Relevant bibliographies by topics / Trauma hemorrhagic shock

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Trauma hemorrhagic shock'

Author: Grafiati
Published: 7 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "Trauma hemorrhagic shock"

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Horton, J. W. "Cardiac contractile effects of ethanolism and hemorrhagic shock." American Journal of Physiology-Heart and Circulatory Physiology 262, no. 4 (1992): H1096—H1103. http://dx.doi.org/10.1152/ajpheart.1992.262.4.h1096.

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Moderate ethanol consumption, associated with cardiac depression, occurs in greater than 50% of trauma. Hemorrhagic shock, a significant component of trauma in the clinical setting, causes intrinsic cardiac contractile dysfunction. In this study, we used an isolated heart model to determine whether acute ethanolism increases the cardiovascular risk associated with hemorrhagic shock. We hypothesized that hemorrhagic shock in the acutely intoxicated subject would cause significantly greater cardiac dysfunction compared with that observed in a nonintoxicated subject. A total of 116 guinea pigs wa
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D'Alessandro, Angelo, Hunter B. Moore, Ernest E. Moore, et al. "Early hemorrhage triggers metabolic responses that build up during prolonged shock." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 308, no. 12 (2015): R1034—R1044. http://dx.doi.org/10.1152/ajpregu.00030.2015.

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Metabolic staging after trauma/hemorrhagic shock is a key driver of acidosis and directly relates to hypothermia and coagulopathy. Metabolic responses to trauma/hemorrhagic shock have been assayed through classic biochemical approaches or NMR, thereby lacking a comprehensive overview of the dynamic metabolic changes occurring after shock. Sprague-Dawley rats underwent progressive hemorrhage and shock. Baseline and postshock blood was collected, and late hyperfibrinolysis was assessed (LY30 >3%) in all of the tested rats. Extreme and intermediate time points were collected to assay the dynam
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GODINHO, MAURICIO, PEDRO PADIM, PAULO ROBERTO B. EVORA, and SANDRO SCARPELINI. "Curbing Inflammation in hemorrhagic trauma: a review." Revista do Colégio Brasileiro de Cirurgiões 42, no. 4 (2015): 273–78. http://dx.doi.org/10.1590/0100-69912015004013.

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Trauma is one of the world's leading causes of death within the first 40 years of life and thus a significant health problem. Trauma accounts for nearly a third of the lost years of productive life before 65 years of age and is associated with infection, hemorrhagic shock, reperfusion syndrome, and inflammation. The control of hemorrhage, coagulopathy, optimal use of blood products, balancing hypo and hyperperfusion, and hemostatic resuscitation improve survival in cases of trauma with massive hemorrhage. This review discusses inflammation in the context of trauma-associated hemorrhagic shock.
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Laserna, Anna Karen Carrasco, Yiyang Lai, Guihua Fang, et al. "Metabolic Profiling of a Porcine Combat Trauma-Injury Model Using NMR and Multi-Mode LC-MS Metabolomics—A Preliminary Study." Metabolites 10, no. 9 (2020): 373. http://dx.doi.org/10.3390/metabo10090373.

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Profiles of combat injuries worldwide have shown that penetrating trauma is one of the most common injuries sustained during battle. This is usually accompanied by severe bleeding or hemorrhage. If the soldier does not bleed to death, he may eventually succumb to complications arising from trauma hemorrhagic shock (THS). THS occurs when there is a deficiency of oxygen reaching the organs due to excessive blood loss. It can trigger massive metabolic derangements and an overwhelming inflammatory response, which can subsequently lead to the failure of organs and possibly death. A better understan
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Liu, Fu-Chao, Chih-Wen Zheng, and Huang-Ping Yu. "Maraviroc-Mediated Lung Protection following Trauma-Hemorrhagic Shock." BioMed Research International 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/5302069.

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Objectives.The peroxisome proliferator-activated receptor gamma (PPARγ) pathway exerts anti-inflammatory effects in response to injury. Maraviroc has been shown to have potent anti-inflammatory effects. The aim of this study was to investigate whether PPARγplays an important role in maraviroc-mediated lung protection following trauma-hemorrhage.Methods.Male Sprague-Dawley rats underwent trauma-hemorrhage (mean blood pressure maintained at approximately 35–40 mmHg for 90 minutes), followed by fluid resuscitation. During resuscitation, a single dose of maraviroc (3 mg/kg, intravenously) with and
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Napolitano, Lena M. "Resuscitation following trauma and hemorrhagic shock." Critical Care Medicine 23, no. 5 (1995): 795–97. http://dx.doi.org/10.1097/00003246-199505000-00001.

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Chu, Xiaogang, Kumar Subramani, Marie Warren, and Raghavan Pillai Raju. "Innate immune response in acute lung injury following hemorrhagic shock." Journal of Immunology 198, no. 1_Supplement (2017): 70.9. http://dx.doi.org/10.4049/jimmunol.198.supp.70.9.

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Abstract Trauma is the major cause of death for Americans under the age of 46 years. Hemorrhagic shock accounts for up to 40% of trauma-related deaths. Hemorrhagic shock evokes an acute, non-specific, systemic inflammatory response syndrome (SIRS) resulting in the damage to multiple organs. Acute lung injury (ALI) is one of the most serious complications in traumatic patients, however, the immunological mechanisms in ALI are still not well understood. Recently studies suggest that mitochondria play an important role in physical injury, leading to the onset of the SIRS. In order to address the
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Gauss, Tobias, Justin E. Richards, Costanza Tortù, et al. "Association of Early Norepinephrine Administration With 24-Hour Mortality Among Patients With Blunt Trauma and Hemorrhagic Shock." JAMA Network Open 5, no. 10 (2022): e2234258. http://dx.doi.org/10.1001/jamanetworkopen.2022.34258.

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ImportanceHemorrhagic shock is a common cause of preventable death after injury. Vasopressor administration for patients with blunt trauma and hemorrhagic shock is often discouraged.ObjectiveTo evaluate the association of early norepinephrine administration with 24-hour mortality among patients with blunt trauma and hemorrhagic shock.Design, Setting, and ParticipantsThis retrospective, multicenter, observational cohort study used data from 3 registries in the US and France on all consecutive patients with blunt trauma from January 1, 2013, to December 31, 2018. Patients were alive on admission
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Schneider, Christian P., Martin G. Schwacha, T. S. Anantha Samy, Kirby I. Bland та Irshad H. Chaudry. "Androgen-mediated modulation of macrophage function after trauma-hemorrhage: central role of 5α-dihydrotestosterone". Journal of Applied Physiology 95, № 1 (2003): 104–12. http://dx.doi.org/10.1152/japplphysiol.00182.2003.

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Androgens have been implicated as the causative factor for the postinjury immune dysfunction in males; however, it remains unknown whether androgens directly affect macrophages. To study this, male mice were sham operated or subjected to trauma (i.e., midline laparotomy) and hemorrhagic shock (mean arterial pressure, 30 ± 5 mmHg for 90 min and then resuscitated). The mice received the 5α-reductase inhibitor 4-hydroxyandrostenedione (4-OHA) before resuscitation. Plasma TNF-α, IL-6, and IL-10 levels were elevated after trauma-hemorrhage and normalized by 4-OHA. TNF-α and IL-6 production by splen
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Salvo, Nicholas, Angel M. Charles, and Alicia M. Mohr. "The Intersection of Trauma and Immunity: Immune Dysfunction Following Hemorrhage." Biomedicines 12, no. 12 (2024): 2889. https://doi.org/10.3390/biomedicines12122889.

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Hemorrhagic shock is caused by rapid loss of a significant blood volume, which leads to insufficient blood flow and oxygen delivery to organs and tissues, resulting in severe physiological derangements, organ failure, and death. Physiologic derangements after hemorrhage are due in a large part to the body’s strong inflammatory response, which leads to severe immune dysfunction, and secondary complications such as chronic immunosuppression, increased susceptibility to infection, coagulopathy, multiple organ failure, and unregulated inflammation. Immediate management of hemorrhagic shock include
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Dissertations / Theses on the topic "Trauma hemorrhagic shock"

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Talving, Peep. "Aspects of hepatoduodenal trauma and fluid therapy in hemorrhagic shock /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-740-5/.

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Alexander, Geoffrey C. "The Effect of Adding Drag-Reducing Polymers to Resuscitation Fluid During Hemorrhagic Shock on Skeletal Muscle Microcirculation." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/1522.

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Previous studies have shown an increase in survival when a minute amount of drag-reducing polymers were added to a resuscitation fluid. The purpose of this investigation was to examine the effect of adding a minute amount of the drag-reducing polymer polyethylene glycol to a resuscitation fluid, on the microcirculation of skeletal muscle during a volume-controlled hemorrhage model. The spinotrapezius muscle in twelve male Sprague Dawley rats was exteriorized for microvascular measurements of the arterioles. The diameters of the three levels of arterioles, interstitial fluid PO2, and RBC veloc
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Labruto, Fausto. "Modifications of cardiovascular response to ischemia and trauma /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-379-5/.

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Ji, Soo-Yeon. "COMPUTER-AIDED TRAUMA DECISION MAKING USING MACHINE LEARNING AND SIGNAL PROCESSING." VCU Scholars Compass, 2008. http://scholarscompass.vcu.edu/etd/1628.

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Over the last 20 years, much work has focused on computer-aided clinical decision support systems due to a rapid increase in the need for management and processing of medical knowledge. Among all fields of medicine, trauma care has the highest need for proper information management due to the high prevalence of complex, life-threatening injuries. In particular, hemorrhage, which is encountered in most traumatic injuries, is a dominant factor in determining survival in both civilian and military settings. This complication can be better managed using a more in-depth analysis of patient informat
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Lundin, Sandra, and Jonas Molin. "Räddar liv eller slösar tid? : prehospital vätskebehandlings effekter på patienter i hemorragisk chock." Thesis, Sophiahemmet Högskola, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:shh:diva-3742.

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Trauma är den ledande dödsorsaken i Sverige för människor mellan 15 och 44 år och en stor andel dör till följd av blödning som uppkommer vid skadetillfället. Blödning fortsätter också vara den ledande orsaken till traumarelaterad död som kunde ha varit förebyggbar både civilt och militärt. Traumaomhändertagandet är komplext, ofta tidskritiskt och ambulanspersonalen är ofta de som först får vårda dessa patienter ute på skadeplats och därav blir ambulanspersonalens första bedömning och omhändertagande av stor betydelse. Vätskebehandling för kritiskt skadade traumapatienter i hemorragisk chock el
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Dufour-Gaume, Frédérique. "Enjeux, préparation et évaluation de produits sanguins labiles innovants adaptés aux blessés de guerre." Electronic Thesis or Diss., université Paris-Saclay, 2023. http://www.theses.fr/2023UPASQ076.

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Le blessé de guerre est un blessé grave qui associe choc hémorragique et polytraumatisme. Malgré les progrès thérapeutiques des dernières années, l'hémorragie reste la première cause de décès évitable chez ce type de blessés. Les patients qui survivent aux premières heures de leurs blessures voient leur pronostic vital et fonctionnel menacé par les complications secondaires. Les protocoles de prise en charge des blessés de guerre basés sur le principe de damage control ressuscitation et la transfusion massive de sang total ont permis de réduire considérablement les décès par choc hémorragique.
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Bergis, Benjamin. "Choc hémorragique traumatique et rhabdomyolyse : caractérisation d'une synergie lésionnelle délétère et prévention de l'insuffisance rénale aiguë post-traumatique." Electronic Thesis or Diss., Université Paris Cité, 2025. http://www.theses.fr/2025UNIP5005.

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Le choc hémorragique et la rhabdomyolyse sont deux causes majeures d'insuffisance rénale aiguë (IRA) dans les suites d'un traumatisme sévère. Bien qu'ils soient souvent associés, leurs effets combinés sur la perfusion, l'oxygénation, la fonction et la morphologie rénales demeurent méconnus. Des études expérimentales ont mis en évidence une synergie délétère entre ces deux agressions sur le rein, aggravant les paramètres de fonction et de perfusion rénale. Parmi les mécanismes possibles, la myoglobine, une métalloprotéine libérée en grande quantité lors de la rhabdomyolyse, semble jouer un rôle
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Prunet, Bertrand. "Contusion pulmonaire : aspects physiopathologiques et conséquences thérapeutiques." Thesis, Aix-Marseille, 2015. http://www.theses.fr/2015AIXM5001.

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L’association lésionnelle d’une contusion pulmonaire et d’un état de choc hémorragique est fréquente et constitue un réel chalenge thérapeutique. La prise en charge de ce choc va nécessiter une réanimation hémodynamique dans laquelle le remplissage vasculaire tient une place centrale. Mais dans ce contexte de poumon contus, il devra être raisonné car délétère sur le plan pulmonaire, notamment en terme d'oedème et d'altération de la compliance. Ce remplissage devra donc être titré, basé sur des objectifs tensionnels clairs et un monitorage hémodynamique fiable. L'utilisation de solutés à haut p
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Morrison, C. Anne Horwitz Irwin Hwang Lu-Yu. "Hypotensive resuscitation versus standard fluid resuscitation for the management of trauma patients in hemorrhagic shock : the safety phase of a randomized controlled trial." 2009. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1465583.

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Fraga, Caroline Anne Pires. "The use of Tranexamic acid in trauma patients with hemorrhagic shock - a retrospective study in a tertiary care hospital." Master's thesis, 2020. https://hdl.handle.net/10216/128787.

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Introdução - O choque hemorrágico é a maior complicação e causa de morbi-mortalidade em doentes com trauma. Neste estudo, pretendemos avaliar o impacto do ácido tranexâmico em doentes com choque hemorrágico por trauma num hospital terciário. Material e métodos - Este estudo, realizado no Centro Hospitalar e Universitário São João, incluiu doentes adultos, admitidos no Serviço de Medicina Intensiva, por choque hemorrágico secundário a trauma. Definimos choque hemorrágico como pressão arterial sistólica ≤90 mmHg e/ou frequência cardíaca ≥110 bpm. Na ausência destes parâmetros, prevaleceu o juízo
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Books on the topic "Trauma hemorrhagic shock"

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J, Sugerman Harvey, and DeMaria Eric J. 1959-, eds. Cytokines in trauma and hemorrhage. Chapman & Hall, 1997.

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1950-, Pope Andrew MacPherson, French Geoffrey, Longnecker David E. 1939-, and Institute of Medicine (U.S.). Committee on Fluid Resuscitation for Combat Casualties, eds. Fluid resuscitation: State of the science for treating combat casualties and civilian injuries. National Academy Press, 1999.

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Sabato, Stefan. Massive Transfusion in a Child. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0024.

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The traditional early management of hemorrhagic shock is currently being challenged, and many centers around the world have already changed their practice. Damage-control resuscitation, in conjunction with damage-control surgery, has been shown to improve major morbidity and mortality outcomes in adults. In children there is little direct evidence for these new approaches, but supporting evidence is accumulating. This chapter will introduce these concepts while also reinforcing the core principles of managing acute hemorrhage in the trauma setting.
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Fluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.

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Medicine, Institute of, and Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.

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Medicine, Institute of, and Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.

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Medicine, Institute of, and Committee on Fluid Resuscitation for Combat Casualties. Fluid Resuscitation: State of the Science for Treating Combat Casualties and Civilian Injuries. National Academies Press, 1999.

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Weyker, Paul David, Christopher Allen-John Webb, and Tricia E. Brentjens. Hypovolemic Shock. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0097.

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Broadly defined, hypovolemia represents inadequate circulating plasma volume leading to decreased cardiac preload and thus decreased cardiac output and blood pressure. Many classification schemes have been proposed to categorize hypovolemia based on relative levels of decreased plasma volume. Common causes of hypovolemic shock during the perioperative period include hemorrhage and diuretic use. In general, studies support a conservative hemoglobin goal of about 7 g/dL as compared with a liberal goal of 10 g/dL in hemodynamically stable patients without active cardiac ischemia or risk factors.
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Finn, Patrick C., and Michael C. Reade. Bleeding Emergencies (DRAFT). Edited by Raghavan Murugan and Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0010.

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This chapter is concerned with coagulopathic and non-coagulopathic bleeding in the perioperative period, after trauma, and spontaneously, as a result of hematologic and other disease. The initial assessment and management of all potentially bleeding patients is to stop any obvious bleeding through mechanical first aid measures, then address airway or breathing compromise, and obtain intravenous (or intraosseous) access. Obvious external hemorrhage is easily identified, but most patients with bleeding emergencies who are already hospitalized will have occult blood loss. Physical examination sho
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Book chapters on the topic "Trauma hemorrhagic shock"

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Krol, Caitlin, Graham Spurzem, Bryan Sandler, and Claire Sampankanpanich Soria. "Hemorrhagic Shock in Trauma (IVC Tear)." In Anesthesiology Simulation. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-80228-7_4.

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Lewis, Scott, Graham Spurzem, Bryan Sandler, and Claire Sampankanpanich Soria. "Hemorrhagic Shock in Trauma (Stab Wound)." In Anesthesiology Simulation. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-80228-7_3.

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Krol, Caitlin, Graham Spurzem, Bryan Sandler, and Claire Sampankanpanich Soria. "Hemorrhagic Shock in Trauma (Solid Organ Injury)." In Anesthesiology Simulation. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-80228-7_5.

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Zhao, Zi-Gang, Yu-Ping Zhang, Li-Min Zhang, and Ya-Xiong Guo. "Lymph Formation and Transport: Role in Trauma-Hemorrhagic Shock." In Severe Trauma and Sepsis. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-3353-8_5.

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Livingston, D. H., J. Hseih, T. Murphy, and B. F. Rush. "Hemorrhagic Shock Inhibits Myelopoiesis Independent of Bacterial Translocation." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_36.

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Ochoa, J. B., T. R. Billiar, and A. B. Peitzman. "The Role of Nitric Oxide in Hemorrhagic Shock and Trauma." In Shock, Sepsis, and Organ Failure — Nitric Oxide. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79343-1_4.

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Livingston, D. H., and M. A. Malangoni. "Effect of Therapeutic Gamma-Interferon Administration on Wound Infection After Resuscitated Hemorrhagic Shock." In Immune Consequences of Trauma, Shock, and Sepsis. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73468-7_66.

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Ertel, W., E. Faist, and I. H. Chaudry. "The Biological Characteristics of Cytokines and Their Interactions with Prostaglandins Following Hemorrhagic Shock." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_83.

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Tolentino, Rafael, Timothy J. Holleran, and Laura S. Johnson. "Futility of Care in Hemorrhagic Shock: When Prolonging the Massive Transfusion Protocol Is of No Benefit." In Difficult Decisions in Trauma Surgery. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81667-4_4.

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Stadlbauer, K. H., H. G. Wagner-Berger, C. Raedler, et al. "Survival with Full Vital Organ Function Recovery after Uncontrolled Hemorrhagic Shock in a Penetrating Liver Trauma Model with Vasopressin in Pigs." In Zurück in die Zukunft. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55611-1_443.

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Conference papers on the topic "Trauma hemorrhagic shock"

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Rahbar, Elaheh, Bryan A. Cotton, John B. Holcomb, and Charles E. Wade. "Reduced Plasma Oncotic Pressure is Indicative of Injury Severity in Trauma Patients." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14539.

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Trauma is the leading cause of death in people aged 1–44 years old, with hemorrhagic shock accounting for nearly half of these deaths. These fatalities often occur early, within 24 hours, but many are deemed potentially preventable with appropriate resuscitation. 1–2 The term “appropriate resuscitation” is somewhat ambiguous and subjective because of the lack of a standardized resuscitation protocol. We attribute this deficiency to our rudimentary understanding of the mechanisms of hemorrhagic shock and resuscitation.
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Thau, M. R., T. Liu, N. A. Sathe, et al. "Latent Class Analysis in a Trauma Cohort with Hemorrhagic Shock Identifies Two Distinct Sub-Phenotypes with a Differential Treatment Response to Blood Transfusion Ratios." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a5038.

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Sotudeh-Chafi, M., N. Abolfathi, A. Nick, V. Dirisala, G. Karami, and M. Ziejewski. "A Multi-Scale Finite Element Model for Shock Wave-Induced Axonal Brain Injury." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192342.

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Traumatic brain injuries (TBIs) involve a significant portion of human injuries resulting from a wide range of civilian accidents as well as many military scenarios. Axonal damage is one of the most common and important pathologic features of traumatic brain injury. Axons become brittle when exposed to rapid deformations associated with brain trauma. Accordingly, rapid stretch of axons can damage the axonal cytoskeleton, resulting in a loss of elasticity and impairment of axoplasmic transport. Subsequent swelling of the axon occurs in discrete bulb formations or in elongated varicosities that
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Nemeth, Christopher, Adam Amos-Binks, Gregory Rule, et al. "Real Time Battlefield Casualty Care Decision Support." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1002112.

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Tactical combat casualty care (TCCC) involves care for casualties in armed conflict from one’s own service (e.g., U.S. Marine Corps), other services (i.e., U.S. Army, Air Force,), allied forces, adversaries, and civilians. To minimize injury and preserve life, medics perform TCCC which includes casualty retrieval, stabilization and documentation, transport, triage, and treatment. In future scenarios, delays in evacuation are expected to require extended care including prolonged field care (PFC) over hours to days, increasing the potential for complications such as bloodstream infection (sepsis
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Guzas, Emily L., Stephen E. Turner, Matthew Babina, Brandon Casper, Thomas N. Fetherston, and Joseph M. Ambrico. "Validation of a Surrogate Model for Marine Mammal Lung Dynamics Under Underwater Explosive Impulse." In ASME 2019 Verification and Validation Symposium. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/vvs2019-5143.

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Abstract Primary blast injury (PBI), which relates gross blast-related trauma or traces of injury in air-filled tissues or those tissues adjacent to air-filled regions (rupture/lesions, contusions, hemorrhaging), has been documented in a number of marine mammal species after blast exposure [1, 2, 3]. However, very little is known about marine mammal susceptibility to PBI except in rare cases of opportunistic studies. As a result, traditional techniques rely on analyses using small-scale terrestrial mammals as surrogates for large-scale marine mammals. For an In-house Laboratory Independent Res
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Reports on the topic "Trauma hemorrhagic shock"

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Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality,Apoptosis, Inflammation and Mitochondrial Dysfunction. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada612817.

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Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada612818.

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Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction Using IL-6 as a Resuscitation Adjuvant. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada612819.

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