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Journal articles on the topic 'Paracetamol'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 May 2025

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1

Hinz, Burkhard, and Kay Brune. "Paracetamol and cyclooxygenase inhibition: is there a cause for concern?" Annals of the Rheumatic Diseases 71, no. 1 (October 28, 2011): 20–25. http://dx.doi.org/10.1136/ard.2011.200087.

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Paracetamol is recommended as first-line therapy for pain associated with osteoarthrosis and is one of the most widely used over-the-counter analgesic drugs worldwide. Despite its extensive use, its mode of action is still unclear. Although it is commonly stated that paracetamol acts centrally, recent data imply an inhibitory effect on the activity of peripheral prostaglandin-synthesising cyclooxygenase enzymes. In this context paracetamol has been suggested to inhibit both isoforms in tissues with low levels of peroxide by reducing the higher oxidation state of cyclooxygenase enzymes. Two recent studies have also demonstrated a preferential cyclooxygenase 2 (COX-2) inhibition by paracetamol under different clinically relevant conditions. This review attempts to relate data on paracetamol's inhibitory action on peripheral cyclooxygenase enzymes to the published literature on its anti-inflammatory action and its hitherto underestimated side-effects elicited by cyclooxygenase inhibition. As a result, a pronounced COX-2 inhibition by paracetamol is expected to occur in the endothelium, possibly explaining its cardiovascular risk in epidemiological studies. A careful analysis of paracetamol's cardiovascular side-effects in randomised studies is therefore strongly advised. On the basis of epidemiological data showing an increased gastrointestinal risk of paracetamol at high doses or when co-administered with classic cyclooxygenase inhibitors, paracetamol's long-term gastrointestinal impact should be investigated in randomised trials. Finally, paracetamol's fast elimination and consequently short-lived COX-2 inhibition, which requires repetitive dosing, should be definitely considered to avoid overdosage leading to hepatotoxicity.

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2

Begum, Shaheen, Poojitha Harisree G, and Rashida Anjum M S. "A Short Review on Biological Activities of Paracetamol Derivatives." International Journal of Pharmaceutical Sciences and Nanotechnology(IJPSN) 16, no. 1 (February 13, 2023): 6309–25. http://dx.doi.org/10.37285/ijpsn.2023.16.1.5.

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Paracetamol reduces body temperature with multiple mechanisms. Paracetamol is chemically 4-hydroxy acetanilide and has a good safety profile. Following its successful use as an over-the-counter antipyretic and analgesic medication, several attempts were made to increase the potency, mask the bitter taste, and decrease the toxicity of this drug by modifications at the phenyl ring, acetamido group, and hydroxyl group. The free hydroxyl group of paracetamols was masked to obtain prodrugs (carbonate prodrugs, ester prodrugs like alanine-prodrug, proline-prodrug, galactosylated prodrug, and mutual prodrugs with other drugs and NSAIDs). Propacetamol is a commercially available prodrug derived from paracetamol that is effective in parenteral form. Paracetamol ester prodrugs with sulfur-containing amino acids such as N-acetyl cysteine, cysteine, and methionine showed low hepatotoxicity compared to the parent drug. In addition, paracetamol derivatives including metal complexes, chalcones, Mannich bases, nucleoside analogs, hybrids with the aryl-imidazolidinyl ring, thymol, and triazole ring displayed diverse activities like antioxidant, anticancer, and antimicrobial activities.

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3

Coman, Laurențiu, Horia Păunescu, Cristina Isabel Viorica Ghiță, Radu Ciprian Țincu, Sorina Vasile, Delia Cinteza, Ion Fulga, and Oana Andreia Coman. "Paracetamol-Induced Hypothermia in Rodents: A Review on Pharmacodynamics." Processes 10, no. 4 (March 31, 2022): 687. http://dx.doi.org/10.3390/pr10040687.

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Paracetamol can induce hypothermia in humans and rodents. The study’s aim is to review the mechanisms of paracetamol-induced hypothermia in rodents or the results issued from in vitro studies on the same species’ tissues (in doses that do not produce hepatic impairment) using the latest developments published in scientific journals over the last 15 years. Available human studies are also analysed. An extensive search in PubMed databases exploring the hypothermic response to paracetamol was conducted. 4669 articles about paracetamol’s effects on body temperature in mice or rats were found. After applying additional filters, 20 articles were selected for review, with 9 of them presented in tabular forms. The analysis of these articles found that the hypothermic effect of paracetamol is due to the inhibition of a cyclooxygenase-1 variant, is potentiated by endothelin receptor antagonists, and can be mediated through GABAA receptors and possibly through transient receptor potential cation channel subfamily A member 1 via N-acetyl-p-benzoquinone imine in the central nervous system. Human studies confirm the in vivo and in vitro experiments in rodents regarding the presence of a hypothermic effect after high, non-toxic doses of paracetamol. Further research is required to understand the mechanisms behind paracetamol’s hypothermic effect in humans.

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Hamed Almurisi, Samah, Khater AL-Japairai, Farhan Alshammari, Fawaz Alheibshy, Rana M. F. Sammour, and Abd Almonem Doolaanea. "Stability of Paracetamol Instant Jelly for Reconstitution: Impact of Packaging, Temperature and Humidity." Gels 8, no. 3 (February 25, 2022): 144. http://dx.doi.org/10.3390/gels8030144.

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The stability of the medicinal product is a major concern in the pharmaceutical industry and health authorities, whose goal is to guarantee that drugs are delivered to patients without loss of therapeutic properties. This study aims to evaluate the effect of environmental conditions and packaging on the stability of paracetamol instant jelly sachets based on both chemical and physical stability. The paracetamol instant jelly was packaged in plastic sachets (packaging 1) and sealed aluminium bags in screw-capped amber glass bottles (packaging 2), which were stored in real-time and accelerated stability chambers for 3 months. Samples were taken out from the chambers and were characterised for appearance, moisture content, texture, viscosity, in vitro drug release, paracetamol content, and 4-aminophenol level at different time points. The real-time storage condition at a lower temperature maintained the stability of the paracetamol instant jelly, while the accelerated condition led to a significant change in the formulation properties. In addition, the proper packaging of paracetamol instant jelly maintained the paracetamol’s stability, regardless of environmental conditions, for three months. The results show that the environmental conditions and packaging play a significant role in maintaining paracetamol instant jelly stability.

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5

Mohamed, Ashma, Alexa Wacker, and Martin Schmidt. "Chronic Misuse of Paracetamol in OCD Without Hepatic Injury: A Case Report and Literature Review." BJPsych Open 8, S1 (June 2022): S123. http://dx.doi.org/10.1192/bjo.2022.364.

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AimsParacetamol is a commonly used antipyretic and analgesic over the counter medication. In acute or chronic overuse it is associated with dose-dependent hepatic injury. There is a narrow therapeutic margin and that consistent use of as little as 7.5 g/day may be hazardous. Unintentional overdose with paracetamol is the most common cause of acute liver failure in the United Kingdom Here we present an unusual case of a 60-year-old lady with a reported chronic history of self-medicating with an above daily recommended dose of paracetamol without evidence of hepatic injury.MethodsA 60-year-old Caucasian lady known to psychiatric services for 20 years with Recurrent Depressive disorder, Obsessive Compulsive Disorder (OCD), Dependent Personality Disorder with Borderline personality traits. She reported consuming 32 tablets of paracetamol (16gm per day) every day for the past 11 years. She experienced obsessions of fear that if she did not take a particular number of paracetamols in a day then her friends will come to harm and her anxiety was relieved by the compulsion of consuming supratherapeutic doses of paracetamol. There was no evidence of misuse of any other medications other than paracetamol. Her blood investigations revealed liver function tests within normal limits and ultrasound of the liver was unremarkable.ResultsA literature search of “paracetamol or acetaminophen” and “no liver or hepatic” and “damage or injury” found only one case report. The case reported that studies of paracetamol metabolism were performed in a 58-year-old female with rheumatoid arthritis who had consumed 15–20 g paracetamol daily for 5 years without developing liver damage and data were compared with results in seven normal volunteers. The report concluded that a combination of slow paracetamol absorption, enhanced detoxication of paracetamol (by sulphation) and reduced metabolism to potentially cytotoxic metabolites may have reduced the risk of liver damage in this patient.ConclusionIn OCD, misusing medications can be an uncommon presentation of compulsive acts to relieve anxiety. The diagnostic dilemma of factitious illness is probable, however supratherapeutic use of paracetamol without physical harm is rare but possible.

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Ahmed Mejbel, Elham, Saif Subhi Noori, Adeeb Shakir Mahmood, Hussein Riyadh Abdul Kareem Al-Hetty, and Mohammed H. Musleh. "Oleuropein Protects against the Development of Kidneys Induced by Paracetamol in Albino Male Rats." HAYATI Journal of Biosciences 32, no. 1 (November 22, 2024): 254–62. http://dx.doi.org/10.4308/hjb.32.1.254-262.

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Paracetamol treatment is considered one of the treatments used to relieve pain and antipyretic. Therefore, excessive doses and long-term use lead to organ toxicity. Paracetamol treatment is considered one of the treatments used to relieve pain and antipyretic. Therefore, excessive doses and long-term use lead to organ toxicity. The aim of the study was to investigate the protective effect of Oleuropein extracted from olive leaves on the physiological and histological aspects induced by Paracetamol in a rat model. The methods used 25 albino Swiss rats randomly distributed into five groups with the same number. The unit of control is given normal saline. Paracetamol (750 mg/kg) was injected into the group once. In the treatment groups (50 mg/kg, 100 mg/kg, 150 mg/kg). The Administration of Paracetamol's result significantly increased blood urea, creatinine, sodium, and potassium levels, and their blood concentrations decreased with Oleuropein (P 0.05). In addition, Oleuropein extracted from olive leaves relieved some symptoms, including acute vascular congestion caused by a dose of Paracetamol. Compared with paracetamol treatment, there is an infiltration of inflammatory cells and severe nephrotoxicity in the tubules. According to this study, the Oleuropein extracted from olive leaves can be used to prevent kidney damage, and It is not recommended to give Paracetamol, which increases kidney disorders.

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LORELL, Juan, Kathy IVANA, Joselyn P. W. TANOTO, Michael MICHAEL, Sarah JESCIKA, Nicolaas R. P. GAUTAMA, Olivia TJOA, Keisha ALINA, Arli A. PARIKESIT, and Fandi SUTANTO. "In silico testing of C9H12ClNO2 and C6H5Cl2NO as derivatives of acetaminophen using molecular docking method." Notulae Scientia Biologicae 16, no. 1 (March 11, 2024): 11632. http://dx.doi.org/10.55779/nsb16111632.

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Paracetamol, a commonly used analgesic and antipyretic medication, is well-known for its ability to relieve pain and reduce temperature. However, there is a constant push to improve its therapeutic efficacy, especially towards increasing its oral bioavailability. The increase in bioavailability will lead to a better reception of the drugs by the body. This research aims to provide valuable insights into the molecular mechanisms underlying paracetamol’s mode of action and propose novel strategies for enhancing its therapeutic effectiveness. We investigated the notion of functional group alteration by molecular docking as a strategy to increase the efficacy of paracetamol in this work. Using modern computational approaches, it could be conducted through the examination of the structural characteristics and active regions of paracetamol and its target receptors. Additionally, molecular docking simulations were used to examine the binding interactions between paracetamol and its target receptors, offering insights into the essential functional groups required for ligand-receptor recognition. Tests of several molecular docking techniques and scoring functions allowed the researchers to find potential alterations that might improve its pharmacological characteristics. By integrating structural analysis, molecular docking studies, and computational screening, the uncovering of promising modifications that can significantly improve paracetamol’s efficacy was expected. Ultimately, this work may lead to the development of next-generation analgesics with superior pharmacological profiles, providing enhanced pain relief and fever reduction for patients.

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8

&NA;. "Paracetamol see Dextropropoxyphene/paracetamol." Reactions Weekly &NA;, no. 350 (May 1991): 10. http://dx.doi.org/10.2165/00128415-199103500-00043.

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9

&NA;. "Paracetamol see Aspirin + paracetamol." Reactions Weekly &NA;, no. 360 (July 1991): 9. http://dx.doi.org/10.2165/00128415-199103600-00053.

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10

Nguyen, Tinh Thu, Dung Thi Ngoc Nguyen, Tam Thi Thanh Pham, and Ju-Lee Oei. "Prophylaxis of Patent Ductus Arteriosus with Paracetamol in Extremely Low Gestational Age Newborns (ELGANs): A Single-Institution Observational Study in Vietnam." Children 10, no. 12 (December 17, 2023): 1934. http://dx.doi.org/10.3390/children10121934.

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Introduction: Prophylactic paracetamol for extremely low gestation age neonates (ELGAN, <27 weeks’ gestation) with symptomatic patent ductus arteriosus (sPDA) in high-income countries (HIC) reduces medical and surgical interventions. Its effectiveness in low-to-middle-income countries (LMIC) remains uncertain. This study assesses prophylactic paracetamol’s impact on sPDA interventions in ELGANs in an LMIC. Methods: This is a retrospective cohort study that compared a historical cohort of ELGANs that were treated with oral ibuprofen or intravenous paracetamol after diagnosis of sPDA (n = 104) with infants (n = 76) treated with prophylactic paracetamol (20 mg/kg loading, 7.5 mg/kg qid for 4 days), in a tertiary neonatal intensive care unit (NICU) in Vietnam. Oral ibuprofen or intravenous therapeutic paracetamol were administered if prophylactic paracetamol failed to close sPDA. Surgical ligation was conducted if targeted medical intervention failed, or the infant deteriorated from conditions attributable to sPDA. Results: In the historical cohort, 57 (55%) infants died within 7 days of life compared to 18 (24%) from the prophylactic cohort (p < 0.01). Of the survivors, 21 (45%) of the historical and 23 (39.7%) of the prophylactic cohort required surgical ligation (p = 0.6). Duration of hospitalization for survivors was lower in the prophylactic cohort (mean 74 vs. 97 days, p = 0.01). In the prophylactic cohort, 24 (41%) infants did not need further treatment while 34 (59%) required further treatment including ibuprofen and/or paracetamol 28 (48%) and surgical ligation 22 (38%). Conclusions: Prophylactic paracetamol for ELGAN in LMIC does not reduce the need for surgical ligation, sPDA rates, and other PDA-related morbidities in infants who survive beyond 7 days of age. It may reduce the risk of death and the duration of hospitalization but further study into the reasons behind this need to be determined with larger studies.

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11

Kamakia, Faith, Stephen Ouma, and Richard Kagia. "Pharmacokinetic and pharmacodynamic profiling of compounds similar to paracetamol from zinc database: an in silico analysis." F1000Research 12 (June 21, 2023): 720. http://dx.doi.org/10.12688/f1000research.133839.1.

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Introduction: Paracetamol is the most used drug for the management of pain and as an antipyretic through its mechanism of action on Cox 1,2 and 3 receptors. Paracetamol is a lipid-soluble molecule that can pass through the Blood Brain Barrier. Paracetamol has been formulated differently to ensure the optimal onset and duration of action as both analgesic and as antipyretic. Paracetamol overdose is associated with major side effects such as liver damage through its metabolite N-acetyl-p-benzoquinone Imine. Methods: This study generated zinc compounds that are similar in structure to Paracetamol through Ligand-based virtual screening. Molecular docking of these compounds to Cox 1, 2, and 3 receptors followed through Structure-based virtual screening. Compounds with better docking scores to these receptors were analyzed for pharmacokinetics and toxicity profiles. Results: ZINC01714506; 0.986; ZINC01714507; 0.986; and ZINC00394165; 0.987 showed the highest docking scores to cox 3 receptor with probability scores of -6.7kcal/mol, -6.4 and -6.2 kcal/ mol as compared to Paracetamol with -5.3kcal/mol. ZINC01714507; 0.986; ZINC01714506; 0.986; and ZINC00394165; 0.987; showed higher docking scores to Cox 2 with docking scores of -8.3kcal.mol, -8.1kcal/mole and -8.0 kcal/mol compared to paracetamol with -6.6kcal/mol. ZINC00394165; 0.987; ZINC00406627; 0.980; and ZINC01714506; 0.986; showed highest docking scores to Cox-1 than paracetamol with scores of -7.7kcal/mol, -7.6 and-7.6kcal/mol. ZINC01714506; 0.986 was predicted the safest with oral LD50 of 2000mg/kg as compared to paracetamol’s 338mg/kg. ZINC00294715; 0.980, ZINC01747085; 0.985, ZINC00394165; 0.987, ZINC00406627; 0.980, ZINC01557001; 0.987 and ZINC19281575; 0.992 were predicted hepatoactive. ZINC00294715; 0.980; ZINC01557001; 0.987; and ZINC19281575; 0.992; lack Blood Brain Barrier permeation. All compounds showed high GIT absorption and all conform to Lipinski’s rule of five. Conclusion: ZINC01557001; 0.987; ZINC01714506; 0.986; ZINC34120167; 0.994; ZINC00394165; 0.987, ZINC01714507; 0.986; and ZINC01747085; 0.985; are promising in drug discovery for new analgesic and antipyretic drugs, based on better docking scores and better oral LD50

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12

&NA;. "Paracetamol see Dextropropoxyphene/paracetamol abuse." Reactions Weekly &NA;, no. 366 (August 1991): 8. http://dx.doi.org/10.2165/00128415-199103660-00035.

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&NA;. "Paracetamol see Phenobarbital + paracetamol poisoning." Reactions Weekly &NA;, no. 313 (August 1990): 7. http://dx.doi.org/10.2165/00128415-199003130-00033.

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Amirul Fauziah, Dewi rashati,. "PENGARUH VARIASI KONSENTRASI AMILUM Zea mays (L) SEBAGAI BAHAN PENGHANCUR SECARA GRANULASI BASAH TERHADAP SIFAT FISIK TABLET PARASETAMOL." JURNAL ILMIAH FARMASI AKADEMI FARMASI JEMBER 2, no. 1 (January 28, 2021): 1–6. http://dx.doi.org/10.53864/jifakfar.v2i1.15.

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This research is aims to determine the effect of variation concentration starch Zea mays (L) as disintegration agent in wet granulation to physical characteristics paracetamo. This research used eperimental method by one shot case study. Paracetamol as active ingredient, starch zea mays (L)as desintegrant agent, PVPK30 as binding agent, avicel as filler and Mg stearat as lubricants. The result of SPSS showed significant (p0,05), that means no difference in the three formulation. The first test is weight uniformity show that from column A and B qualify weight requirement range, hardness test. Showed significant (p0,05) that means nodifference in the three formulation. Friability test showed significant (p 0,05) the last is disintegration time test showed that significant (P0,05) no difference in the three formulation. The research of physic characteristic of the tablet showed that hardness test and time test not qualif. Analysis SPSS showed (p0,05) no difference in the three formulation and amilum had no effect on physical test of paracetamol tablet.Keywords: zea mays (L),paracetamol,physicial characteristic

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15

Rahimi, Omid, Nilufar Asadi Louie, Alireza Salehi, and Firouz Faed Maleki. "Hepatorenal Protective Effects of Hydroalcoholic Extract of Solidago canadensis L. against Paracetamol-Induced Toxicity in Mice." Journal of Toxicology 2022 (December 17, 2022): 1–11. http://dx.doi.org/10.1155/2022/9091605.

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Paracetamol (AKA acetaminophen) is a widely used drug and is used for mild to moderate pains, such as mild osteoarthritis, toothache, headache, and pain caused by minimally invasive surgeries. Despite being a harmless drug in lower doses, acetaminophen can be toxic to the liver and kidneys if overdosed and even results in death. In this study, the therapeutic effects of Solidago canadensis L. extract (SCE) were investigated. 48 adult male Swiss albino mice (20–30 grams) were randomly divided into six groups of 8. The control group was gavaged with normal saline every 12 hours for 6 days. The second group received paracetamol at a 500 mg/kg intraperitoneally (i.p) dose on the sixth day. The third, fourth, and fifth groups were gavaged doses of 125, 250, and 500 mg/kg of SCE every 12 hours for six days, respectively, and on the sixth day, we received paracetamol at a dose of 500 mg/kg i.p. The sixth group only received SCE every 12 hours at a dose of 1000 mg/kg via gavaging for six days. On the seventh day (24 hours after paracetamol injection), blood samples were collected to measure the serum level of creatinine, uric acid, blood urea nitrogen (BUN), total protein, albumin, alanine transaminase (ALT), aspartate transaminase (AST), alkaline phosphatase (ALP), and total and direct bilirubin, and liver and kidney tissues were also sampled for histopathological examination. It was observed that paracetamol caused a considerable increase in the ALT, AST, ALP, uric Acid, and BUN levels ( P < 0.01 ), while those in SCE-treated groups were significantly lower. In addition, various lesions in the paracetamol group were observed, while in the SCE-receiving groups, receiving prophylactic SCE inhibited the high-intense lesions such as the infiltration of inflammatory cells, hyperemia, and vacuolar degeneration, which decreased significantly in the control group in comparison with that of the paracetamol group ( P < 0.05 ). In conclusion, SCE can have substantial protective effects against paracetamol’s hepatorenal toxicity.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1377 (November 2011): 29–30. http://dx.doi.org/10.2165/00128415-201113770-00098.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 700 (May 1998): 10. http://dx.doi.org/10.2165/00128415-199807000-00033.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 738 (February 1999): 10. http://dx.doi.org/10.2165/00128415-199907380-00028.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 741 (March 1999): 12. http://dx.doi.org/10.2165/00128415-199907410-00044.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1174 (October 2007): 22–23. http://dx.doi.org/10.2165/00128415-200711740-00065.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1184 (January 2008): 29. http://dx.doi.org/10.2165/00128415-200811840-00089.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1187 (February 2008): 22–23. http://dx.doi.org/10.2165/00128415-200811870-00072.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1194-1195 (March 2008): 29. http://dx.doi.org/10.2165/00128415-200811940-00106.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1133 (January 2007): 20–21. http://dx.doi.org/10.2165/00128415-200711330-00072.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1134 (January 2007): 21. http://dx.doi.org/10.2165/00128415-200711340-00071.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1139 (February 2007): 18. http://dx.doi.org/10.2165/00128415-200711390-00056.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1143 (March 2007): 20. http://dx.doi.org/10.2165/00128415-200711430-00067.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1148 (April 2007): 27. http://dx.doi.org/10.2165/00128415-200711480-00086.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1149 (April 2007): 20. http://dx.doi.org/10.2165/00128415-200711490-00063.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1149 (April 2007): 21. http://dx.doi.org/10.2165/00128415-200711490-00064.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1155 (June 2007): 14–15. http://dx.doi.org/10.2165/00128415-200711550-00041.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1156 (June 2007): 20–21. http://dx.doi.org/10.2165/00128415-200711560-00063.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1357 (June 2011): 27. http://dx.doi.org/10.2165/00128415-201113570-00088.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1359 (July 2011): 30. http://dx.doi.org/10.2165/00128415-201113590-00118.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1361 (July 2011): 32. http://dx.doi.org/10.2165/00128415-201113610-00117.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1371 (October 2011): 30. http://dx.doi.org/10.2165/00128415-201113710-00110.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1371 (October 2011): 30. http://dx.doi.org/10.2165/00128415-201113710-00114.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 1372 (October 2011): 27. http://dx.doi.org/10.2165/00128415-201113720-00085.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 581 (December 1995): 10. http://dx.doi.org/10.2165/00128415-199505810-00035.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 591 (March 1996): 10. http://dx.doi.org/10.2165/00128415-199605910-00032.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 592 (March 1996): 8. http://dx.doi.org/10.2165/00128415-199605920-00024.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 424 (October 1992): 10. http://dx.doi.org/10.2165/00128415-199204240-00047.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 426 (November 1992): 11. http://dx.doi.org/10.2165/00128415-199204260-00049.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 429 (November 1992): 11. http://dx.doi.org/10.2165/00128415-199204290-00058.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 438 (February 1993): 11. http://dx.doi.org/10.2165/00128415-199304380-00044.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 455 (June 1993): 11. http://dx.doi.org/10.2165/00128415-199304550-00049.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 461 (July 1993): 10. http://dx.doi.org/10.2165/00128415-199304610-00054.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 609 (July 1996): 10. http://dx.doi.org/10.2165/00128415-199606090-00030.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 651 (May 1997): 9. http://dx.doi.org/10.2165/00128415-199706510-00028.

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&NA;. "Paracetamol." Reactions Weekly &NA;, no. 660 (July 1997): 9–10. http://dx.doi.org/10.2165/00128415-199706600-00030.

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