamin D Binding Protein (DBP), can enhance bone formation (osteogenesis). These peptides may have potential as controllable bone growth stimulators without the adverse side effects and cost of bone growth hormones. Rats, injected every other day for

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Ramos Castro, D., S. Leal Rodriguez, E. Grau García, et al. "POS0578 STUDY OF BONE METABOLISM IN PATIENTS DIAGNOSED WITH OSTEOGENESIS IMPERFECTA." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.4410.

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Reports on the topic "Osteogenesis bone"

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Villegas Aguilar, Julio Cesar, Marco Felipe Salas Orozco, Maria de los Angeles Moyaho Bernal, et al. Mechanical vibrations and increased alveolar bone density in animal models as an alternative to improve bone quality during orthodontic treatment: A systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0103.

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Review question / Objective: The aim of this systematic review is to determine whether mechanical vibration increases alveolar bone density in animals models and their possible application during orthodontic treatment. In this sense, the focused question is: Is the increase in alveolar bone density by mechanical vibrations in animal models an alternative to improve bone quality during orthodontic treatment? Eligibility criteria: All published animal studies will be included. Animal studies where high or low frequency vibrations were be applied, Articles where density or osteogenesis were be me

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