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Books on the topic 'Oxytocin and Vasopressin'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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1

Zingg, Hans H., Charles W. Bourque, and Daniel G. Bichet, eds. Vasopressin and Oxytocin. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4871-3.

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2

Neumann, Inga D. Advances in vasopressin and oxytocin: From genes to behaviour to disease. Elsevier, 2008.

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3

Advances in vasopressin and oxytocin: From genes to behaviour to disease. Elsevier, 2008.

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4

Edinburgh), World Congress on Neurohypophysial Hormones (1999. Oxytocin and vasopressin: From molecules to function. Published for the Physiological Society by Cambridge University Press, 2000.

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5

Dominique, Poulain, Oliet, Stéphane H. R., 1965-, and Theodosis Dionysia, eds. Vasopressin and oxytocin: From genes to clinical applications. Elsevier, 2002.

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6

Choleris, Elena, Donald W. Pfaff, and Martin Kavaliers, eds. Oxytocin, Vasopressin and Related Peptides in the Regulation of Behavior. Cambridge University Press, 2013. http://dx.doi.org/10.1017/cbo9781139017855.

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7

1936-, Saitō Toshikazu, Kurokawa Kiyoshi, and Yoshida Shō, eds. Neurohypophysis: Recent progress of vasopressin and oxytocin research : proceedings of the 1st Joint World Congress of Neurohypophysis and Vasopressin, Nasu, Tochigi, Japan, 16-21 July 1995. Elsevier, 1995.

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8

Hu, Shuang-Bao. Corticothropin-releasing factor-41 (CRF-41), arginne vasopressin (AVP), and oxytocin regulation in hypothalamic cellsin culture. Brunel University, 1992.

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9

Shō, Yoshida, and Share Leonard, eds. Recent progress in posterior pituitary hormones 1988: Proceedings of the Satellite Symposium on Posterior Pituitary Hormones, Hakone, Japan, 14-16 July 1988. Excerpta Medica, 1988.

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10

Zingg, Hans H., Charles W. Bourque, and Daniel G. Bichet. Vasopressin and Oxytocin. Springer, 2012.

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11

Vasopressin and oxytocin: Molecular, cellular, and clinical advances. Plenum, 1998.

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12

(Editor), Hans H. Zingg, Charles W. Bourque (Editor), and Daniel G. Bichet (Editor), eds. Vasopressin and Oxytocin: Molecular, Cellular, and Clinical Advances (Advances in Experimental Medicine and Biology). Springer, 1998.

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13

Vasopressin and Oxytocin: From Genes to Clinical Applications. Elsevier, 2002. http://dx.doi.org/10.1016/s0079-6123(00)x0011-8.

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14

Vasopressin and Oxytocin: Molecular, Cellular, and Clinical Advances. Springer, 2011.

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15

McEwen, Barbara. Roles of Vasopressin and Oxytocin in Memory Processing, Volume 50 (Advances in Pharmacology). Academic Press, 2004.

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16

McEwen, Barbara. Roles of Vasopressin and Oxytocin in Memory Processing, Volume 50 (Advances in Pharmacology). Academic Press, 2004.

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17

The Roles of Vasopressin and Oxytocin in Memory Processing. Elsevier, 2004. http://dx.doi.org/10.1016/s1054-3589(00)x0003-8.

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18

Kavaliers, Martin, Donald W. Pfaff, and Elena Choleris. Oxytocin, Vasopressin and Related Peptides in the Regulation of Behavior. Cambridge University Press, 2013.

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19

Oxytocin, Vasopressin and Related Peptides in the Regulation of Behavior. Cambridge University Press, 2018.

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20

(Editor), D. Poulain, S. Oliet (Editor), and D. Theodosis (Editor), eds. Vasopressin and Oxytocin: From Genes to Clinical Applications (Progress in Brain Research). Elsevier Science, 2002.

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21

Westberg, Lars, and Hasse Walum. Oxytocin and Vasopressin Gene Variation and the Neural Basis of Social Behaviors. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.011.

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Experimental studies in rodents and humans show that the neuropeptides oxytocin and vasopressin are important regulators of behaviors related to social interactions. Evidence for positive effects of oxytocin treatment on symptoms of psychiatric disorders characterized by impaired social functioning has emerged. Numerous studies report associations between various social behaviors, the risk of autism, and polymorphisms inOXTRandAVPR1A. This chapter provides an overview of these genetic association studies. Although many of the published findings are inconclusive and need replication in independ

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22

Carter, C. Sue, Inbal Ben-Ami Bartal, and Eric C. Porges. The Roots of Compassion. Edited by Emma M. Seppälä, Emiliana Simon-Thomas, Stephanie L. Brown, Monica C. Worline, C. Daryl Cameron, and James R. Doty. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190464684.013.14.

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Compassion for others and social support have survival value and health benefits. Although compassion is sometimes considered uniquely human, critical components of compassion have been described in nonhuman mammals. Studies originally conducted in social mammals and now in humans have implicated neuropeptide hormones, especially oxytocin, in social cognition, a sense of safety, and the capacity of sociality to permit compassionate responses. In contrast, the related peptide vasopressin and its receptor may be necessary for forming selective relationships and for the apparently paradoxical eff

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23

Carter, C. Sue, and Suma Jacob. Oxytocin and Vasopressin: Mechanisms for Potential Sex Differences Observed in Autism Spectrum Disorders. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199744312.003.0018.

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The effects of oxytocin and vasopressin on the brain and behavior can be sexually dimorphic, especially during the course of development (Bales, Kim, et al., 2004; Bales, Pfeifer, et al., 2004; Bales, Plotsky, et al., 2007; Bielsky et al., 2005a; Carter, 2003; Thompson et al., 2006; Yamamoto et al., 2005; Yamamoto et al., 2004). Given the sexual discrepancy observed in autism spectrum disorders (ASDs), these two neuropeptides, oxytocin (OT) and arginine vasopressin (AVP), have received attention for their potential role in ASDs (Green and Hollander, 2010; Insel et al., 1999; Leckman & Herm

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24

1946-, Baylis Peter H., and Padfield Paul L, eds. The Posterior pituitary: Hormone secretion in health and disease. M. Dekker, 1985.

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25

Gorman, Jack M. Love, Reward, and Social Connections. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850128.003.0007.

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Just as there are pathways for negative emotions and behaviors like fear and despair, the brain has networks that accompany positive ones, such as parental behavior, love, and social connectedness. One such system involves the brain hormones oxytocin and vasopressin, which are known to play a critical role in monogamous pair bonding and in both maternal and paternal nurturing behavior. Another is the so-called reward pathway that originates in the brain stem and terminates in the nucleus acumbens. This pathway allows us to anticipate reward and identify risk, and it reinforces pleasurable expe

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26

North, William G., and Arnold M. Moses. The Neurohypophysis: A Window on Brain Function (Annals of the New York Academy of Sciences). New York Academy of Sciences, 1993.

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27

1942-, North William G., Moses A. M, and Share Leonard, eds. The Neurohypophysis: A window on brain function. New York Academy of Sciences, 1993.

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28

Fanning, Jennifer R., and Emil F. Coccaro. Neurobiology of Impulsive Aggression. Edited by Phillip M. Kleespies. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199352722.013.24.

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Aggression is a behavior with evolutionary origins, but in today’s society it’s often both destructive and maladaptive. The fact that aggression has a strong basis in biological factors has long been apparent from case histories of traumatic brain damage. Research over the past several decades has confirmed the involvement of neurotransmitter function and abnormalities in brain structure and function in aggressive behavior. This research has centered around the “serotonin hypothesis” and on dysfunction in prefrontal brain regions. As this literature continues to grow, guided by preclinical res

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29

Barrett, Catherine E., and Larry J. Young. Molecular Neurobiology of Social Bonding. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.001.

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Many psychiatric illnesses, including autism spectrum disorders (ASD), schizophrenia, and depression, are characterized by impaired social cognition and a compromised ability to form social relationships. Although drugs are currently available to treat other symptoms of these disorders, none specifically target the social deficits. In order to develop pharmacotherapies to enhance social functioning, particularly for ASD where social impairment is a core symptom, we must first understand the basic neurobiology underlying complex social behaviors. The socially monogamous prairie vole (Microtus o

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30

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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31

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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