Relevant bibliographies by topics / Pathogenetic mechanisms

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Pathogenetic mechanisms'

Author: Grafiati
Published: 28 May 2022
Last updated: 31 July 2025

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Journal articles on the topic "Pathogenetic mechanisms"

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Makhmudovich, Asadullayev Maksud, Rakhimbayeva Gulnara Sattarovna, Vakhabova Nargiza Maksudovna, and Jangirov Shukhrat Azamatovich. "Hyperhomocysteinemia And Pathogenetic Mechanisms Of Ischemic Stroke." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 02 (2021): 66–76. http://dx.doi.org/10.37547/tajmspr/volume03issue02-10.

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The article is intended to give basic information and the role of homocysteine in the human body. The amino acid homocysteine is a product of methionine demethylation. When the level of homocysteine increases, it damages the tissue structures of the arteries, initiating the release of cytokines, cyclins and other inflammatory mediators. Its accumulation leads to loosening of the arterial walls, the formation of local defects in the endothelium, which, in turn, leads to the deposition of cholesterol and calcium on the vascular wall. Hyperhomocysteinemia as a consequence of impaired homocysteine
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Armanini, Decio, Lorenzo Calò, and Andrea Semplicini. "Pseudohyperaldosteronism: Pathogenetic Mechanisms." Critical Reviews in Clinical Laboratory Sciences 40, no. 3 (2003): 295–335. http://dx.doi.org/10.1080/713609355.

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Wollenberg, A., S. Kraft, T. Oppel, and T. Bieber. "Atopic dermatitis: pathogenetic mechanisms." Clinical and Experimental Dermatology 25, no. 7 (2000): 530–34. http://dx.doi.org/10.1046/j.1365-2230.2000.00699.x.

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Trubitsyna, I., T. Tarasova, L. Vinokurova, and A. Smirnova. "Pathogenetic mechanisms acute pancreatitis." Pancreatology 13, no. 3 (2013): S56. http://dx.doi.org/10.1016/j.pan.2013.04.190.

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Cottier, H., J. Hodler, and R. Kraft. "Oxidative Stress: Pathogenetic Mechanisms." Complementary Medicine Research 2, no. 5 (1995): 233–39. http://dx.doi.org/10.1159/000210155.

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Tanashyan, M. M., K. V. Antonova, N. E. Spryshkov, A. А. Panina, O. V. Lagoda, and E. P. Shchukina. "Diabetic Polyneuropathy: from Pathogenesis to Pathogenetic Therapy." Effective Pharmacotherapy 20, no. 8 (2024): 54–62. http://dx.doi.org/10.33978/2307-3586-2024-20-8-54-62.

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The growing worldwide prevalence of prediabetes and diabetes mellitus (DM) has led to an increase in the incidence of related chronic complications, the most common of which is diabetic polyneuropathy (DPN). The development of DPN is caused by chronic hyperglycemia and potentially modifiable cardiovascular risk factors, including elevated triglyceride levels, body mass index, smoking and hypertension. Social determinants of health are also risk factors for DPN. The review presents data on the pathophysiology of DPN with consideration of the causes of the sequence of symptoms. Discussed the mec
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Petrovič, Daniel. "Candidate Genes for Proliferative Diabetic Retinopathy." BioMed Research International 2013 (2013): 1–9. http://dx.doi.org/10.1155/2013/540416.

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Several candidate genes have been so far implicated in the pathogenesis of proliferative diabetic retinopathy (PDR) in subjects with type 2 diabetes. Since the principal pathogenetic mechanisms for diabetic retinopathy (DR) and PDR are different, the main pathogenetic mechanism in DR is increased vascular permeability, whereas in PDR the crucial pathogenetic mechanisms are fibrosis and neoangiogenesis. Due to that fact, different candidate genes are expected to be involved in the development of either DR or PDR. None of the candidate genes, however, can be fully and solely responsible for the
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Chumakova, G. A., A. V. Ott, N. G. Veselovskaya, O. V. Gritsenko, and N. N. Shenkova. "PATHOGENETIC MECHANISMS OF LEPTIN RESISTANCE." Russian journal of Cardiology, no. 4 (November 27, 2015): 107. http://dx.doi.org/10.15829/1560-4071-2015-4-107-110.

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Cooke, T. D. V. "Pathogenetic Mechanisms in Polyarticular Osteoarthritis." Clinics in Rheumatic Diseases 11, no. 2 (1985): 203–38. http://dx.doi.org/10.1016/s0307-742x(21)00540-3.

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Schena, Francesco P., and Loreto Gesualdo. "Pathogenetic Mechanisms of Diabetic Nephropathy." Journal of the American Society of Nephrology 16, no. 3 suppl 1 (2005): S30—S33. http://dx.doi.org/10.1681/asn.2004110970.

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Dissertations / Theses on the topic "Pathogenetic mechanisms"

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Booler, Helen. "Pathogenetic mechanisms in the dystroglycanopathies pathogenesis." Thesis, Royal Veterinary College (University of London), 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669190.

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Törnblom, Hans. "Pathogenetic mechanisms in irritable bowel syndrome /." Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-287-3/.

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Pappa, Anastasia. "Pathogenetic mechanisms in thyroid-associated ophthalmopathy." Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299354.

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Orsetti, Valeria. "Pathogenetic mechanisms in motor neuron diseases." Doctoral thesis, Università degli studi di Padova, 2010. http://hdl.handle.net/11577/3421997.

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Amyotrophic Lateral Sclerosis, ALS, and Spinal and Bulbar Muscular Atrophy, SBMA (or Kennedy’s disease, KD) are two rare degenerative disorders of nervous system, characterized by early involvement of motor neurons. ALS is characterized by degeneration and death of upper and lower motor neurons, which leads to generalized muscle weakness and atrophy, speech and swallowing disabilities, and progressive paralysis until death is caused by respiratory failure. Its pathogenesis is still unknown. KD is an inherited X-linked motor neuronal disorder caused by an abnormal expansion of a trinucleotide C
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Guadagnin, Eleonora. "Pathogenetic Mechanisms of Collagen Type VI-Related Muscular Dystrophies." Doctoral thesis, Università degli studi di Padova, 2016. http://hdl.handle.net/11577/3424331.

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The overall focus of thesis is determining key disease-driving molecular pathways involved in the progression of a specific subtype of neuromuscular diseases, the collagen type VI-related muscular dystrophies (COL6RD). These kinds of muscular disorder encompass a wide spectrum of diseases, ranging from the severe Ullrich muscular dystrophy (UCMD) to the mild Bethlem myopathy (BM), characterized by a plethora of clinical manifestations. Both UCMD and BM usually arise at birth or early childhood with hypotonia and progressive muscle weakness, joint contractures and hyperlaxity of the distal join
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Gunnarsson, Iva. "SLE : pathogenetic mechanisms in nephritis and sulphasalazine-induced lupus reactions /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3654-4/.

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Tymofiychuk, I. R. "Sex hormones participation in the pathogenetic mechanisms of Alzheimer's disease." Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18014.

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Pancrazi, Laura. "Foxg1: intracellular localization and possible pathogenetic mechanisms in Rett syndrome." Doctoral thesis, Scuola Normale Superiore, 2014. http://hdl.handle.net/11384/85985.

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The Forkhead box G1 (FoxG1) is a transcription factor essential for the forebrain development and involved in pathogenesis of Rett syndrome. Notwithstanding the importance of this protein, little is known about the modalities by which it exerts its cellular functions. In this thesis I investigated, in cell culture and in animal model, the molecular mechanisms of Foxg1 action and the pathophysiological consequences of Foxg1 haploinsufficiency. Using fluorescence recovery after photobleaching strategy, I investigated the chromatin binding dynamics of Foxg1 in its wild-type form or carrying som
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Kvasnytska, О. В. "Pathogenetic mechanisms of functional renal impairment in patients with chronic hepatitis." Thesis, БДМУ, 2021. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18593.

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Bua, Gloria <1987&gt. "Parvovirus B19: Virus-Cell Interactions, Pathogenetic Mechanisms and Development of Compounds with Antiviral Activity." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2016. http://amsdottorato.unibo.it/7624/1/PhDthesis_BUA.pdf.

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The pathogenic Parvovirus B19 is characterized by a strict adaptation to erythroid progenitor cells (EPCs), a heterogeneous population of differentiating cells. The aim of the work was the characterization of B19V infection dynamics in EPCs in dependence on the cell differentiation stage, in terms of distribution of infected cells, synthesis of viral nucleic acids and production of infectious virus. EPCs at early differentiation stage led to an abortive infection, without viral replication. EPCs at later stages were permissive, with highest levels of viral replicative activity at day 9 and low
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Books on the topic "Pathogenetic mechanisms"

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D, Andreani, Gueriguian John L, and Striker Gary E. 1934-, eds. Diabetic complications: Epidemiology and pathogenetic mechanisms. Raven Press, 1991.

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A, Gimbrone Michael, Schoen Frederick J, and United States and Canadian Academy of Pathology., eds. Cardiovascular pathology: Clinicopathologic correlations and pathogenetic mechanisms. Williams & Wilkins, 1995.

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Tse, Wai Yee. Neutrophil Fc[gamma] receptor polymorphisms and pathogenetic mechanisms of neutrophil activation in anti-neutrophil cytoplasm (ANCA)-associated vasculitis. University of Birmingham, 1999.

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Chistyakova, Guzel, Lyudmila Ustyantseva, Irina Remizova, Vladislav Ryumin, and Svetlana Bychkova. CHILDREN WITH EXTREMELY LOW BODY WEIGHT: CLINICAL CHARACTERISTICS, FUNCTIONAL STATE OF THE IMMUNE SYSTEM, PATHOGENETIC MECHANISMS OF THE FORMATION OF NEONATAL PATHOLOGY. AUS PUBLISHERS, 2022. http://dx.doi.org/10.26526/monography_62061e70cc4ed1.46611016.

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The purpose of the monograph, which contains a modern view of the problem of adaptation of&#x0D; children with extremely low body weight, is to provide a wide range of doctors with basic information&#x0D; about the clinical picture, functional activity of innate and adaptive immunity, prognostic criteria&#x0D; of postnatal pathology, based on their own research. The specific features of the immunological&#x0D; reactivity of premature infants of various gestational ages who have developed bronchopulmonary&#x0D; dysplasia (BPD) and retinopathy of newborns (RN) from the moment of birth and after
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Sir Dorabji Tata Symposium (10th 2009 Bangalore, India). Mechanisms of microbial pathogenesis. Macmillan Publishers India, 2010.

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Genco, C. A. Neisseria: Molecular mechanisms of pathogenesis. Caister Academic Press, 2010.

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Hilbi, Hubert, ed. Molecular Mechanisms in Legionella Pathogenesis. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-40591-4.

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Gemma, Carmelina. Neuroinflammation: Pathogenesis, mechanisms and management. Nova Science Publisher's, 2011.

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Paul, Prem S., David H. Francis, and David A. Benfield, eds. Mechanisms in the Pathogenesis of Enteric Diseases. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1828-4.

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Howden, Peter John. Potential oxidative mechanisms involved in asbestos pathogenesis. University of Birmingham, 1998.

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Book chapters on the topic "Pathogenetic mechanisms"

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Ostojic, Miodrag C., Eugenio Picano, and Aleksandra Arandjelovic. "Pathogenetic Mechanisms of Stress." In Stress Echocardiography. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20958-6_5.

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Picano, Eugenio. "Pathogenetic Mechanisms of Stress." In Stress Echocardiography. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-76466-3_5.

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Picano, Eugenio. "Pathogenetic Mechanisms of Stresses." In Stress Echocardiography. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-662-13061-2_6.

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Picano, Eugenio. "Pathogenetic Mechanisms of Stress." In Stress Echocardiography. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-10090-5_8.

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Adorini, L. "Pathogenetic Mechanisms of Autoimmunity." In From Basic Immunology to Immune-Mediated Demyelination. Springer Milan, 1999. http://dx.doi.org/10.1007/978-88-470-2143-3_4.

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Picano, Eugenio. "Pathogenetic Mechanisms of Stress." In Stress Echocardiography. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05096-5_8.

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Picano, Eugenio. "Pathogenetic Mechanisms of Stresses." In Stress Echocardiography. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-662-02979-4_7.

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Dörr, W., and T. Herrmann. "Pathogenetic Mechanisms of Lung Fibrosis." In Medical Radiology. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55613-5_3.

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Pastore, Saveria, Francesca Mascia, Maria Laura Giustizieri, Alberto Giannetti, and Giampiero Girolomoni. "Pathogenetic Mechanisms of Atopic Dermatitis." In Inflammation. Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9702-9_9.

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Conti, Ilaria, Christine Dube, and Barrett J. Rollins. "Chemokine-Based Pathogenetic Mechanisms in Cancer." In Novartis Foundation Symposia. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470856734.ch3.

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Conference papers on the topic "Pathogenetic mechanisms"

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Kotiuzhynska, S. G. "Endothelial dysfunction and haemostatic disorders: common pathogenetic mechanisms." In GLOBAL MODERN TRENDS IN RESEARCH. Baltija Publishing, 2024. http://dx.doi.org/10.30525/978-9934-26-446-7-18.

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Ciresi, Michele, Daniele Previtero, Giusi L'Abbate, et al. "Sarcoidosis meets COVID-19: do they share common pathogenetic mechanisms?" In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.pa2483.

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Malyutina, N. N., and O. U. Shilkova. "SUDDEN CARDIAC DEATH PREVALENCE, RISK FACTORS, CAUSES AND MECHANISMS (REVIEW)." In The 17th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2023). FSBSI «IRIOH», 2023. http://dx.doi.org/10.31089/978-5-6042929-1-4-2023-1-296-300.

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The article presents materials on the relevance and the prevalence of sudden cardiac death with the description of epidemiological data, risk factors, causes and the mechanisms of its development. Purpose: To analyze the reported data on the prevalence, causes, risk factors and the main pathogenetic mechanisms of sudden cardiac death. Conclusion: Sudden cardiac death is a global problem of modern medicine.This issue is particularly relevant for the workers of the working age. Cardiovascular mortality in Russia is one of the highest in the world. Therefore, we have identified and summarized the
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Umnyagina, I. A. "METHODOLOGICAL APPROACHES TO THE EARLY DIAGNOSIS OF CARDIOVASCULAR PATHOLOGY IN WORKERS IN HARMFUL WORKING CONDITIONS." In The 16th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2021). FSBSI “IRIOH”, 2021. http://dx.doi.org/10.31089/978-5-6042929-2-1-2021-1-519-522.

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Abstract. The aim of the study is to assess the informativeness of a number of clinical, functional, laboratory parameters as indicators of cardiovascular pathology in workers exposed to harmful industrial factors during mandatory periodic medical examinations, to identify some pathogenetic mechanisms of the formation of the risk of cardiovascular diseases (CVD). The experienced workers (n=310) of the metallurgical enterprise of the Nizhny Novgorod region were examined. The sphygmometry indicators, the content of endothelin-1, nitric oxide, lipid profile, ox-LDL, and the levels of IgG-class au
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Zöller, Marie, Christoph H. Mayr, Michal Mastalerz, et al. "Investigating cigarette smoke-induced pathogenetic mechanisms in interstitial lung disease: A multi-omics approach." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.pa1718.

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Melnikov, A., P. D. Solymosi, P. Pennitz, et al. "Cellular Mechanisms Underlying the Pathogenetic Role of the Primary Cilium in Pulmonary Arterial Hypertension." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6790.

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Guselnikova, V. V., O. V. Kirik, I. A. Nikitina, et al. "NEW METHODS FOR MORPHOLOGICAL DIAGNOSTICS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-314.

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The work is devoted to the development and improvement of methods and approaches for morphological biomedical research. The use of this methods can help clarify the pathogenetic mechanisms and improve diagnostic accuracy of socially significant diseases, such as amyloid cardiomyopathy, Alzheimer’s disease, myocardial infarction, and liver fibrosis.
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Kim, Da Som, Youngjae Park, Mi-La Cho, and Seung-Ki Kwok. "LO-013 Novel pathogenetic mechanisms mediated by dysregulation of TRIM21-STING-type I interferon axis in lupus." In The 15th International Congress on Systemic Lupus Erythematosus and The 43rd KCR Annual Scientific Meeting & 17th International Symposium (LUPUS & KCR 2023). Lupus Foundation of America, 2023. http://dx.doi.org/10.1136/lupus-2023-kcr.13.

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Carleo, Giusy, Yi-Shin Lee, Agnese Secondo, Francesco Miceli, and Maurizio Taglialatela. "Multi-electrode array (MEASs) to investigate pathogenetic disease mechanisms and pharmacological properties in iPSC-derived neurons modelling neuropsychiatric diseases." In 2022 IEEE International Conference on Metrology for Extended Reality, Artificial Intelligence and Neural Engineering (MetroXRAINE). IEEE, 2022. http://dx.doi.org/10.1109/metroxraine54828.2022.9967629.

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Guzzo, M. P., B. Lucchino, G. Bonfiglio, et al. "AB0112 Pathogenetic mechanisms in early rheumatoid arthritis: possible correlation between th17 and treg cells and gut microbiota structure: a pilot study." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.6654.

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Reports on the topic "Pathogenetic mechanisms"

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Sidenkova, Alena, Olga Kremleva, Sergey Bogdanov, Olga Esina, and Alena Melnik. Electronic training manual "Psychiatry, medical psychology". SIB-Expertise, 2024. http://dx.doi.org/10.12731/er0786.29012024.

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The electronic training course ""Psychiatry, Medical Psychology"" was compiled in accordance with the requirements of the Federal State Educational Standard of Higher Education, specialty 31.05.01 General Medicine, approved by order of the Ministry of Education and Science of the Russian Federation dated February 9, 2016 No. 95, and taking into account the requirements of the professional standard 02.009 “Physician (precinct general practitioner)”, approved by order of the Ministry of Labor and Social Protection of the Russian Federation dated March 21, 2017 No. 293n, as well as in accordance
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Splitter, Gary, and Menachem Banai. Microarray Analysis of Brucella melitensis Pathogenesis. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7709884.bard.

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Original Objectives 1. To determine the Brucella genes that lead to chronic macrophage infection. 2. To identify Brucella genes that contribute to infection. 3. To confirm the importance of Brucella genes in macrophages and placental cells by mutational analysis. Background Brucella spp. is a Gram-negative facultative intracellular bacterium that infects ruminants causing abortion or birth of severely debilitated animals. Brucellosis continues in Israel, caused by B. melitensis despite an intensive eradication campaign. Problems with the Rev1 vaccine emphasize the need for a greater understand
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Eisenstein, Edward. Elucidating Mechanisms of Rust Pathogenesis for Engineering Resistance in Poplar. Office of Scientific and Technical Information (OSTI), 2022. http://dx.doi.org/10.2172/1905547.

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Mawassi, Munir, and Valerian Dolja. Role of RNA Silencing Suppression in the Pathogenicity and Host Specificity of the Grapevine Virus A. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7592114.bard.

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RNA silencing is a defense mechanism that functions against virus infection and involves sequence-specific degradation of viral RNA. Diverse RNA and DNA viruses of plants encode RNA silencing suppressors (RSSs), which, in addition to their role in viral counterdefense, were implicated in the efficient accumulation of viral RNAs, virus transport, pathogenesis, and determination of the virus host range. Despite rapidly growing understanding of the mechanisms of RNA silencing suppression, systematic analysis of the roles played by diverse RSSs in virus biology and pathology is yet to be completed
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Montie, Thomas C. Mechanism of Flagellar Vaccine Protection Related to Pseudomonas Pathogenesis in Trauma Burns. Defense Technical Information Center, 1989. http://dx.doi.org/10.21236/ada203539.

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Orkin, Stuart H. Exploring the Mechanisms of Pathogenesis in Prostate Cancer Involving TMPRSS2-ERG (Or ETV1) Gene Rearrangement. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada491425.

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Orkin, Stuart H. Exploring the Mechanisms of Pathogenesis in Prostate Cancer Involving TMPRSS2-ERG (Or ETV1) Gene Rearrangement. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada501163.

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Lazarus, Rachel C. Protein Modification: A Proposed Mechanism for the Long-Term Pathogenesis of Traumatic Brain Injury. Defense Technical Information Center, 2015. http://dx.doi.org/10.21236/ad1012716.

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Sordillo, Lorraine, Don Wojchowski, Gary Perdew, Arthur Saran, and Gabriel Leitner. Identification of Staphylococcus aureaus Virulence Factors Associated with Bovine Mastitis. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7574340.bard.

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Staphylococcus aureus is a major cause of mastitis in dairy cattle. The organism is able to adhere to and penetrate mammary epithelium, forming deep seated abscesses that result in chronic infections. This study was based on the observation that certain genotypes of S. aureus are isolated more frequently from field cases of bovine mastitis than others and the most prevalent genotypes of S. aureus have an increased ability to resist neutrophil phagocytosis and killing compared to the rare variants. It was hypothesized that these predominating genotypes differentially express virulence factors t
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Zhu, Qiaochu, Jin Zhou, Hai Huang, et al. Risk factors associated with amyotrophic lateral sclerosis: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.9.0118.

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Review question / Objective: To identify and list the risk factors associated with the onset and progression of ALS. Condition being studied: Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disorder affecting the upper and lower motor neurons in the spinal bulb, cerebral cortex, and spinal cord. The clinical processing symptoms accompany muscle atrophy, fasciculation, and fatigue of limbs, which can lead to general paralysis and death from respiratory failure within 3-5 years after the onset of this disease. Though the pathogenesis of ALS is still unclear, exploring the associations
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